ResearcherUNSW SydneyActive in the last 2 years
John Kokkinos
Matched on ORCID iD 0000-0002-9819-2404; OpenAlex author ID A5081491851. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 24
2020: 1 paper · 2021: 2 papers · 2022: 1 paper · 2023: none · 2024: none · 2025: 1 paper · 2026: 2 papers
Papers 7
- HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. Co-author DOI 10.1038/s41388-026-03865-y↗
- βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. First author DOI 10.1038/s41419-026-08657-6↗
- MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. Co-author DOI 10.1016/j.neo.2025.101138↗
- βIII-Tubulin is a Brake on Extrinsic Cell-Death in Pancreatic Cancer Co-author DOI 10.1101/2022.09.29.510034↗
- Cancer-Associated Fibroblasts in Pancreatic Ductal Adenocarcinoma Determine Response to SLC7A11 Inhibition. Co-author DOI 10.1158/0008-5472.can-20-2496↗
- Does the Microenvironment Hold the Hidden Key for Functional Precision Medicine in Pancreatic Cancer? Co-author DOI 10.3390/cancers13102427↗
- Targeting the undruggable in pancreatic cancer using nano-based gene silencing drugs. Co-author DOI 10.1016/j.biomaterials.2019.119742↗
Closest collaborators
The 24 strongest of 24 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- David Goldstein · 7 shared papers, 2020–2026 · strength 1.05
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Joshua A. McCarroll · 7 shared papers, 2020–2026 · strength 1.05
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Phoebe A. Phillips · 7 shared papers, 2020–2026 · strength 1.05
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- George Sharbeen · 7 shared papers, 2020–2026 · strength 1.05
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Koroush Shahsavar Haghighi · 6 shared papers, 2021–2026 · strength 0.93
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Amber L. Johns · 5 shared papers, 2021–2026 · strength 0.79
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Mert Erkan · 5 shared papers, 2021–2026 · strength 0.79
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Anthony James Gill · 5 shared papers, 2021–2026 · strength 0.79
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Val Gebski · 4 shared papers, 2021–2026 · strength 0.59
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper βIII-Tubulin is a Brake on Extrinsic Cell-Death in Pancreatic Cancer DOI 10.1101/2022.09.29.510034↗
- Jennifer P. Morton · 4 shared papers, 2021–2026 · strength 0.59
- Paper HSP47 is a potential dual cell target and prognostic factor in pancreatic cancer. DOI 10.1038/s41388-026-03865-y↗
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper βIII-Tubulin is a Brake on Extrinsic Cell-Death in Pancreatic Cancer DOI 10.1101/2022.09.29.510034↗
- Marina Pajic · 4 shared papers, 2021–2026 · strength 0.58
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Paper βIII-Tubulin is a Brake on Extrinsic Cell-Death in Pancreatic Cancer DOI 10.1101/2022.09.29.510034↗
- Australian Pancreatic Cancer Genome Initiative · 3 shared papers, 2020–2026 · strength 0.50
- Paper βIII-tubulin can act as a brake on extrinsic apoptosis in pancreatic cancer. DOI 10.1038/s41419-026-08657-6↗
- Paper MYH knockdown in pancreatic cancer cells creates an exploitable DNA repair vulnerability. DOI 10.1016/j.neo.2025.101138↗
- Paper Targeting the undruggable in pancreatic cancer using nano-based gene silencing drugs. DOI 10.1016/j.biomaterials.2019.119742↗
- Minoti Vivek Apte · 2 shared papers, 2021–2026 · strength 0.29
- Paul Timpson · 2 shared papers, 2021–2026 · strength 0.29
- Kendelle Joan Murphy · 1 shared paper, 2026 · strength 0.21
- James Ephraums · 1 shared paper, 2025 · strength 0.19
- Angela S. Y. Chou · 1 shared paper, 2021 · strength 0.08
- Thomas R. Cox · 1 shared paper, 2021 · strength 0.08
- Nigel A. Turner · 1 shared paper, 2021 · strength 0.08
- Jie Liu · 1 shared paper, 2021 · strength 0.08
- Jessica L. Chitty · 1 shared paper, 2021 · strength 0.08
- Brooke Pereira · 1 shared paper, 2021 · strength 0.08
- Sigrid K. Fey · 1 shared paper, 2021 · strength 0.08
- Owen James Sansom · 1 shared paper, 2021 · strength 0.08
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.