ResearcherSheng Jing HospitalActive in the last 2 years
Xiangpeng Meng
Matched on ORCID iD 0000-0002-8174-0494; OpenAlex author ID A5019993666. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 0, last author 6)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 10
2014: 1 paper · 2015: none · 2016: none · 2017: none · 2018: none · 2019: none · 2020: 1 paper · 2021: 1 paper · 2022: none · 2023: 3 papers · 2024: 3 papers
Papers 9
- PELI1 overexpression contributes to pancreatic cancer progression through upregulating ubiquitination-mediated INPP5J degradation. Co-author DOI 10.1016/j.cellsig.2024.111194↗
- HTF4 modulates the transcription of GID2 to promote the malignant biological behavior of pancreatic cancer. Last author DOI 10.1016/j.pan.2024.08.008↗
- The DDX6/KIFC1 signaling axis, as regulated by YY1, contributes to the malignant behavior of pancreatic cancer. Last author DOI 10.1096/fj.202400166r↗
- Aberrant expression of PELI1 caused by Jagged1 accelerates the malignant phenotype of pancreatic cancer. Last author DOI 10.1016/j.cellsig.2023.110877↗
- GID2 Interacts With CDKN3 and Regulates Pancreatic Cancer Growth and Apoptosis. Last author DOI 10.1016/j.labinv.2023.100122↗
- PSMD12 interacts with CDKN3 and facilitates pancreatic cancer progression. Last author DOI 10.1038/s41417-023-00609-y↗
- Case report: identification of ERC1-RET fusion in a patient with pancreatic ductal adenocarcinoma. Last author DOI 10.21037/gs-21-469↗
- Long non-coding RNA OIP5-AS1 promotes pancreatic cancer cell growth through sponging miR-342-3p via AKT/ERK signaling pathway. Co-author DOI 10.1007/s13105-020-00734-4↗
- Expression of stromal cell-derived factor 1 and CXCR7 ligand receptor system in pancreatic adenocarcinoma. Co-author DOI 10.1186/1477-7819-12-348↗
Closest collaborators
The 10 strongest of 10 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Wenyang Zhou · 5 shared papers, 2023–2024 · strength 1.19
- Paper PELI1 overexpression contributes to pancreatic cancer progression through upregulating ubiquitination-mediated INPP5J d… DOI 10.1016/j.cellsig.2024.111194↗
- Paper HTF4 modulates the transcription of GID2 to promote the malignant biological behavior of pancreatic cancer. DOI 10.1016/j.pan.2024.08.008↗
- Paper PSMD12 interacts with CDKN3 and facilitates pancreatic cancer progression. DOI 10.1038/s41417-023-00609-y↗
- Baosheng Wang · 6 shared papers, 2014–2023 · strength 1.07
- Paper PSMD12 interacts with CDKN3 and facilitates pancreatic cancer progression. DOI 10.1038/s41417-023-00609-y↗
- Paper Aberrant expression of PELI1 caused by Jagged1 accelerates the malignant phenotype of pancreatic cancer. DOI 10.1016/j.cellsig.2023.110877↗
- Paper GID2 Interacts With CDKN3 and Regulates Pancreatic Cancer Growth and Apoptosis. DOI 10.1016/j.labinv.2023.100122↗
- Jia Ma · 4 shared papers, 2023–2024 · strength 0.97
- Paper PELI1 overexpression contributes to pancreatic cancer progression through upregulating ubiquitination-mediated INPP5J d… DOI 10.1016/j.cellsig.2024.111194↗
- Paper HTF4 modulates the transcription of GID2 to promote the malignant biological behavior of pancreatic cancer. DOI 10.1016/j.pan.2024.08.008↗
- Paper PSMD12 interacts with CDKN3 and facilitates pancreatic cancer progression. DOI 10.1038/s41417-023-00609-y↗
- Xin Deng · 3 shared papers, 2023–2024 · strength 0.73
- Paper PELI1 overexpression contributes to pancreatic cancer progression through upregulating ubiquitination-mediated INPP5J d… DOI 10.1016/j.cellsig.2024.111194↗
- Paper HTF4 modulates the transcription of GID2 to promote the malignant biological behavior of pancreatic cancer. DOI 10.1016/j.pan.2024.08.008↗
- Paper GID2 Interacts With CDKN3 and Regulates Pancreatic Cancer Growth and Apoptosis. DOI 10.1016/j.labinv.2023.100122↗
- Jia Ma · 3 shared papers, 2020–2023 · strength 0.56
- Paper Aberrant expression of PELI1 caused by Jagged1 accelerates the malignant phenotype of pancreatic cancer. DOI 10.1016/j.cellsig.2023.110877↗
- Paper Case report: identification of ERC1-RET fusion in a patient with pancreatic ductal adenocarcinoma. DOI 10.21037/gs-21-469↗
- Paper Long non-coding RNA OIP5-AS1 promotes pancreatic cancer cell growth through sponging miR-342-3p via AKT/ERK signaling p… DOI 10.1007/s13105-020-00734-4↗
- Baosheng Wang · 2 shared papers, 2024 · strength 0.54
- Xin Deng · 1 shared paper, 2024 · strength 0.28
- Zhen Liu · 1 shared paper, 2024 · strength 0.28
- Zhen Liu · 2 shared papers, 2014–2020 · strength 0.21
- Paper Long non-coding RNA OIP5-AS1 promotes pancreatic cancer cell growth through sponging miR-342-3p via AKT/ERK signaling p… DOI 10.1007/s13105-020-00734-4↗
- Paper Expression of stromal cell-derived factor 1 and CXCR7 ligand receptor system in pancreatic adenocarcinoma. DOI 10.1186/1477-7819-12-348↗
- Xin Wu · 1 shared paper, 2020 · strength 0.15
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.