ResearcherHuazhong University of Science and TechnologyNo recent work listed
Pingting Ying
Matched on ORCID iD 0000-0002-8884-1618; OpenAlex author ID A5082428035. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 13
2019: 2 papers · 2020: 2 papers · 2021: 2 papers · 2022: 2 papers
Papers 8
- Systematic analysis on expression quantitative trait loci identifies a novel regulatory variant in ring finger and WD repeat domain 3 associated with prognosis… Co-author DOI 10.1097/cm9.0000000000002180↗
- Aberrant RNA Splicing Is a Primary Link between Genetic Variation and Pancreatic Cancer Risk. Co-author DOI 10.1158/0008-5472.can-21-4367↗
- Identification of genetic variants in m6A modification genes associated with pancreatic cancer risk in the Chinese population. Co-author DOI 10.1007/s00204-021-02978-5↗
- A genetic variant conferred high expression of CAV2 promotes pancreatic cancer progression and associates with poor prognosis. Co-author DOI 10.1016/j.ejca.2021.04.008↗
- Evaluation of polymorphisms in microRNA-binding sites and pancreatic cancer risk in Chinese population. Co-author DOI 10.1111/jcmm.14906↗
- N6-methyladenosine mRNA methylation of PIK3CB regulates AKT signalling to promote PTEN-deficient pancreatic cancer progression. Co-author DOI 10.1136/gutjnl-2019-320179↗
- A genetic variant in PIK3R1 is associated with pancreatic cancer survival in the Chinese population. Co-author DOI 10.1002/cam4.2228↗
- A functional variant in the boundary of a topological association domain is associated with pancreatic cancer risk. Co-author DOI 10.1002/mc.23077↗
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jianbo Tian · 8 shared papers, 2019–2022 · strength 0.79
- Paper Systematic analysis on expression quantitative trait loci identifies a novel regulatory variant in ring finger and WD r… DOI 10.1097/cm9.0000000000002180↗
- Paper Identification of genetic variants in m6A modification genes associated with pancreatic cancer risk in the Chinese popu… DOI 10.1007/s00204-021-02978-5↗
- Paper Aberrant RNA Splicing Is a Primary Link between Genetic Variation and Pancreatic Cancer Risk. DOI 10.1158/0008-5472.can-21-4367↗
- Jiang Chang · 8 shared papers, 2019–2022 · strength 0.79
- Paper Systematic analysis on expression quantitative trait loci identifies a novel regulatory variant in ring finger and WD r… DOI 10.1097/cm9.0000000000002180↗
- Paper Identification of genetic variants in m6A modification genes associated with pancreatic cancer risk in the Chinese popu… DOI 10.1007/s00204-021-02978-5↗
- Paper Aberrant RNA Splicing Is a Primary Link between Genetic Variation and Pancreatic Cancer Risk. DOI 10.1158/0008-5472.can-21-4367↗
- Xiaoping Miao · 7 shared papers, 2019–2022 · strength 0.71
- Paper Systematic analysis on expression quantitative trait loci identifies a novel regulatory variant in ring finger and WD r… DOI 10.1097/cm9.0000000000002180↗
- Paper Identification of genetic variants in m6A modification genes associated with pancreatic cancer risk in the Chinese popu… DOI 10.1007/s00204-021-02978-5↗
- Paper Aberrant RNA Splicing Is a Primary Link between Genetic Variation and Pancreatic Cancer Risk. DOI 10.1158/0008-5472.can-21-4367↗
- Rong Zhong · 7 shared papers, 2019–2022 · strength 0.69
- Paper Systematic analysis on expression quantitative trait loci identifies a novel regulatory variant in ring finger and WD r… DOI 10.1097/cm9.0000000000002180↗
- Paper Identification of genetic variants in m6A modification genes associated with pancreatic cancer risk in the Chinese popu… DOI 10.1007/s00204-021-02978-5↗
- Paper A genetic variant conferred high expression of CAV2 promotes pancreatic cancer progression and associates with poor pro… DOI 10.1016/j.ejca.2021.04.008↗
- Ying Zhu · 6 shared papers, 2019–2021 · strength 0.56
- Paper Identification of genetic variants in m6A modification genes associated with pancreatic cancer risk in the Chinese popu… DOI 10.1007/s00204-021-02978-5↗
- Paper A genetic variant conferred high expression of CAV2 promotes pancreatic cancer progression and associates with poor pro… DOI 10.1016/j.ejca.2021.04.008↗
- Paper N6-methyladenosine mRNA methylation of PIK3CB regulates AKT signalling to promote PTEN-deficient pancreatic cancer prog… DOI 10.1136/gutjnl-2019-320179↗
- Ming Zhang · 4 shared papers, 2019–2022 · strength 0.40
- Paper Systematic analysis on expression quantitative trait loci identifies a novel regulatory variant in ring finger and WD r… DOI 10.1097/cm9.0000000000002180↗
- Paper Aberrant RNA Splicing Is a Primary Link between Genetic Variation and Pancreatic Cancer Risk. DOI 10.1158/0008-5472.can-21-4367↗
- Paper N6-methyladenosine mRNA methylation of PIK3CB regulates AKT signalling to promote PTEN-deficient pancreatic cancer prog… DOI 10.1136/gutjnl-2019-320179↗
- Shufang Mei · 3 shared papers, 2019–2020 · strength 0.24
- Paper Evaluation of polymorphisms in microRNA-binding sites and pancreatic cancer risk in Chinese population. DOI 10.1111/jcmm.14906↗
- Paper A functional variant in the boundary of a topological association domain is associated with pancreatic cancer risk. DOI 10.1002/mc.23077↗
- Paper A genetic variant in PIK3R1 is associated with pancreatic cancer survival in the Chinese population. DOI 10.1002/cam4.2228↗
- Juntao Ke · 3 shared papers, 2019–2020 · strength 0.24
- Paper Evaluation of polymorphisms in microRNA-binding sites and pancreatic cancer risk in Chinese population. DOI 10.1111/jcmm.14906↗
- Paper A functional variant in the boundary of a topological association domain is associated with pancreatic cancer risk. DOI 10.1002/mc.23077↗
- Paper A genetic variant in PIK3R1 is associated with pancreatic cancer survival in the Chinese population. DOI 10.1002/cam4.2228↗
- Ying Zhu · 2 shared papers, 2022 · strength 0.24
- Bin Li · 2 shared papers, 2019–2021 · strength 0.17
- Zhiyong Yang · 1 shared paper, 2022 · strength 0.11
- Min Wang · 1 shared paper, 2022 · strength 0.11
- Xiaoping Miao · 1 shared paper, 2019 · strength 0.08
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.