ResearcherShanghai Jiao Tong UniversityNo recent work listed
Yi Zhu
Matched on ORCID iD 0000-0002-8783-319X; OpenAlex author ID A5049166487. Sources: OpenAlex.
Their work
- Papers
- 19 (first author 4, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 53
2011: 1 paper · 2012: 2 papers · 2013: none · 2014: 6 papers · 2015: 1 paper · 2016: 1 paper · 2017: 2 papers · 2018: 2 papers · 2019: 1 paper · 2020: 2 papers · 2021: 1 paper
Papers 19
- Exosomes Derived from Pancreatic Cancer Cells Induce Osteoclast Differentiation Through the miR125a-5p/TNFRSF1B Pathway. First author DOI 10.2147/ott.s282319↗
- Glycolysis promotes the progression of pancreatic cancer and reduces cancer cell sensitivity to gemcitabine. Co-author DOI 10.1016/j.biopha.2019.109521↗
- FHL3 promotes pancreatic cancer invasion and metastasis through preventing the ubiquitination degradation of EMT associated transcription factors. Co-author DOI 10.18632/aging.102564↗
- Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancreatic cancer" [Cancer Le… Co-author DOI 10.1016/j.canlet.2019.03.031↗
- MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition via the Wnt/β-catenin signaling pathway b… Co-author DOI 10.1002/jcb.26270↗
- PIK3R3 Promotes Metastasis of Pancreatic Cancer via ZEB1 Induced Epithelial-Mesenchymal Transition. First author DOI 10.1159/000489382↗
- PEG10 overexpression induced by E2F-1 promotes cell proliferation, migration, and invasion in pancreatic cancer. First author DOI 10.1186/s13046-017-0500-x↗
- The clinical value, regulatory mechanisms, and gene network of the cancer-testis gene STK31 in pancreatic cancer. Co-author DOI 10.18632/oncotarget.16814↗
- GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2016.06.016↗
- NPM1 activates metabolic changes by inhibiting FBP1 while promoting the tumorigenicity of pancreatic cancer cells. Co-author DOI 10.18632/oncotarget.4167↗
- Snail recruits Ring1B to mediate transcriptional repression and cell migration in pancreatic cancer cells. Co-author DOI 10.1158/0008-5472.can-14-0181↗
- Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. Last author DOI 10.1186/1471-2407-14-738↗
- Specific-detection of clinical samples, systematic functional investigations, and transcriptome analysis reveals that splice variant MUC4/Y contributes to the … Co-author DOI 10.1186/s12967-014-0309-8↗
- H2AK119Ub1 and H3K27Me3 in molecular staging for survival prediction of patients with pancreatic ductal adenocarcinoma. Co-author DOI 10.18632/oncotarget.2126↗
- hsa-miR-141 downregulates TM4SF1 to inhibit pancreatic cancer cell invasion and migration. Co-author DOI 10.3892/ijo.2013.2189↗
- Pancreatic cancer counterattack: MUC4 mediates Fas-independent apoptosis of antigen-specific cytotoxic T lymphocyte. Co-author DOI 10.3892/or.2014.3016↗
- High expression of Galectin-1 in pancreatic stellate cells plays a role in the development and maintenance of an immunosuppressive microenvironment in pancreat… Co-author DOI 10.1002/ijc.26290↗
- Association of increased DNA methyltransferase expression with carcinogenesis and poor prognosis in pancreatic ductal adenocarcinoma. First author DOI 10.1007/s12094-012-0770-x↗
- The increase in the expression and hypomethylation of MUC4 gene with the progression of pancreatic ductal adenocarcinoma. Co-author DOI 10.1007/s12032-010-9683-0↗
Closest collaborators
The 25 strongest of 53 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Yi Miao · 10 shared papers, 2011–2020 · strength 0.54
- Paper Glycolysis promotes the progression of pancreatic cancer and reduces cancer cell sensitivity to gemcitabine. DOI 10.1016/j.biopha.2019.109521↗
- Paper PIK3R3 Promotes Metastasis of Pancreatic Cancer via ZEB1 Induced Epithelial-Mesenchymal Transition. DOI 10.1159/000489382↗
- Paper The clinical value, regulatory mechanisms, and gene network of the cancer-testis gene STK31 in pancreatic cancer. DOI 10.18632/oncotarget.16814↗
- Hao Chen · 6 shared papers, 2014–2019 · strength 0.34
- Paper Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mi… DOI 10.1016/j.canlet.2019.03.031↗
- Paper MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition v… DOI 10.1002/jcb.26270↗
