Arun Kumar Selvam

Closest collaborators

The 9 strongest of 9 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

1. Mikael Björnstedt · 6 shared papers, 2022–2026 · strength 1.66
   • Paper Se-methylselenocysteine induces NRF2-ATF4 stress signaling and ferroptosis-associated remodeling in pancreatic cancer. DOI 10.1016/j.freeradbiomed.2026.08.003↗
   • Paper Transcriptomic profiling reveals complement activation in chronic pancreatitis adjacent to pancreatic ductal adenocarci… DOI 10.1016/j.imbio.2026.153239↗
   • Paper Investigating the Molecular Impact of GGMSC on Redox and Metabolic Pathways in Pancreatic Cancer Cells. DOI 10.3390/antiox14101163↗
2. Joakim Dillner · 5 shared papers, 2022–2026 · strength 1.45
   • Paper Se-methylselenocysteine induces NRF2-ATF4 stress signaling and ferroptosis-associated remodeling in pancreatic cancer. DOI 10.1016/j.freeradbiomed.2026.08.003↗
   • Paper Transcriptomic profiling reveals complement activation in chronic pancreatitis adjacent to pancreatic ductal adenocarci… DOI 10.1016/j.imbio.2026.153239↗
   • Paper Investigating the Molecular Impact of GGMSC on Redox and Metabolic Pathways in Pancreatic Cancer Cells. DOI 10.3390/antiox14101163↗
3. Mehran Ghaderi · 5 shared papers, 2022–2026 · strength 1.45
   • Paper Se-methylselenocysteine induces NRF2-ATF4 stress signaling and ferroptosis-associated remodeling in pancreatic cancer. DOI 10.1016/j.freeradbiomed.2026.08.003↗
   • Paper Transcriptomic profiling reveals complement activation in chronic pancreatitis adjacent to pancreatic ductal adenocarci… DOI 10.1016/j.imbio.2026.153239↗
   • Paper Investigating the Molecular Impact of GGMSC on Redox and Metabolic Pathways in Pancreatic Cancer Cells. DOI 10.3390/antiox14101163↗
4. Carlos Fernández Moro · 4 shared papers, 2022–2026 · strength 0.90
   • Paper Transcriptomic profiling reveals complement activation in chronic pancreatitis adjacent to pancreatic ductal adenocarci… DOI 10.1016/j.imbio.2026.153239↗
   • Paper Exploration of Patient-Derived Pancreatic Ductal Adenocarcinoma Ex Vivo Tissue for Treatment Response. DOI 10.3390/antiox12010167↗
   • Paper An anti-CEA affibody showing high-definition staining in human pancreatic cancer tissue sections and selective tumor ta… DOI 10.1016/j.tranon.2025.102512↗
5. Tímea Szekerczés · 2 shared papers, 2023–2025 · strength 0.41
   • Paper Exploration of Patient-Derived Pancreatic Ductal Adenocarcinoma Ex Vivo Tissue for Treatment Response. DOI 10.3390/antiox12010167↗
   • Paper An anti-CEA affibody showing high-definition staining in human pancreatic cancer tissue sections and selective tumor ta… DOI 10.1016/j.tranon.2025.102512↗
6. Béla Bozóky · 2 shared papers, 2022–2025 · strength 0.36
   • Paper An anti-CEA affibody showing high-definition staining in human pancreatic cancer tissue sections and selective tumor ta… DOI 10.1016/j.tranon.2025.102512↗
   • Paper Drug-induced tumor-specific cytotoxicity in a whole tissue ex vivo model of human pancreatic ductal adenocarcinoma. DOI 10.3389/fonc.2022.965182↗
7. Johan Nilvebrant · 1 shared paper, 2025 · strength 0.20
   • Paper An anti-CEA affibody showing high-definition staining in human pancreatic cancer tissue sections and selective tumor ta… DOI 10.1016/j.tranon.2025.102512↗
8. Per‐Åke Nygren · 1 shared paper, 2025 · strength 0.20
   • Paper An anti-CEA affibody showing high-definition staining in human pancreatic cancer tissue sections and selective tumor ta… DOI 10.1016/j.tranon.2025.102512↗
9. Marco Gerling · 1 shared paper, 2022 · strength 0.16
   • Paper Drug-induced tumor-specific cytotoxicity in a whole tissue ex vivo model of human pancreatic ductal adenocarcinoma. DOI 10.3389/fonc.2022.965182↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.