Pancreatic Cancer Research

ResearcherLiaoning Provincial People's HospitalActive in the last 2 years

Qingfeng Liu

Matched on ORCID iD 0000-0003-2492-8092; OpenAlex author ID A5100349237. Sources: OpenAlex.

Their work

Papers
4 (first author 1, last author 0)
Trials
0
Grants
0
Labs led
0
Collaborators
10
Papers, trials and grants by year

2014: 2 papers · 2015: 1 paper · 2016: none · 2017: none · 2018: none · 2019: none · 2020: none · 2021: none · 2022: none · 2023: none · 2024: none · 2025: none · 2026: 1 paper

Papers 4

  1. Dual role of exosomes in pancreatic cancer: Underlying mechanisms and research advances (Review). First author DOI 10.3892/ol.2026.15547↗
  2. Expression of vitamin D receptor as a potential prognostic factor and therapeutic target in pancreatic cancer. Co-author DOI 10.1111/his.12663↗
  3. Overexpression of calreticulin contributes to the development and progression of pancreatic cancer. Co-author DOI 10.1002/jcp.24519↗
  4. [The relationship and clinicopathological significance of Numb,MDM2 and p53 expression in human pancreatic cancer]. Co-author PMID 25410780↗

Closest collaborators

The 10 strongest of 10 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Zhao Jun Guo · 1 shared paper, 2026 · strength 0.63
  2. Weiwei Sheng · 3 shared papers, 2014–2015 · strength 0.15
  3. Ming Dong · 2 shared papers, 2014–2015 · strength 0.10
  4. Qi Dong · 2 shared papers, 2014 · strength 0.09
  5. Jianping Zhou · 2 shared papers, 2014 · strength 0.09
  6. Kewei Wang · 1 shared paper, 2015 · strength 0.05
  7. Qingchang Li · 1 shared paper, 2015 · strength 0.05
  8. Junqiang Wang · 1 shared paper, 2015 · strength 0.05
  9. Feng Li · 1 shared paper, 2014 · strength 0.05
  10. Feng Li · 1 shared paper, 2014 · strength 0.05

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.