- Paper GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancr… DOI 10.1016/j.canlet.2016.06.016↗
- Baiyong Shen · 6 shared papers, 2014–2019 · strength 0.34
- Paper Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mi… DOI 10.1016/j.canlet.2019.03.031↗
- Paper MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition v… DOI 10.1002/jcb.26270↗
- Paper GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancr… DOI 10.1016/j.canlet.2016.06.016↗
- Yunpeng Peng · 4 shared papers, 2014–2020 · strength 0.30
- Paper Glycolysis promotes the progression of pancreatic cancer and reduces cancer cell sensitivity to gemcitabine. DOI 10.1016/j.biopha.2019.109521↗
- Paper PIK3R3 Promotes Metastasis of Pancreatic Cancer via ZEB1 Induced Epithelial-Mesenchymal Transition. DOI 10.1159/000489382↗
- Paper PEG10 overexpression induced by E2F-1 promotes cell proliferation, migration, and invasion in pancreatic cancer. DOI 10.1186/s13046-017-0500-x↗
- Jiangning Gu · 4 shared papers, 2015–2019 · strength 0.26
- Paper Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mi… DOI 10.1016/j.canlet.2019.03.031↗
- Paper MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition v… DOI 10.1002/jcb.26270↗
- Paper GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancr… DOI 10.1016/j.canlet.2016.06.016↗
- Chenghong Peng · 5 shared papers, 2014–2019 · strength 0.26
- Paper Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mi… DOI 10.1016/j.canlet.2019.03.031↗
- Paper GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancr… DOI 10.1016/j.canlet.2016.06.016↗
- Paper NPM1 activates metabolic changes by inhibiting FBP1 while promoting the tumorigenicity of pancreatic cancer cells. DOI 10.18632/oncotarget.4167↗
- Qian Zhan · 4 shared papers, 2014–2019 · strength 0.26
- Paper Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mi… DOI 10.1016/j.canlet.2019.03.031↗
- Paper MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition v… DOI 10.1002/jcb.26270↗
- Paper GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancr… DOI 10.1016/j.canlet.2016.06.016↗
- Kuirong Jiang · 5 shared papers, 2012–2017 · strength 0.24
- Paper The clinical value, regulatory mechanisms, and gene network of the cancer-testis gene STK31 in pancreatic cancer. DOI 10.18632/oncotarget.16814↗
- Paper PEG10 overexpression induced by E2F-1 promotes cell proliferation, migration, and invasion in pancreatic cancer. DOI 10.1186/s13046-017-0500-x↗
- Paper Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. DOI 10.1186/1471-2407-14-738↗
- Wentao Gao · 5 shared papers, 2011–2017 · strength 0.24
- Paper The clinical value, regulatory mechanisms, and gene network of the cancer-testis gene STK31 in pancreatic cancer. DOI 10.18632/oncotarget.16814↗
- Paper PEG10 overexpression induced by E2F-1 promotes cell proliferation, migration, and invasion in pancreatic cancer. DOI 10.1186/s13046-017-0500-x↗
- Paper Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. DOI 10.1186/1471-2407-14-738↗
- Yizhao Zhou · 2 shared papers, 2020–2021 · strength 0.24
- Paper Exosomes Derived from Pancreatic Cancer Cells Induce Osteoclast Differentiation Through the miR125a-5p/TNFRSF1B Pathway. DOI 10.2147/ott.s282319↗
- Paper FHL3 promotes pancreatic cancer invasion and metastasis through preventing the ubiquitination degradation of EMT associ… DOI 10.18632/aging.102564↗
- Yulian Wu · 2 shared papers, 2020–2021 · strength 0.24
- Paper Exosomes Derived from Pancreatic Cancer Cells Induce Osteoclast Differentiation Through the miR125a-5p/TNFRSF1B Pathway. DOI 10.2147/ott.s282319↗
- Paper FHL3 promotes pancreatic cancer invasion and metastasis through preventing the ubiquitination degradation of EMT associ… DOI 10.18632/aging.102564↗
- Jishu Wei · 4 shared papers, 2012–2018 · strength 0.22
- Paper PIK3R3 Promotes Metastasis of Pancreatic Cancer via ZEB1 Induced Epithelial-Mesenchymal Transition. DOI 10.1159/000489382↗
- Paper PEG10 overexpression induced by E2F-1 promotes cell proliferation, migration, and invasion in pancreatic cancer. DOI 10.1186/s13046-017-0500-x↗
- Paper Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. DOI 10.1186/1471-2407-14-738↗
