ResearcherUniversity of OxfordActive in the last 2 years
Ashwin Jainarayanan
Matched on ORCID iD 0000-0003-3523-1477; OpenAlex author ID A5029635005. Sources: OpenAlex.
Their work
- Papers
- 3 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 10
2021: 1 paper · 2022: none · 2023: 1 paper · 2024: none · 2025: 1 paper
Papers 3
- Distinct immune cell infiltration patterns in pancreatic ductal adenocarcinoma (PDAC) exhibit divergent immune cell selection and immunosuppressive mechanisms. First author DOI 10.1038/s41467-024-55424-2↗
- Pseudotime dynamics of T cells in pancreatic ductal adenocarcinoma inform distinct functional states within the regulatory and cytotoxic T cells. Co-author DOI 10.1016/j.isci.2023.106324↗
- Activated Regulatory T-Cells, Dysfunctional and Senescent T-Cells Hinder the Immunity in Pancreatic Cancer. Co-author DOI 10.3390/cancers13081776↗
Closest collaborators
The 10 strongest of 10 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Lara Rosaline Heij · 3 shared papers, 2021–2025 · strength 0.44
- Paper Pseudotime dynamics of T cells in pancreatic ductal adenocarcinoma inform distinct functional states within the regulat… DOI 10.1016/j.isci.2023.106324↗
- Paper Distinct immune cell infiltration patterns in pancreatic ductal adenocarcinoma (PDAC) exhibit divergent immune cell sel… DOI 10.1038/s41467-024-55424-2↗
- Paper Activated Regulatory T-Cells, Dysfunctional and Senescent T-Cells Hinder the Immunity in Pancreatic Cancer. DOI 10.3390/cancers13081776↗
- Rachael J. M. Bashford-Rogers · 3 shared papers, 2021–2025 · strength 0.44
- Paper Pseudotime dynamics of T cells in pancreatic ductal adenocarcinoma inform distinct functional states within the regulat… DOI 10.1016/j.isci.2023.106324↗
- Paper Distinct immune cell infiltration patterns in pancreatic ductal adenocarcinoma (PDAC) exhibit divergent immune cell sel… DOI 10.1038/s41467-024-55424-2↗
- Paper Activated Regulatory T-Cells, Dysfunctional and Senescent T-Cells Hinder the Immunity in Pancreatic Cancer. DOI 10.3390/cancers13081776↗
- Michael Loran Dustin · 3 shared papers, 2021–2025 · strength 0.44
- Paper Pseudotime dynamics of T cells in pancreatic ductal adenocarcinoma inform distinct functional states within the regulat… DOI 10.1016/j.isci.2023.106324↗
- Paper Distinct immune cell infiltration patterns in pancreatic ductal adenocarcinoma (PDAC) exhibit divergent immune cell sel… DOI 10.1038/s41467-024-55424-2↗
- Paper Activated Regulatory T-Cells, Dysfunctional and Senescent T-Cells Hinder the Immunity in Pancreatic Cancer. DOI 10.3390/cancers13081776↗
- Shivan Sivakumar · 3 shared papers, 2021–2025 · strength 0.44
- Paper Pseudotime dynamics of T cells in pancreatic ductal adenocarcinoma inform distinct functional states within the regulat… DOI 10.1016/j.isci.2023.106324↗
- Paper Distinct immune cell infiltration patterns in pancreatic ductal adenocarcinoma (PDAC) exhibit divergent immune cell sel… DOI 10.1038/s41467-024-55424-2↗
- Paper Activated Regulatory T-Cells, Dysfunctional and Senescent T-Cells Hinder the Immunity in Pancreatic Cancer. DOI 10.3390/cancers13081776↗
- Adam Enver Frampton · 2 shared papers, 2023–2025 · strength 0.33
- Paper Pseudotime dynamics of T cells in pancreatic ductal adenocarcinoma inform distinct functional states within the regulat… DOI 10.1016/j.isci.2023.106324↗
- Paper Distinct immune cell infiltration patterns in pancreatic ductal adenocarcinoma (PDAC) exhibit divergent immune cell sel… DOI 10.1038/s41467-024-55424-2↗
- Samarth Hegde · 1 shared paper, 2025 · strength 0.16
- Miriam Merad · 1 shared paper, 2025 · strength 0.16
- Georg J. Wiltberger · 1 shared paper, 2025 · strength 0.16
- Shihong Wu · 1 shared paper, 2025 · strength 0.16
- Nagina Mangal · 1 shared paper, 2021 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.