ResearcherJohns Hopkins UniversityNo recent work listed
Zhengdong Jiang
Matched on ORCID iD 0000-0003-0234-5478; OpenAlex author ID A5000195252. Sources: OpenAlex.
Their work
- Papers
- 25 (first author 4, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 55
2015: 4 papers · 2016: 5 papers · 2017: 4 papers · 2018: 8 papers · 2019: 3 papers · 2020: none · 2021: none · 2022: none · 2023: 1 paper
Papers 25
- Mutant p53 driven-LINC00857, a protein scaffold between FOXM1 and deubiquitinase OTUB1, promotes the metastasis of pancreatic cancer. Co-author DOI 10.1016/j.canlet.2022.215976↗
- Histomorphology of pancreatic cancer in patients with inherited ATM serine/threonine kinase pathogenic variants. First author DOI 10.1038/s41379-019-0317-6↗
- Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cancer cells via targeting SREBP1. Co-author DOI 10.1111/cpr.12514↗
- High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. Co-author DOI 10.1186/s13046-019-1288-7↗
- Metformin suppresses tumor angiogenesis and enhances the chemosensitivity of gemcitabine in a genetically engineered mouse model of pancreatic cancer. Co-author DOI 10.1016/j.lfs.2018.07.046↗
- Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of YAP/HIF-1α. First author DOI 10.1016/j.yexcr.2018.07.041↗
- Resveratrol-Induced Downregulation of NAF-1 Enhances the Sensitivity of Pancreatic Cancer Cells to Gemcitabine via the ROS/Nrf2 Signaling Pathways. Co-author DOI 10.1155/2018/9482018↗
- Activation of Nrf2 by Sulforaphane Inhibits High Glucose-Induced Progression of Pancreatic Cancer via AMPK Dependent Signaling. First author DOI 10.1159/000494547↗
- Inhibiting YAP expression suppresses pancreatic cancer progression by disrupting tumor-stromal interactions. Co-author DOI 10.1186/s13046-018-0740-4↗
- Curcumin inhibits superoxide dismutase-induced epithelial-to-mesenchymal transition via the PI3K/Akt/NF-κB pathway in pancreatic cancer cells. First author DOI 10.3892/ijo.2018.4295↗
- Itraconazole inhibits invasion and migration of pancreatic cancer cells by suppressing TGF-β/SMAD2/3 signaling. Co-author DOI 10.3892/or.2018.6281↗
- Norepinephrine enhances cell viability and invasion, and inhibits apoptosis of pancreatic cancer cells in a Notch‑1‑dependent manner. Co-author DOI 10.3892/or.2018.6696↗
- Loss of AMPK activation promotes the invasion and metastasis of pancreatic cancer through an HSF1-dependent pathway. Co-author DOI 10.1002/1878-0261.12116↗
- Desmoplasia suppression by metformin-mediated AMPK activation inhibits pancreatic cancer progression. Co-author DOI 10.1016/j.canlet.2016.10.019↗
- Metformin suppresses cancer initiation and progression in genetic mouse models of pancreatic cancer. Co-author DOI 10.1186/s12943-017-0701-0↗
- Lipoxin A4 reverses mesenchymal phenotypes to attenuate invasion and metastasis via the inhibition of autocrine TGF-β1 signaling in pancreatic cancer. Co-author DOI 10.1186/s13046-017-0655-5↗
- Reactive Oxygen Species and Targeted Therapy for Pancreatic Cancer. Co-author DOI 10.1155/2016/1616781↗
- The Relevance of Nrf2 Pathway and Autophagy in Pancreatic Cancer Cells upon Stimulation of Reactive Oxygen Species. Co-author DOI 10.1155/2016/3897250↗
- Hyperglycemia Promotes the Epithelial-Mesenchymal Transition of Pancreatic Cancer via Hydrogen Peroxide. Co-author DOI 10.1155/2016/5190314↗
- β2-adrenogenic signaling regulates NNK-induced pancreatic cancer progression via upregulation of HIF-1α. Co-author DOI 10.18632/oncotarget.5677↗
