ResearcherZhejiang Chinese Medical UniversityActive in the last 2 years
Rubin Cheng
Matched on ORCID iD 0000-0003-3952-2730; OpenAlex author ID A5019812345.
Their work
Counts cover pancreatic cancer work only: a researcher’s other papers, trials and grants are not in these records.
- Pancreatic cancer papers
- 4 (first author 0, last author 3)
- Pancreatic cancer trials
- 0
- Pancreatic cancer grants
- 0
- Labs led
- 1
- Collaborators
- 4
2015: 1 paper · 2016: none · 2017: none · 2018: none · 2019: 1 paper · 2020: 1 paper · 2021: none · 2022: none · 2023: none · 2024: 1 paper
Papers 4
- The Tanshinones (Tan) Extract From Salvia miltiorrhiza Bunge Induces ROS-Dependent Apoptosis in Pancreatic Cancer via AKT Hyperactivation-Mediated FOXO3/SOD2 S… Co-author DOI 10.1177/15347354241258961↗
- Saikosaponin D Inhibits Proliferation and Promotes Apoptosis Through Activation of MKK4-JNK Signaling Pathway in Pancreatic Cancer Cells. Last author DOI 10.2147/ott.s263322↗
- [Current status and future prospect of pharmacotherapy for pancreatic cancer]. Last author DOI 10.19540/j.cnki.cjcmm.20190104.002↗
- Cryptotanshinone suppresses the proliferation and induces the apoptosis of pancreatic cancer cells via the STAT3 signaling pathway. Last author DOI 10.3892/mmr.2015.4379↗
Closest collaborators
The 4 strongest of 4 ranked by shared papers, trials and grants, with recent and small-team work weighing most.
- Yuqing Ge · 4 shared papers, 2015–2024
- Paper The Tanshinones (Tan) Extract From Salvia miltiorrhiza Bunge Induces ROS-Dependent Apoptosis in Pancreatic Cancer via A… DOI 10.1177/15347354241258961↗
- Paper Saikosaponin D Inhibits Proliferation and Promotes Apoptosis Through Activation of MKK4-JNK Signaling Pathway in Pancre… DOI 10.2147/ott.s263322↗
- Paper [Current status and future prospect of pharmacotherapy for pancreatic cancer]. DOI 10.19540/j.cnki.cjcmm.20190104.002↗
- Mengru Lai · 2 shared papers, 2019–2020
- Qin Xu · 1 shared paper, 2024
- Yun Fang Ma · 1 shared paper, 2019
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (the fewest links first, then the links with the most shared work). Built from public records only; no contact details are kept.