ResearcherFujian Medical UniversityNo recent work listed
Yaodong Wang
Matched on ORCID iD 0000-0003-3308-475X; OpenAlex author ID A5100779967. Sources: OpenAlex.
Their work
- Papers
- 3 (first author 0, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 9
2021: 1 paper · 2022: 1 paper · 2023: 1 paper
Papers 3
- miR-24-3p Regulates Epithelial-Mesenchymal Transition and the Malignant Phenotype of Pancreatic Adenocarcinoma by Regulating ASF1B Expression. Last author DOI 10.1007/s10528-022-10278-5↗
- SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancreatic cancer to gemcitabine. Co-author DOI 10.1016/j.celrep.2022.110813↗
- CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancreatic cancer. Last author DOI 10.1186/s13045-021-01072-8↗
Closest collaborators
The 9 strongest of 9 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Yifeng Tian · 3 shared papers, 2021–2023 · strength 0.44
- Paper miR-24-3p Regulates Epithelial-Mesenchymal Transition and the Malignant Phenotype of Pancreatic Adenocarcinoma by Regul… DOI 10.1007/s10528-022-10278-5↗
- Paper SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancr… DOI 10.1016/j.celrep.2022.110813↗
- Paper CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancre… DOI 10.1186/s13045-021-01072-8↗
- Cheng‐Yu Liao · 2 shared papers, 2021–2022 · strength 0.25
- Paper SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancr… DOI 10.1016/j.celrep.2022.110813↗
- Paper CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancre… DOI 10.1186/s13045-021-01072-8↗
- Can Yang · 2 shared papers, 2021–2022 · strength 0.25
- Paper SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancr… DOI 10.1016/j.celrep.2022.110813↗
- Paper CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancre… DOI 10.1186/s13045-021-01072-8↗
- Zuwei Wang · 2 shared papers, 2021–2022 · strength 0.25
- Paper SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancr… DOI 10.1016/j.celrep.2022.110813↗
- Paper CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancre… DOI 10.1186/s13045-021-01072-8↗
- Long Huang · 2 shared papers, 2021–2022 · strength 0.25
- Paper SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancr… DOI 10.1016/j.celrep.2022.110813↗
- Paper CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancre… DOI 10.1186/s13045-021-01072-8↗
- Shi Chen · 2 shared papers, 2021–2022 · strength 0.25
- Paper SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancr… DOI 10.1016/j.celrep.2022.110813↗
- Paper CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancre… DOI 10.1186/s13045-021-01072-8↗
- Baiyong Shen · 2 shared papers, 2021–2022 · strength 0.25
- Paper SRSF3-mediated regulation of N6-methyladenosine modification-related lncRNA ANRIL splicing promotes resistance of pancr… DOI 10.1016/j.celrep.2022.110813↗
- Paper CLK1/SRSF5 pathway induces aberrant exon skipping of METTL14 and Cyclin L2 and promotes growth and metastasis of pancre… DOI 10.1186/s13045-021-01072-8↗
- Wentao Huang · 1 shared paper, 2023 · strength 0.19
- Qian Zhan · 1 shared paper, 2021 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.