- Jingjing Zhang · 4 shared papers, 2014–2017 · strength 0.21
- Paper The clinical value, regulatory mechanisms, and gene network of the cancer-testis gene STK31 in pancreatic cancer. DOI 10.18632/oncotarget.16814↗
- Paper PEG10 overexpression induced by E2F-1 promotes cell proliferation, migration, and invasion in pancreatic cancer. DOI 10.1186/s13046-017-0500-x↗
- Paper Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. DOI 10.1186/1471-2407-14-738↗
- Xiaxing Deng · 4 shared papers, 2014–2018 · strength 0.21
- Paper MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition v… DOI 10.1002/jcb.26270↗
- Paper NPM1 activates metabolic changes by inhibiting FBP1 while promoting the tumorigenicity of pancreatic cancer cells. DOI 10.18632/oncotarget.4167↗
- Paper H2AK119Ub1 and H3K27Me3 in molecular staging for survival prediction of patients with pancreatic ductal adenocarcinoma. DOI 10.18632/oncotarget.2126↗
- Xiaole Zhu · 2 shared papers, 2018–2020 · strength 0.19
- Zekuan Xu · 5 shared papers, 2011–2014 · strength 0.18
- Paper Pancreatic cancer counterattack: MUC4 mediates Fas-independent apoptosis of antigen-specific cytotoxic T lymphocyte. DOI 10.3892/or.2014.3016↗
- Paper Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. DOI 10.1186/1471-2407-14-738↗
- Paper Specific-detection of clinical samples, systematic functional investigations, and transcriptome analysis reveals that s… DOI 10.1186/s12967-014-0309-8↗
- Junli Wu · 3 shared papers, 2012–2020 · strength 0.17
- Paper Glycolysis promotes the progression of pancreatic cancer and reduces cancer cell sensitivity to gemcitabine. DOI 10.1016/j.biopha.2019.109521↗
- Paper Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. DOI 10.1186/1471-2407-14-738↗
- Paper Association of increased DNA methyltransferase expression with carcinogenesis and poor prognosis in pancreatic ductal a… DOI 10.1007/s12094-012-0770-x↗
- Lingdi Yin · 2 shared papers, 2017–2018 · strength 0.16
- Xinchun Liu · 2 shared papers, 2017–2018 · strength 0.16
- Zipeng Lu · 3 shared papers, 2012–2017 · strength 0.14
- Paper The clinical value, regulatory mechanisms, and gene network of the cancer-testis gene STK31 in pancreatic cancer. DOI 10.18632/oncotarget.16814↗
- Paper Elevation of MMP-9 and IDO induced by pancreatic cancer cells mediates natural killer cell dysfunction. DOI 10.1186/1471-2407-14-738↗
- Paper High expression of Galectin-1 in pancreatic stellate cells plays a role in the development and maintenance of an immuno… DOI 10.1002/ijc.26290↗
- Gang Wei · 2 shared papers, 2016–2019 · strength 0.14
- Paper Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mi… DOI 10.1016/j.canlet.2019.03.031↗
- Paper GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancr… DOI 10.1016/j.canlet.2016.06.016↗
- Xiaxing Deng · 2 shared papers, 2016–2019 · strength 0.14
- Paper Corrigendum to "GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mi… DOI 10.1016/j.canlet.2019.03.031↗
- Paper GFRα2 prompts cell growth and chemoresistance through down-regulating tumor suppressor gene PTEN via Mir-17-5p in pancr… DOI 10.1016/j.canlet.2016.06.016↗
- Yiqun Fan · 1 shared paper, 2021 · strength 0.13
- Shi Chen · 2 shared papers, 2014–2018 · strength 0.12
- Paper MicroRNA-300 promotes apoptosis and inhibits proliferation, migration, invasion and epithelial-mesenchymal transition v… DOI 10.1002/jcb.26270↗
- Paper H2AK119Ub1 and H3K27Me3 in molecular staging for survival prediction of patients with pancreatic ductal adenocarcinoma. DOI 10.18632/oncotarget.2126↗
- Jingjing Zhang · 3 shared papers, 2011–2014 · strength 0.10
- Paper Pancreatic cancer counterattack: MUC4 mediates Fas-independent apoptosis of antigen-specific cytotoxic T lymphocyte. DOI 10.3892/or.2014.3016↗
- Paper Association of increased DNA methyltransferase expression with carcinogenesis and poor prognosis in pancreatic ductal a… DOI 10.1007/s12094-012-0770-x↗
- Paper The increase in the expression and hypomethylation of MUC4 gene with the progression of pancreatic ductal adenocarcinom… DOI 10.1007/s12032-010-9683-0↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.