ResearcherStanford MedicineActive in the last 2 years
Michael Thornton Longaker
Matched on ORCID iD 0000-0003-1430-8914; OpenAlex author ID A5005728573. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 0, last author 3)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 14
2020: 1 paper · 2021: none · 2022: none · 2023: 2 papers · 2024: 1 paper · 2025: 3 papers · 2026: 2 papers
Papers 9
- NFAT mediates pro-tumorigenic inflammation in cancer-associated fibroblasts in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.celrep.2025.116849↗
- Protocol for orthotopic implantation of a collagen hydrogel to model pancreatic ductal adenocarcinoma in mice. Co-author DOI 10.1016/j.xpro.2025.104337↗
- Disruption of fibroblast MYD88 signaling promotes antitumor immunity in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.celrep.2025.116347↗
- Precision Medicine: IL-1RA and Pancreatic Cancer Organoids. Co-author DOI 10.3390/biology14060604↗
- Correction: Morgan et al. Precision Medicine: IL-1RA and Pancreatic Cancer Organoids. Biology 2025, 14, 604. Co-author DOI 10.3390/biology14091133↗
- Hematoxylin and Eosin Architecture Uncovers Clinically Divergent Niches in Pancreatic Cancer. Last author DOI 10.1089/ten.tea.2024.0039↗
- Desmoplastic stromal signatures predict patient outcomes in pancreatic ductal adenocarcinoma. Last author DOI 10.1016/j.xcrm.2023.101248↗
- Medical Biology of Cancer-Associated Fibroblasts in Pancreatic Cancer. Co-author DOI 10.3390/biology12081044↗
- Pancreatic Cancer Associated Fibroblasts (CAF): Under-Explored Target for Pancreatic Cancer Treatment. Last author DOI 10.3390/cancers12051347↗
Closest collaborators
The 14 strongest of 14 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jeffrey Allen Norton · 8 shared papers, 2020–2026 · strength 1.80
- Paper Precision Medicine: IL-1RA and Pancreatic Cancer Organoids. DOI 10.3390/biology14060604↗
- Paper Correction: Morgan et al. Precision Medicine: IL-1RA and Pancreatic Cancer Organoids. Biology 2025, 14, 604. DOI 10.3390/biology14091133↗
- Paper Medical Biology of Cancer-Associated Fibroblasts in Pancreatic Cancer. DOI 10.3390/biology12081044↗
- Annah Grace Morgan · 4 shared papers, 2023–2026 · strength 1.17
- Paper Precision Medicine: IL-1RA and Pancreatic Cancer Organoids. DOI 10.3390/biology14060604↗
- Paper Correction: Morgan et al. Precision Medicine: IL-1RA and Pancreatic Cancer Organoids. Biology 2025, 14, 604. DOI 10.3390/biology14091133↗
- Paper Medical Biology of Cancer-Associated Fibroblasts in Pancreatic Cancer. DOI 10.3390/biology12081044↗
- Deshka S. Foster · 6 shared papers, 2020–2026 · strength 1.12
- Paper Protocol for orthotopic implantation of a collagen hydrogel to model pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.xpro.2025.104337↗
- Paper NFAT mediates pro-tumorigenic inflammation in cancer-associated fibroblasts in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116849↗
- Paper Disruption of fibroblast MYD88 signaling promotes antitumor immunity in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116347↗
- Daniel Delitto · 5 shared papers, 2023–2026 · strength 0.97
- Paper Protocol for orthotopic implantation of a collagen hydrogel to model pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.xpro.2025.104337↗
- Paper NFAT mediates pro-tumorigenic inflammation in cancer-associated fibroblasts in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116849↗
- Paper Disruption of fibroblast MYD88 signaling promotes antitumor immunity in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116347↗
- Jason L. Guo · 4 shared papers, 2023–2026 · strength 0.70
- Paper NFAT mediates pro-tumorigenic inflammation in cancer-associated fibroblasts in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116849↗
- Paper Disruption of fibroblast MYD88 signaling promotes antitumor immunity in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116347↗
- Paper Hematoxylin and Eosin Architecture Uncovers Clinically Divergent Niches in Pancreatic Cancer. DOI 10.1089/ten.tea.2024.0039↗
- Maria Moozhiyil Korah · 3 shared papers, 2025–2026 · strength 0.66
- Paper Protocol for orthotopic implantation of a collagen hydrogel to model pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.xpro.2025.104337↗
- Paper NFAT mediates pro-tumorigenic inflammation in cancer-associated fibroblasts in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116849↗
- Paper Disruption of fibroblast MYD88 signaling promotes antitumor immunity in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116347↗
- James Paul Agolia · 3 shared papers, 2025–2026 · strength 0.66
- Paper Protocol for orthotopic implantation of a collagen hydrogel to model pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.xpro.2025.104337↗
- Paper NFAT mediates pro-tumorigenic inflammation in cancer-associated fibroblasts in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116849↗
- Paper Disruption of fibroblast MYD88 signaling promotes antitumor immunity in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116347↗
- Chuner Guo · 3 shared papers, 2025–2026 · strength 0.66
- Paper Protocol for orthotopic implantation of a collagen hydrogel to model pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.xpro.2025.104337↗
- Paper NFAT mediates pro-tumorigenic inflammation in cancer-associated fibroblasts in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116849↗
- Paper Disruption of fibroblast MYD88 signaling promotes antitumor immunity in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.116347↗
- George A. Poultsides · 2 shared papers, 2025–2026 · strength 0.39
- Brendan Christopher Visser · 2 shared papers, 2025–2026 · strength 0.39
- Shamik Mascharak · 2 shared papers, 2023–2024 · strength 0.31
- Teri A. Longacre · 2 shared papers, 2023–2024 · strength 0.31
- Byrne Lee · 1 shared paper, 2025 · strength 0.17
- Amanda Kirane · 1 shared paper, 2025 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.