ResearcherShanghai Medical College of Fudan UniversityActive in the last 2 years
Tong Li
Matched on ORCID iD 0000-0002-9987-8256; OpenAlex author ID A5100359044. Sources: OpenAlex.
Their work
- Papers
- 9
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 33
2020: 4 papers · 2021: 3 papers · 2022: 1 paper · 2023: none · 2024: 1 paper
Papers 9
- Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvironment. Co-author DOI 10.1016/j.canlet.2024.217289↗
- Up-Regulated MISP Is Associated With Poor Prognosis and Immune Infiltration in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3389/fonc.2022.827051↗
- Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. Co-author DOI 10.1007/s00262-020-02816-0↗
- The promoting effects of hsa_circ_0050102 in pancreatic cancer and the molecular mechanism by targeting miR-1182/NPSR1. Co-author DOI 10.1093/carcin/bgaa130↗
- Immune subtyping for pancreatic cancer with implication in clinical outcomes and improving immunotherapy. Co-author DOI 10.1186/s12935-021-01824-z↗
- Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor microenvironment. Co-author DOI 10.1007/s00262-020-02542-7↗
- Up-regulation of CDHR5 expression promotes malignant phenotype of pancreatic ductal adenocarcinoma. Co-author DOI 10.1111/jcmm.15856↗
- Identification of prognosis-related genes and construction of multi-regulatory networks in pancreatic cancer microenvironment by bioinformatics analysis. Co-author DOI 10.1186/s12935-020-01426-1↗
- CXCL5 overexpression predicts a poor prognosis in pancreatic ductal adenocarcinoma and is correlated with immune cell infiltration. Co-author DOI 10.7150/jca.40517↗
Closest collaborators
The 25 strongest of 33 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Qiaofei Liu · 5 shared papers, 2020–2024 · strength 0.71
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper Immune subtyping for pancreatic cancer with implication in clinical outcomes and improving immunotherapy. DOI 10.1186/s12935-021-01824-z↗
- Paper Identification of prognosis-related genes and construction of multi-regulatory networks in pancreatic cancer microenvir… DOI 10.1186/s12935-020-01426-1↗
- Junyi Gao · 5 shared papers, 2020–2024 · strength 0.65
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper The promoting effects of hsa_circ_0050102 in pancreatic cancer and the molecular mechanism by targeting miR-1182/NPSR1. DOI 10.1093/carcin/bgaa130↗
- Paper Up-regulation of CDHR5 expression promotes malignant phenotype of pancreatic ductal adenocarcinoma. DOI 10.1111/jcmm.15856↗
- Ming Cui · 5 shared papers, 2020–2024 · strength 0.63
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper Immune subtyping for pancreatic cancer with implication in clinical outcomes and improving immunotherapy. DOI 10.1186/s12935-021-01824-z↗
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Ronghua Zhang · 5 shared papers, 2020–2024 · strength 0.63
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper Identification of prognosis-related genes and construction of multi-regulatory networks in pancreatic cancer microenvir… DOI 10.1186/s12935-020-01426-1↗
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Quan Liao · 5 shared papers, 2020–2021 · strength 0.58
- Paper The promoting effects of hsa_circ_0050102 in pancreatic cancer and the molecular mechanism by targeting miR-1182/NPSR1. DOI 10.1093/carcin/bgaa130↗
- Paper Up-regulation of CDHR5 expression promotes malignant phenotype of pancreatic ductal adenocarcinoma. DOI 10.1111/jcmm.15856↗
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Quan Liao · 3 shared papers, 2020–2024 · strength 0.47
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper Immune subtyping for pancreatic cancer with implication in clinical outcomes and improving immunotherapy. DOI 10.1186/s12935-021-01824-z↗
- Paper Identification of prognosis-related genes and construction of multi-regulatory networks in pancreatic cancer microenvir… DOI 10.1186/s12935-020-01426-1↗
- Xiang Zhang · 4 shared papers, 2020–2021 · strength 0.46
- Paper Immune subtyping for pancreatic cancer with implication in clinical outcomes and improving immunotherapy. DOI 10.1186/s12935-021-01824-z↗
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Paper CXCL5 overexpression predicts a poor prognosis in pancreatic ductal adenocarcinoma and is correlated with immune cell i… DOI 10.7150/jca.40517↗
- Jingkai Liu · 4 shared papers, 2020–2021 · strength 0.46
- Paper Immune subtyping for pancreatic cancer with implication in clinical outcomes and improving immunotherapy. DOI 10.1186/s12935-021-01824-z↗
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Paper CXCL5 overexpression predicts a poor prognosis in pancreatic ductal adenocarcinoma and is correlated with immune cell i… DOI 10.7150/jca.40517↗
- Yalu Zhang · 3 shared papers, 2021–2024 · strength 0.44
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper Immune subtyping for pancreatic cancer with implication in clinical outcomes and improving immunotherapy. DOI 10.1186/s12935-021-01824-z↗
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Mengyi Wang · 3 shared papers, 2020–2021 · strength 0.38
- Paper The promoting effects of hsa_circ_0050102 in pancreatic cancer and the molecular mechanism by targeting miR-1182/NPSR1. DOI 10.1093/carcin/bgaa130↗
- Paper Up-regulation of CDHR5 expression promotes malignant phenotype of pancreatic ductal adenocarcinoma. DOI 10.1111/jcmm.15856↗
- Paper CXCL5 overexpression predicts a poor prognosis in pancreatic ductal adenocarcinoma and is correlated with immune cell i… DOI 10.7150/jca.40517↗
- Yupei Zhao · 3 shared papers, 2020 · strength 0.34
- Paper Identification of prognosis-related genes and construction of multi-regulatory networks in pancreatic cancer microenvir… DOI 10.1186/s12935-020-01426-1↗
- Paper CXCL5 overexpression predicts a poor prognosis in pancreatic ductal adenocarcinoma and is correlated with immune cell i… DOI 10.7150/jca.40517↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Sen Yang · 2 shared papers, 2021–2024 · strength 0.29
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Junya Peng · 2 shared papers, 2020–2024 · strength 0.28
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper CXCL5 overexpression predicts a poor prognosis in pancreatic ductal adenocarcinoma and is correlated with immune cell i… DOI 10.7150/jca.40517↗
- Lei You · 2 shared papers, 2020–2021 · strength 0.27
- Jorg H. Kleeff · 2 shared papers, 2020–2024 · strength 0.26
- Paper Pancreatic cancer cell-derived migrasomes promote cancer progression by fostering an immunosuppressive tumor microenvir… DOI 10.1016/j.canlet.2024.217289↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Qiaofei Liu · 2 shared papers, 2020–2021 · strength 0.20
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Huanwen Wu · 2 shared papers, 2020–2021 · strength 0.20
- Paper Dopamine improves chemotherapeutic efficacy for pancreatic cancer by regulating macrophage-derived inflammations. DOI 10.1007/s00262-020-02816-0↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Xinyang Huang · 1 shared paper, 2022 · strength 0.16
- Liangchao Zhao · 1 shared paper, 2022 · strength 0.16
- Lifu Wang · 1 shared paper, 2022 · strength 0.16
- Hui Xu · 1 shared paper, 2022 · strength 0.16
- Zhuoxin Wang · 1 shared paper, 2022 · strength 0.16
- Yixun Jin · 1 shared paper, 2022 · strength 0.16
- Qi Wang · 1 shared paper, 2022 · strength 0.16
- Surong Hua · 1 shared paper, 2021 · strength 0.14
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.