- YAP Inhibition by Resveratrol via Activation of AMPK Enhances the Sensitivity of Pancreatic Cancer Cells to Gemcitabine. Co-author DOI 10.3390/nu8100546↗
- Resveratrol in the treatment of pancreatic cancer. Co-author DOI 10.1111/nyas.12837↗
- Overexpression of Nodal induces a metastatic phenotype in pancreatic cancer cells via the Smad2/3 pathway. Co-author DOI 10.18632/oncotarget.2686↗
- Ginkgolic acid suppresses the development of pancreatic cancer by inhibiting pathways driving lipogenesis. Co-author DOI 10.18632/oncotarget.3663↗
- miR-221/222 induces pancreatic cancer progression through the regulation of matrix metalloproteinases. Co-author DOI 10.18632/oncotarget.3686↗
Closest collaborators
The 25 strongest of 55 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Qingyong Ma · 23 shared papers, 2015–2023 · strength 1.58
- Paper Mutant p53 driven-LINC00857, a protein scaffold between FOXM1 and deubiquitinase OTUB1, promotes the metastasis of panc… DOI 10.1016/j.canlet.2022.215976↗
- Paper Curcumin inhibits superoxide dismutase-induced epithelial-to-mesenchymal transition via the PI3K/Akt/NF-κB pathway in p… DOI 10.3892/ijo.2018.4295↗
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Wanxing Duan · 18 shared papers, 2015–2019 · strength 1.10
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Metformin suppresses tumor angiogenesis and enhances the chemosensitivity of gemcitabine in a genetically engineered mo… DOI 10.1016/j.lfs.2018.07.046↗
- Cancan Zhou · 13 shared papers, 2016–2023 · strength 1.01
- Paper Mutant p53 driven-LINC00857, a protein scaffold between FOXM1 and deubiquitinase OTUB1, promotes the metastasis of panc… DOI 10.1016/j.canlet.2022.215976↗
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Weikun Qian · 10 shared papers, 2017–2023 · strength 0.82
- Paper Mutant p53 driven-LINC00857, a protein scaffold between FOXM1 and deubiquitinase OTUB1, promotes the metastasis of panc… DOI 10.1016/j.canlet.2022.215976↗
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Jiguang Ma · 13 shared papers, 2015–2019 · strength 0.79
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Activation of Nrf2 by Sulforaphane Inhibits High Glucose-Induced Progression of Pancreatic Cancer via AMPK Dependent Si… DOI 10.1159/000494547↗
- Xuqi Li · 13 shared papers, 2015–2019 · strength 0.75
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Activation of Nrf2 by Sulforaphane Inhibits High Glucose-Induced Progression of Pancreatic Cancer via AMPK Dependent Si… DOI 10.1159/000494547↗
- Zheng Wang · 11 shared papers, 2015–2023 · strength 0.74
- Paper Mutant p53 driven-LINC00857, a protein scaffold between FOXM1 and deubiquitinase OTUB1, promotes the metastasis of panc… DOI 10.1016/j.canlet.2022.215976↗
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Paper Activation of Nrf2 by Sulforaphane Inhibits High Glucose-Induced Progression of Pancreatic Cancer via AMPK Dependent Si… DOI 10.1159/000494547↗
- Liang Cheng · 10 shared papers, 2017–2019 · strength 0.73
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Metformin suppresses tumor angiogenesis and enhances the chemosensitivity of gemcitabine in a genetically engineered mo… DOI 10.1016/j.lfs.2018.07.046↗
- Zheng Wu · 8 shared papers, 2015–2023 · strength 0.61
- Paper Mutant p53 driven-LINC00857, a protein scaffold between FOXM1 and deubiquitinase OTUB1, promotes the metastasis of panc… DOI 10.1016/j.canlet.2022.215976↗
