Pancreatic Cancer Research

ResearcherShanghai Jiao Tong UniversityActive in the last 2 years

Rong Wan

Matched on ORCID iD 0000-0003-3359-6181; OpenAlex author ID A5071176196. Sources: OpenAlex.

Their work

Papers
12 (first author 0, last author 4)
Trials
0
Grants
0
Labs led
1
Collaborators
20
Papers, trials and grants by year

2015: 1 paper · 2016: 3 papers · 2017: 2 papers · 2018: 1 paper · 2019: 1 paper · 2020: 1 paper · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: none · 2025: 1 paper

Papers 12

  1. Machine learning-Based integration develops a metastasis-Related rhythmic gene signature for improving outcomes in pancreatic cancer. Co-author DOI 10.1007/s12672-025-04127-1↗
  2. Erratum: Shikonin induces apoptosis and necroptosis in pancreatic cancer via regulating the expression of RIP1/RIP3 and synergizes the activity of gemcitabine. Co-author PMID 38074805↗
  3. Hypoxia-induced circRNF13 promotes the progression and glycolysis of pancreatic cancer. Last author DOI 10.1038/s12276-022-00877-y↗
  4. RNA demethylase ALKBH5 prevents pancreatic cancer progression by posttranscriptional activation of PER1 in an m6A-YTHDF2-dependent manner. Last author DOI 10.1186/s12943-020-01158-w↗
  5. KMT2D deficiency enhances the anti-cancer activity of L48H37 in pancreatic ductal adenocarcinoma. Last author DOI 10.4251/wjgo.v11.i8.599↗
  6. Identification of Sox6 as a regulator of pancreatic cancer development. Co-author DOI 10.1111/jcmm.13470↗
  7. Thalidomide inhibits proliferation and epithelial-mesenchymal transition by modulating CD133 expression in pancreatic cancer cells. Co-author DOI 10.3892/ol.2017.7213↗
  8. Shikonin induces apoptosis and necroptosis in pancreatic cancer via regulating the expression of RIP1/RIP3 and synergizes the activity of gemcitabine. Co-author PMID 29312502↗
  9. The circadian clock gene Bmal1 acts as a potential anti-oncogene in pancreatic cancer by activating the p53 tumor suppressor pathway. Last author DOI 10.1016/j.canlet.2015.12.002↗
  10. HIC1 attenuates invasion and metastasis by inhibiting the IL-6/STAT3 signalling pathway in human pancreatic cancer. Co-author DOI 10.1016/j.canlet.2016.04.013↗
  11. Transcriptional repression of SOCS3 mediated by IL-6/STAT3 signaling via DNMT1 promotes pancreatic cancer growth and metastasis. Co-author DOI 10.1186/s13046-016-0301-7↗
  12. The inhibitory effects of xanthohumol, a prenylated chalcone derived from hops, on cell growth and tumorigenesis in human pancreatic cancer. Co-author DOI 10.1016/j.biopha.2015.05.020↗

Closest collaborators

The 20 strongest of 20 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Weiliang Jiang · 9 shared papers, 2015–2025 · strength 0.84
  2. Guoyong Hu · 7 shared papers, 2016–2023 · strength 0.58
  3. Congying Chen · 7 shared papers, 2015–2023 · strength 0.58
  4. Lijuan Yang · 5 shared papers, 2015–2023 · strength 0.42
  5. Xingya Guo · 3 shared papers, 2019–2022 · strength 0.34
  6. Li Huang · 4 shared papers, 2016–2018 · strength 0.26
  7. Xingpeng Wang · 4 shared papers, 2016–2018 · strength 0.24
  8. Qiuyan Zhao · 2 shared papers, 2019–2022 · strength 0.23
  9. Zhiyuan Cheng · 1 shared paper, 2025 · strength 0.21
  10. Li Huang · 1 shared paper, 2023 · strength 0.19
  11. Zhonglin Zhu · 1 shared paper, 2022 · strength 0.14
  12. Yinshi Huang · 1 shared paper, 2020 · strength 0.11
  13. Kai Li · 1 shared paper, 2020 · strength 0.11
  14. Bin Hu · 2 shared papers, 2016 · strength 0.11
  15. Mingjie Jiang · 1 shared paper, 2019 · strength 0.09
  16. Sisi Li · 1 shared paper, 2019 · strength 0.09
  17. Qiong-Ying Yuan · 1 shared paper, 2018 · strength 0.08
  18. Lijuan Yang · 1 shared paper, 2018 · strength 0.08
  19. Yingying Lu · 1 shared paper, 2015 · strength 0.05
  20. Xiaohua Jiang · 1 shared paper, 2016 · strength 0.05

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.