ResearcherNational Institutes of HealthActive in the last 2 years
Yuuki Ohara
Matched on ORCID iD 0000-0002-9171-4724; OpenAlex author ID A5034007772. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 16
2022: 2 papers · 2023: 1 paper · 2024: 4 papers · 2025: none · 2026: 2 papers
Papers 9
- Basal Marker ΔNp63α Is Enriched in Pancreatic Cancer Metastases but Not in Primary Tumors Post-Chemotherapy. First author DOI 10.1002/cam4.72322↗
- Molecular, metabolic, and histological subtypes of pancreatic ductal adenocarcinoma and its tumor microenvironment: Insights into tumor heterogeneity and clini… Co-author DOI 10.1016/j.pharmthera.2025.108946↗
- ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic reprogramming in pancreatic cancer. Co-author DOI 10.1002/ijc.34960↗
- LMO3 is a suppressor of the basal-like/squamous subtype and reduces disease aggressiveness of pancreatic cancer through glycerol 3-phosphate metabolism. Co-author DOI 10.1093/carcin/bgae011↗
- MIF/NR3C2 axis regulates glucose metabolism reprogramming in pancreatic cancer through MAPK-ERK and AP-1 pathways. Co-author DOI 10.1093/carcin/bgae025↗
- ADRA2A promotes the classical/progenitor subtype and reduces disease aggressiveness of pancreatic cancer. First author DOI 10.1093/carcin/bgae056↗
- SERPINB3-MYC axis induces the basal-like/squamous subtype and enhances disease progression in pancreatic cancer. Co-author DOI 10.1016/j.celrep.2023.113434↗
- The interactive role of inflammatory mediators and metabolic reprogramming in pancreatic cancer. Co-author DOI 10.1016/j.trecan.2022.03.004↗
- Dysregulation of HNF1B/Clusterin axis enhances disease progression in a highly aggressive subset of pancreatic cancer patients. Co-author DOI 10.1093/carcin/bgac092↗
Closest collaborators
The 16 strongest of 16 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- S. Perwez Hussain · 8 shared papers, 2022–2026 · strength 1.67
- Paper Molecular, metabolic, and histological subtypes of pancreatic ductal adenocarcinoma and its tumor microenvironment: Ins… DOI 10.1016/j.pharmthera.2025.108946↗
- Paper The interactive role of inflammatory mediators and metabolic reprogramming in pancreatic cancer. DOI 10.1016/j.trecan.2022.03.004↗
- Paper ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic re… DOI 10.1002/ijc.34960↗
- Paloma Moreno · 5 shared papers, 2023–2026 · strength 1.10
- Paper Molecular, metabolic, and histological subtypes of pancreatic ductal adenocarcinoma and its tumor microenvironment: Ins… DOI 10.1016/j.pharmthera.2025.108946↗
- Paper ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic re… DOI 10.1002/ijc.34960↗
- Paper LMO3 is a suppressor of the basal-like/squamous subtype and reduces disease aggressiveness of pancreatic cancer through… DOI 10.1093/carcin/bgae011↗
- Nader N Hanna · 6 shared papers, 2022–2024 · strength 1.03
- Paper ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic re… DOI 10.1002/ijc.34960↗
- Paper LMO3 is a suppressor of the basal-like/squamous subtype and reduces disease aggressiveness of pancreatic cancer through… DOI 10.1093/carcin/bgae011↗
- Paper ADRA2A promotes the classical/progenitor subtype and reduces disease aggressiveness of pancreatic cancer. DOI 10.1093/carcin/bgae056↗
- Jochen Gaedcke · 6 shared papers, 2022–2024 · strength 1.03
- Paper ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic re… DOI 10.1002/ijc.34960↗
- Paper LMO3 is a suppressor of the basal-like/squamous subtype and reduces disease aggressiveness of pancreatic cancer through… DOI 10.1093/carcin/bgae011↗
- Paper ADRA2A promotes the classical/progenitor subtype and reduces disease aggressiveness of pancreatic cancer. DOI 10.1093/carcin/bgae056↗
- Azadeh Azizian · 6 shared papers, 2022–2024 · strength 1.03
- Paper ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic re… DOI 10.1002/ijc.34960↗
- Paper LMO3 is a suppressor of the basal-like/squamous subtype and reduces disease aggressiveness of pancreatic cancer through… DOI 10.1093/carcin/bgae011↗
- Paper ADRA2A promotes the classical/progenitor subtype and reduces disease aggressiveness of pancreatic cancer. DOI 10.1093/carcin/bgae056↗
- Michael B. Ghadimi · 5 shared papers, 2023–2024 · strength 0.90
- Paper ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic re… DOI 10.1002/ijc.34960↗
- Paper LMO3 is a suppressor of the basal-like/squamous subtype and reduces disease aggressiveness of pancreatic cancer through… DOI 10.1093/carcin/bgae011↗
- Paper ADRA2A promotes the classical/progenitor subtype and reduces disease aggressiveness of pancreatic cancer. DOI 10.1093/carcin/bgae056↗
- S. S. Yang · 3 shared papers, 2024 · strength 0.56
- Paper LMO3 is a suppressor of the basal-like/squamous subtype and reduces disease aggressiveness of pancreatic cancer through… DOI 10.1093/carcin/bgae011↗
- Paper ADRA2A promotes the classical/progenitor subtype and reduces disease aggressiveness of pancreatic cancer. DOI 10.1093/carcin/bgae056↗
- Paper MIF/NR3C2 axis regulates glucose metabolism reprogramming in pancreatic cancer through MAPK-ERK and AP-1 pathways. DOI 10.1093/carcin/bgae025↗
- Shouhui Yang · 3 shared papers, 2022–2024 · strength 0.47
- Paper ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic re… DOI 10.1002/ijc.34960↗
- Paper SERPINB3-MYC axis induces the basal-like/squamous subtype and enhances disease progression in pancreatic cancer. DOI 10.1016/j.celrep.2023.113434↗
- Paper Dysregulation of HNF1B/Clusterin axis enhances disease progression in a highly aggressive subset of pancreatic cancer p… DOI 10.1093/carcin/bgac092↗
- Daniel H. Ryan · 1 shared paper, 2026 · strength 0.33
- Christine Alewine · 1 shared paper, 2026 · strength 0.33
- Lin Zhang · 1 shared paper, 2024 · strength 0.18
- Jianjun Gao · 1 shared paper, 2024 · strength 0.18
- Subrata Sen · 1 shared paper, 2024 · strength 0.18
- Nagireddy Putluri · 1 shared paper, 2024 · strength 0.18
- Β. Michael Ghadimi · 1 shared paper, 2022 · strength 0.13
- Lin Zhang · 1 shared paper, 2022 · strength 0.13
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.