ResearcherFirst Affiliated Hospital of Nanchang UniversityActive in the last 2 years
Weidong Xiao
Matched on ORCID iD 0000-0003-3266-6440; OpenAlex author ID A5053954336. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 0, last author 9)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 13
2017: 1 paper · 2018: 1 paper · 2019: 1 paper · 2020: 2 papers · 2021: none · 2022: 1 paper · 2023: 4 papers · 2024: 2 papers
Papers 12
- LINC02086 inhibits ferroptosis and promotes malignant phenotypes of pancreatic cancer via miR-342-3p/CA9 axis. Last author DOI 10.1007/s10142-024-01329-8↗
- Clinical significance of tumor suppressor genes methylation in circulating tumor DNA of patients with pancreatic cancer. Last author DOI 10.1016/j.gene.2023.148078↗
- Comparison of oncologic outcomes between pancreaticoduodenectomy and total pancreatectomy for pancreatic adenocarcinoma. Co-author DOI 10.1007/s00464-022-09441-1↗
- Establishment of a novel signature to predict prognosis and immune characteristics of pancreatic cancer based on necroptosis-related long non-coding RNA. Last author DOI 10.1007/s11033-023-08663-3↗
- Distribution characteristics and clinical significance of infiltrating T cells in the tumor microenvironment of pancreatic cancer. Last author DOI 10.3892/ol.2023.13847↗
- [Retracted] MicroRNA‑148a suppresses epithelial‑mesenchymal transition and invasion of pancreatic cancer cells by targeting Wnt10b and inhibiting the Wnt/β‑cat… Last author DOI 10.3892/or.2023.8493↗
- Hsa_circ_0046523 Mediates an Immunosuppressive Tumor Microenvironment by Regulating MiR-148a-3p/PD-L1 Axis in Pancreatic Cancer. Last author DOI 10.3389/fonc.2022.877376↗
- MicroRNA-148a-3p suppresses epithelial-to-mesenchymal transition and stemness properties via Wnt1-mediated Wnt/β-catenin pathway in pancreatic cancer. Last author DOI 10.1111/jcmm.15900↗
- Downregulation of NEAT1 sensitizes gemcitabine-resistant pancreatic cancer cells to gemcitabine through modulation of the miR-506-3p/ZEB2/EMT axis Co-author DOI 10.21203/rs.3.rs-86053/v1↗
- Long-Term Oncological Outcomes in Laparoscopic Versus Open Pancreaticoduodenectomy for Pancreatic Cancer: A Systematic Review and Meta-Analysis. Co-author DOI 10.1089/lap.2018.0683↗
- The interaction between miR‑148a and DNMT1 suppresses cell migration and invasion by reactivating tumor suppressor genes in pancreatic cancer. Last author DOI 10.3892/or.2018.6700↗
- MicroRNA-148a suppresses epithelial-mesenchymal transition and invasion of pancreatic cancer cells by targeting Wnt10b and inhibiting the Wnt/β-catenin signali… Last author DOI 10.3892/or.2017.5705↗
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Gen Sun · 6 shared papers, 2018–2024 · strength 0.98
- Paper Clinical significance of tumor suppressor genes methylation in circulating tumor DNA of patients with pancreatic cancer. DOI 10.1016/j.gene.2023.148078↗
- Paper Distribution characteristics and clinical significance of infiltrating T cells in the tumor microenvironment of pancrea… DOI 10.3892/ol.2023.13847↗
- Paper Establishment of a novel signature to predict prognosis and immune characteristics of pancreatic cancer based on necrop… DOI 10.1007/s11033-023-08663-3↗
- Yuanpeng Xiong · 4 shared papers, 2023–2024 · strength 0.86
- Paper Clinical significance of tumor suppressor genes methylation in circulating tumor DNA of patients with pancreatic cancer. DOI 10.1016/j.gene.2023.148078↗
- Paper LINC02086 inhibits ferroptosis and promotes malignant phenotypes of pancreatic cancer via miR-342-3p/CA9 axis. DOI 10.1007/s10142-024-01329-8↗
- Paper Distribution characteristics and clinical significance of infiltrating T cells in the tumor microenvironment of pancrea… DOI 10.3892/ol.2023.13847↗
- Wanpeng Xin · 3 shared papers, 2020–2024 · strength 0.58
- Paper Clinical significance of tumor suppressor genes methylation in circulating tumor DNA of patients with pancreatic cancer. DOI 10.1016/j.gene.2023.148078↗
- Paper LINC02086 inhibits ferroptosis and promotes malignant phenotypes of pancreatic cancer via miR-342-3p/CA9 axis. DOI 10.1007/s10142-024-01329-8↗
- Paper MicroRNA-148a-3p suppresses epithelial-to-mesenchymal transition and stemness properties via Wnt1-mediated Wnt/β-cateni… DOI 10.1111/jcmm.15900↗
- Long Peng · 4 shared papers, 2017–2023 · strength 0.47
- Paper [Retracted] MicroRNA‑148a suppresses epithelial‑mesenchymal transition and invasion of pancreatic cancer cells by targe… DOI 10.3892/or.2023.8493↗
- Paper Long-Term Oncological Outcomes in Laparoscopic Versus Open Pancreaticoduodenectomy for Pancreatic Cancer: A Systematic … DOI 10.1089/lap.2018.0683↗
- Paper The interaction between miR‑148a and DNMT1 suppresses cell migration and invasion by reactivating tumor suppressor gene… DOI 10.3892/or.2018.6700↗
- Xiaowei Fu · 3 shared papers, 2020–2022 · strength 0.40
- Paper Hsa_circ_0046523 Mediates an Immunosuppressive Tumor Microenvironment by Regulating MiR-148a-3p/PD-L1 Axis in Pancreati… DOI 10.3389/fonc.2022.877376↗
- Paper Downregulation of NEAT1 sensitizes gemcitabine-resistant pancreatic cancer cells to gemcitabine through modulation of t… DOI 10.21203/rs.3.rs-86053/v1↗
- Paper MicroRNA-148a-3p suppresses epithelial-to-mesenchymal transition and stemness properties via Wnt1-mediated Wnt/β-cateni… DOI 10.1111/jcmm.15900↗
- Jisheng Zhu · 1 shared paper, 2023 · strength 0.22
- Yong Li · 1 shared paper, 2023 · strength 0.22
- Le Hong · 2 shared papers, 2018–2020 · strength 0.20
- Paper MicroRNA-148a-3p suppresses epithelial-to-mesenchymal transition and stemness properties via Wnt1-mediated Wnt/β-cateni… DOI 10.1111/jcmm.15900↗
- Paper The interaction between miR‑148a and DNMT1 suppresses cell migration and invasion by reactivating tumor suppressor gene… DOI 10.3892/or.2018.6700↗
- Yonghao Ouyang · 1 shared paper, 2023 · strength 0.18
- Yeqing Zou · 1 shared paper, 2020 · strength 0.13
- Bo Huang · 1 shared paper, 2020 · strength 0.13
- Xueqiang Deng · 1 shared paper, 2020 · strength 0.13
- Jiaqing Cao · 1 shared paper, 2019 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.