ResearcherUniversitätsmedizin RostockActive in the last 2 years
Robert Jaster
Matched on ORCID iD 0000-0002-8220-4570; OpenAlex author ID A5019793360. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 0, last author 3)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 21
2011: 1 paper · 2012: 2 papers · 2013: none · 2014: none · 2015: none · 2016: none · 2017: none · 2018: 1 paper · 2019: none · 2020: 1 paper · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: 2 papers
Papers 9
- Effects of triggers of senescence and senolysis in murine pancreatic cancer cells. Last author DOI 10.1016/j.hbpd.2024.06.001↗
- Evidence-Based Severity Assessment of Animal Models for Pancreatic Cancer. Co-author DOI 10.3390/biomedicines12071494↗
- Uncoupling protein 2 deficiency of non-cancerous tissues inhibits the progression of pancreatic cancer in mice. Last author DOI 10.1016/j.hbpd.2022.12.003↗
- Inhibition of KRAS, MEK and PI3K Demonstrate Synergistic Anti-Tumor Effects in Pancreatic Ductal Adenocarcinoma Cell Lines. Co-author DOI 10.3390/cancers14184467↗
- Towards biomarkers for outcomes after pancreatic ductal adenocarcinoma and ischaemic stroke, with focus on (co)-morbidity and ageing/cellular senescence (SASKi… Co-author DOI 10.1136/bmjopen-2020-039560↗
- Blocking Autophagy in Cancer-Associated Fibroblasts Supports Chemotherapy of Pancreatic Cancer Cells. Co-author DOI 10.3389/fonc.2018.00590↗
- Mathematical modelling unravels regulatory mechanisms of interferon-γ-induced STAT1 serine-phosphorylation and MUC4 expression in pancreatic cancer cells. Co-author DOI 10.1049/iet-syb.2011.0017↗
- Parameter identifiability and sensitivity analysis predict targets for enhancement of STAT1 activity in pancreatic cancer and stellate cells. Co-author DOI 10.1371/journal.pcbi.1002815↗
- Studies on mechanisms of interferon-gamma action in pancreatic cancer using a data-driven and model-based approach. Last author DOI 10.1186/1476-4598-10-13↗
Closest collaborators
The 21 strongest of 21 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Dietmar Zechner · 5 shared papers, 2018–2024 · strength 0.77
- Paper Effects of triggers of senescence and senolysis in murine pancreatic cancer cells. DOI 10.1016/j.hbpd.2024.06.001↗
- Paper Evidence-Based Severity Assessment of Animal Models for Pancreatic Cancer. DOI 10.3390/biomedicines12071494↗
- Paper Uncoupling protein 2 deficiency of non-cancerous tissues inhibits the progression of pancreatic cancer in mice. DOI 10.1016/j.hbpd.2022.12.003↗
- Georg Fuellen · 3 shared papers, 2020–2024 · strength 0.47
- Paper Effects of triggers of senescence and senolysis in murine pancreatic cancer cells. DOI 10.1016/j.hbpd.2024.06.001↗
- Paper Uncoupling protein 2 deficiency of non-cancerous tissues inhibits the progression of pancreatic cancer in mice. DOI 10.1016/j.hbpd.2022.12.003↗
- Paper Towards biomarkers for outcomes after pancreatic ductal adenocarcinoma and ischaemic stroke, with focus on (co)-morbidi… DOI 10.1136/bmjopen-2020-039560↗
- Hugo Murua Escobar · 3 shared papers, 2020–2023 · strength 0.38
- Paper Uncoupling protein 2 deficiency of non-cancerous tissues inhibits the progression of pancreatic cancer in mice. DOI 10.1016/j.hbpd.2022.12.003↗
- Paper Inhibition of KRAS, MEK and PI3K Demonstrate Synergistic Anti-Tumor Effects in Pancreatic Ductal Adenocarcinoma Cell Li… DOI 10.3390/cancers14184467↗
- Paper Towards biomarkers for outcomes after pancreatic ductal adenocarcinoma and ischaemic stroke, with focus on (co)-morbidi… DOI 10.1136/bmjopen-2020-039560↗
- Denis Revskij · 2 shared papers, 2023–2024 · strength 0.38
- Olaf Wolkenhauer A · 5 shared papers, 2011–2023 · strength 0.35
- Paper Uncoupling protein 2 deficiency of non-cancerous tissues inhibits the progression of pancreatic cancer in mice. DOI 10.1016/j.hbpd.2022.12.003↗
- Paper Towards biomarkers for outcomes after pancreatic ductal adenocarcinoma and ischaemic stroke, with focus on (co)-morbidi… DOI 10.1136/bmjopen-2020-039560↗
- Paper Mathematical modelling unravels regulatory mechanisms of interferon-γ-induced STAT1 serine-phosphorylation and MUC4 exp… DOI 10.1049/iet-syb.2011.0017↗
- Brigitte Vollmar · 2 shared papers, 2018–2024 · strength 0.26
- Simone Kumstel · 2 shared papers, 2018–2024 · strength 0.26
- Jakob Brandstetter · 1 shared paper, 2024 · strength 0.18
- Tim Schreiber · 1 shared paper, 2024 · strength 0.18
- Falko Lange · 3 shared papers, 2011–2020 · strength 0.16
- Paper Towards biomarkers for outcomes after pancreatic ductal adenocarcinoma and ischaemic stroke, with focus on (co)-morbidi… DOI 10.1136/bmjopen-2020-039560↗
- Paper Parameter identifiability and sensitivity analysis predict targets for enhancement of STAT1 activity in pancreatic canc… DOI 10.1371/journal.pcbi.1002815↗
- Paper Studies on mechanisms of interferon-gamma action in pancreatic cancer using a data-driven and model-based approach. DOI 10.1186/1476-4598-10-13↗
- Yixuan Ma · 1 shared paper, 2022 · strength 0.14
- Frank Ulrich Weiß · 1 shared paper, 2022 · strength 0.14
- Benjamin Schulz · 1 shared paper, 2022 · strength 0.14
- Markus M. Lerch · 1 shared paper, 2022 · strength 0.14
- Katja Rateitschak · 3 shared papers, 2011–2012 · strength 0.11
- Paper Mathematical modelling unravels regulatory mechanisms of interferon-γ-induced STAT1 serine-phosphorylation and MUC4 exp… DOI 10.1049/iet-syb.2011.0017↗
- Paper Parameter identifiability and sensitivity analysis predict targets for enhancement of STAT1 activity in pancreatic canc… DOI 10.1371/journal.pcbi.1002815↗
- Paper Studies on mechanisms of interferon-gamma action in pancreatic cancer using a data-driven and model-based approach. DOI 10.1186/1476-4598-10-13↗
- Larissa Henze · 1 shared paper, 2020 · strength 0.09
- Daniel H. Palmer · 1 shared paper, 2020 · strength 0.09
- Edgar Heinz Uwe Wendt · 1 shared paper, 2018 · strength 0.08
- Maria Schönrogge · 1 shared paper, 2018 · strength 0.08
- Xianbin Zhang · 1 shared paper, 2018 · strength 0.08
- Christina Kossow · 1 shared paper, 2012 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.