- Paper Curcumin inhibits superoxide dismutase-induced epithelial-to-mesenchymal transition via the PI3K/Akt/NF-κB pathway in p… DOI 10.3892/ijo.2018.4295↗
- Paper High glucose microenvironment accelerates tumor growth via SREBP1-autophagy axis in pancreatic cancer. DOI 10.1186/s13046-019-1288-7↗
- Liankang Sun · 9 shared papers, 2016–2019 · strength 0.59
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of Y… DOI 10.1016/j.yexcr.2018.07.041↗
- Paper Resveratrol-Induced Downregulation of NAF-1 Enhances the Sensitivity of Pancreatic Cancer Cells to Gemcitabine via the … DOI 10.1155/2018/9482018↗
- Bin Yan · 8 shared papers, 2017–2019 · strength 0.57
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Metformin suppresses tumor angiogenesis and enhances the chemosensitivity of gemcitabine in a genetically engineered mo… DOI 10.1016/j.lfs.2018.07.046↗
- Paper Norepinephrine enhances cell viability and invasion, and inhibits apoptosis of pancreatic cancer cells in a Notch‑1‑dep… DOI 10.3892/or.2018.6696↗
- Ke Chen · 8 shared papers, 2016–2018 · strength 0.54
- Paper Metformin suppresses tumor angiogenesis and enhances the chemosensitivity of gemcitabine in a genetically engineered mo… DOI 10.1016/j.lfs.2018.07.046↗
- Paper Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of Y… DOI 10.1016/j.yexcr.2018.07.041↗
- Paper Resveratrol-Induced Downregulation of NAF-1 Enhances the Sensitivity of Pancreatic Cancer Cells to Gemcitabine via the … DOI 10.1155/2018/9482018↗
- Jianjun Lei · 10 shared papers, 2015–2018 · strength 0.52
- Paper Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of Y… DOI 10.1016/j.yexcr.2018.07.041↗
- Paper Inhibiting YAP expression suppresses pancreatic cancer progression by disrupting tumor-stromal interactions. DOI 10.1186/s13046-018-0740-4↗
- Paper Lipoxin A4 reverses mesenchymal phenotypes to attenuate invasion and metastasis via the inhibition of autocrine TGF-β1 … DOI 10.1186/s13046-017-0655-5↗
- Qinhong Xu · 10 shared papers, 2015–2018 · strength 0.52
- Paper Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of Y… DOI 10.1016/j.yexcr.2018.07.041↗
- Paper Inhibiting YAP expression suppresses pancreatic cancer progression by disrupting tumor-stromal interactions. DOI 10.1186/s13046-018-0740-4↗
- Paper Lipoxin A4 reverses mesenchymal phenotypes to attenuate invasion and metastasis via the inhibition of autocrine TGF-β1 … DOI 10.1186/s13046-017-0655-5↗
- Junyu Cao · 7 shared papers, 2017–2019 · strength 0.50
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Metformin suppresses tumor angiogenesis and enhances the chemosensitivity of gemcitabine in a genetically engineered mo… DOI 10.1016/j.lfs.2018.07.046↗
- Paper Norepinephrine enhances cell viability and invasion, and inhibits apoptosis of pancreatic cancer cells in a Notch‑1‑dep… DOI 10.3892/or.2018.6696↗
- Liang Zong · 7 shared papers, 2015–2018 · strength 0.39
- Paper Hyperglycemia Promotes the Epithelial-Mesenchymal Transition of Pancreatic Cancer via Hydrogen Peroxide. DOI 10.1155/2016/5190314↗
- Paper Inhibiting YAP expression suppresses pancreatic cancer progression by disrupting tumor-stromal interactions. DOI 10.1186/s13046-018-0740-4↗
- Paper Lipoxin A4 reverses mesenchymal phenotypes to attenuate invasion and metastasis via the inhibition of autocrine TGF-β1 … DOI 10.1186/s13046-017-0655-5↗
- Xin Chen · 5 shared papers, 2015–2018 · strength 0.30
