ResearcherTianjin Medical University Cancer Institute and HospitalActive in the last 2 years
Kaiyuan Wang
Matched on ORCID iD 0000-0002-8374-6427; OpenAlex author ID A5100681013. Sources: OpenAlex.
Their work
- Papers
- 3 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 16
2023: 1 paper · 2024: none · 2025: 2 papers
Papers 3
- Nociceptor neurons promote PDAC progression and cancer pain by interaction with cancer-associated fibroblasts and suppression of natural killer cells. Co-author DOI 10.1038/s41422-025-01098-4↗
- CLT-003 exerts anti-tumor activity in pancreatic cancer by blocking the PI3K/AKT/HIF-1α pathway. Co-author DOI 10.20892/j.issn.2095-3941.2025.0058↗
- Crosstalk Between Peripheral Innervation and Pancreatic Ductal Adenocarcinoma. Last author DOI 10.1007/s12264-023-01082-1↗
Closest collaborators
The 16 strongest of 16 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Xiuchao Wang · 3 shared papers, 2023–2025 · strength 0.59
- Paper Crosstalk Between Peripheral Innervation and Pancreatic Ductal Adenocarcinoma. DOI 10.1007/s12264-023-01082-1↗
- Paper Nociceptor neurons promote PDAC progression and cancer pain by interaction with cancer-associated fibroblasts and suppr… DOI 10.1038/s41422-025-01098-4↗
- Paper CLT-003 exerts anti-tumor activity in pancreatic cancer by blocking the PI3K/AKT/HIF-1α pathway. DOI 10.20892/j.issn.2095-3941.2025.0058↗
- Bo Ni · 3 shared papers, 2023–2025 · strength 0.59
- Paper Crosstalk Between Peripheral Innervation and Pancreatic Ductal Adenocarcinoma. DOI 10.1007/s12264-023-01082-1↗
- Paper Nociceptor neurons promote PDAC progression and cancer pain by interaction with cancer-associated fibroblasts and suppr… DOI 10.1038/s41422-025-01098-4↗
- Paper CLT-003 exerts anti-tumor activity in pancreatic cancer by blocking the PI3K/AKT/HIF-1α pathway. DOI 10.20892/j.issn.2095-3941.2025.0058↗
- Chongbiao Huang · 2 shared papers, 2025 · strength 0.37
- Paper Nociceptor neurons promote PDAC progression and cancer pain by interaction with cancer-associated fibroblasts and suppr… DOI 10.1038/s41422-025-01098-4↗
- Paper CLT-003 exerts anti-tumor activity in pancreatic cancer by blocking the PI3K/AKT/HIF-1α pathway. DOI 10.20892/j.issn.2095-3941.2025.0058↗
- Tiansuo Zhao · 2 shared papers, 2025 · strength 0.37
- Paper Nociceptor neurons promote PDAC progression and cancer pain by interaction with cancer-associated fibroblasts and suppr… DOI 10.1038/s41422-025-01098-4↗
- Paper CLT-003 exerts anti-tumor activity in pancreatic cancer by blocking the PI3K/AKT/HIF-1α pathway. DOI 10.20892/j.issn.2095-3941.2025.0058↗
- Yongjie Xie · 2 shared papers, 2025 · strength 0.37
- Paper Nociceptor neurons promote PDAC progression and cancer pain by interaction with cancer-associated fibroblasts and suppr… DOI 10.1038/s41422-025-01098-4↗
- Paper CLT-003 exerts anti-tumor activity in pancreatic cancer by blocking the PI3K/AKT/HIF-1α pathway. DOI 10.20892/j.issn.2095-3941.2025.0058↗
- Tianxing Zhou · 2 shared papers, 2025 · strength 0.37
- Paper Nociceptor neurons promote PDAC progression and cancer pain by interaction with cancer-associated fibroblasts and suppr… DOI 10.1038/s41422-025-01098-4↗
- Paper CLT-003 exerts anti-tumor activity in pancreatic cancer by blocking the PI3K/AKT/HIF-1α pathway. DOI 10.20892/j.issn.2095-3941.2025.0058↗
- Junjin Wang · 1 shared paper, 2023 · strength 0.22
- Jihui Hao · 1 shared paper, 2025 · strength 0.19
- Ying Ma · 1 shared paper, 2025 · strength 0.19
- Chao Yang · 1 shared paper, 2025 · strength 0.19
- Shengyu Yang · 1 shared paper, 2025 · strength 0.18
- Guannan Sheng · 1 shared paper, 2025 · strength 0.18
- Chunhua She · 1 shared paper, 2025 · strength 0.18
- Liangliang Wu · 1 shared paper, 2025 · strength 0.18
- Chao Xu · 1 shared paper, 2025 · strength 0.18
- Song Gao · 1 shared paper, 2025 · strength 0.18
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.