ResearcherIndian Institute of Integrative MedicineNo recent work listed
Anindya Goswami
Matched on ORCID iD 0000-0002-8920-2383; OpenAlex author ID A5043352981. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 0, last author 5)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 8
2015: 1 paper · 2016: none · 2017: none · 2018: 1 paper · 2019: none · 2020: 2 papers · 2021: 2 papers
Papers 6
- Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of pancreatic cancer cells. Last author DOI 10.1016/j.tranon.2020.100879↗
- Corrigendum to 'Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of pancreatic cancer cells' [Translational O… Last author DOI 10.1016/j.tranon.2021.101136↗
- Par-4 mediated Smad4 induction in PDAC cells restores canonical TGF-β/ Smad4 axis driving the cells towards lethal EMT. Last author DOI 10.1016/j.ejcb.2020.151076↗
- Par-4 activation restrains EMT-induced chemoresistance in PDAC by attenuating MDM-2. Last author DOI 10.1016/j.pan.2020.09.021↗
- Dual role of Par-4 in abrogation of EMT and switching on Mesenchymal to Epithelial Transition (MET) in metastatic pancreatic cancer cells. Last author DOI 10.1002/mc.22828↗
- Inhibition of Invasion in Pancreatic Cancer Cells by Conjugate of EPA with β(3,3)-Pip-OH via PI3K/Akt/NF-kB Pathway. Co-author DOI 10.1021/acsmedchemlett.5b00257↗
Closest collaborators
The 8 strongest of 8 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Mir Mohd Faheem · 5 shared papers, 2018–2021 · strength 0.59
- Paper Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of pancreatic cancer… DOI 10.1016/j.tranon.2020.100879↗
- Paper Corrigendum to 'Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of p… DOI 10.1016/j.tranon.2021.101136↗
- Paper Par-4 activation restrains EMT-induced chemoresistance in PDAC by attenuating MDM-2. DOI 10.1016/j.pan.2020.09.021↗
- Archana Katoch · 4 shared papers, 2018–2021 · strength 0.47
- Paper Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of pancreatic cancer… DOI 10.1016/j.tranon.2020.100879↗
- Paper Corrigendum to 'Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of p… DOI 10.1016/j.tranon.2021.101136↗
- Paper Par-4 mediated Smad4 induction in PDAC cells restores canonical TGF-β/ Smad4 axis driving the cells towards lethal EMT. DOI 10.1016/j.ejcb.2020.151076↗
- Shantibhusan Senapati · 3 shared papers, 2018–2021 · strength 0.35
- Paper Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of pancreatic cancer… DOI 10.1016/j.tranon.2020.100879↗
- Paper Corrigendum to 'Overlapping targets exist between the Par-4 and miR-200c axis which regulate EMT and proliferation of p… DOI 10.1016/j.tranon.2021.101136↗
- Paper Dual role of Par-4 in abrogation of EMT and switching on Mesenchymal to Epithelial Transition (MET) in metastatic pancr… DOI 10.1002/mc.22828↗
- Debasis Nayak · 3 shared papers, 2015–2020 · strength 0.25
- Paper Par-4 activation restrains EMT-induced chemoresistance in PDAC by attenuating MDM-2. DOI 10.1016/j.pan.2020.09.021↗
- Paper Dual role of Par-4 in abrogation of EMT and switching on Mesenchymal to Epithelial Transition (MET) in metastatic pancr… DOI 10.1002/mc.22828↗
- Paper Inhibition of Invasion in Pancreatic Cancer Cells by Conjugate of EPA with β(3,3)-Pip-OH via PI3K/Akt/NF-kB Pathway. DOI 10.1021/acsmedchemlett.5b00257↗
- Syed Mudabir Ahmad · 2 shared papers, 2020 · strength 0.24
- Sujit Suklabaidya · 1 shared paper, 2018 · strength 0.07
- Hina Amin · 1 shared paper, 2015 · strength 0.05
- Rajkishor Rai · 1 shared paper, 2015 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.