- Paper Activation of Nrf2 by Sulforaphane Inhibits High Glucose-Induced Progression of Pancreatic Cancer via AMPK Dependent Si… DOI 10.1159/000494547↗
- Paper Inhibiting YAP expression suppresses pancreatic cancer progression by disrupting tumor-stromal interactions. DOI 10.1186/s13046-018-0740-4↗
- Paper Lipoxin A4 reverses mesenchymal phenotypes to attenuate invasion and metastasis via the inhibition of autocrine TGF-β1 … DOI 10.1186/s13046-017-0655-5↗
- Ligang Nan · 6 shared papers, 2015–2016 · strength 0.27
- Paper Reactive Oxygen Species and Targeted Therapy for Pancreatic Cancer. DOI 10.1155/2016/1616781↗
- Paper β2-adrenogenic signaling regulates NNK-induced pancreatic cancer progression via upregulation of HIF-1α. DOI 10.18632/oncotarget.5677↗
- Paper Overexpression of Nodal induces a metastatic phenotype in pancreatic cancer cells via the Smad2/3 pathway. DOI 10.18632/oncotarget.2686↗
- Erxi Wu · 5 shared papers, 2015–2019 · strength 0.27
- Paper Resveratrol enhances the chemotherapeutic response and reverses the stemness induced by gemcitabine in pancreatic cance… DOI 10.1111/cpr.12514↗
- Paper Desmoplasia suppression by metformin-mediated AMPK activation inhibits pancreatic cancer progression. DOI 10.1016/j.canlet.2016.10.019↗
- Paper β2-adrenogenic signaling regulates NNK-induced pancreatic cancer progression via upregulation of HIF-1α. DOI 10.18632/oncotarget.5677↗
- Jie Li · 4 shared papers, 2017–2018 · strength 0.27
- Paper Paracrine HGF/c-MET enhances the stem cell-like potential and glycolysis of pancreatic cancer cells via activation of Y… DOI 10.1016/j.yexcr.2018.07.041↗
- Paper Resveratrol-Induced Downregulation of NAF-1 Enhances the Sensitivity of Pancreatic Cancer Cells to Gemcitabine via the … DOI 10.1155/2018/9482018↗
- Paper Metformin suppresses cancer initiation and progression in genetic mouse models of pancreatic cancer. DOI 10.1186/s12943-017-0701-0↗
- Wei Li · 4 shared papers, 2016–2018 · strength 0.26
- Paper Curcumin inhibits superoxide dismutase-induced epithelial-to-mesenchymal transition via the PI3K/Akt/NF-κB pathway in p… DOI 10.3892/ijo.2018.4295↗
- Paper Hyperglycemia Promotes the Epithelial-Mesenchymal Transition of Pancreatic Cancer via Hydrogen Peroxide. DOI 10.1155/2016/5190314↗
- Paper Reactive Oxygen Species and Targeted Therapy for Pancreatic Cancer. DOI 10.1155/2016/1616781↗
- Lun Zhang · 4 shared papers, 2015–2016 · strength 0.21
- Paper Hyperglycemia Promotes the Epithelial-Mesenchymal Transition of Pancreatic Cancer via Hydrogen Peroxide. DOI 10.1155/2016/5190314↗
- Paper Reactive Oxygen Species and Targeted Therapy for Pancreatic Cancer. DOI 10.1155/2016/1616781↗
- Paper The Relevance of Nrf2 Pathway and Autophagy in Pancreatic Cancer Cells upon Stimulation of Reactive Oxygen Species. DOI 10.1155/2016/3897250↗
- Zhenhua Ma · 4 shared papers, 2015–2017 · strength 0.21
- Paper Loss of AMPK activation promotes the invasion and metastasis of pancreatic cancer through an HSF1-dependent pathway. DOI 10.1002/1878-0261.12116↗
- Paper Desmoplasia suppression by metformin-mediated AMPK activation inhibits pancreatic cancer progression. DOI 10.1016/j.canlet.2016.10.019↗
- Paper Reactive Oxygen Species and Targeted Therapy for Pancreatic Cancer. DOI 10.1155/2016/1616781↗
- Simei Zhang · 1 shared paper, 2023 · strength 0.16
- Wunai Zhang · 1 shared paper, 2023 · strength 0.16
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.