ResearcherZhejiang Provincial People's HospitalNo recent work listed
Ming‐Hai Wang
Matched on ORCID iD 0000-0003-4402-5018; OpenAlex author ID A5101742376. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 0, last author 5)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 12
2011: 1 paper · 2012: 1 paper · 2013: none · 2014: 1 paper · 2015: none · 2016: 1 paper · 2017: none · 2018: 1 paper · 2019: 1 paper · 2020: 1 paper
Papers 7
- RON receptor tyrosine kinase in pancreatic ductal adenocarcinoma: Pathogenic mechanism in malignancy and pharmaceutical target for therapy. Last author DOI 10.1016/j.bbcan.2020.188360↗
- Aberrant RON and MET Co-overexpression as Novel Prognostic Biomarkers of Shortened Patient Survival and Therapeutic Targets of Tyrosine Kinase Inhibitors in Pa… Co-author DOI 10.3389/fonc.2019.01377↗
- Preclinical Efficacy of Anti-RON Antibody-Drug Conjugate Zt/g4-MMAE for Targeted Therapy of Pancreatic Cancer Overexpressing RON Receptor Tyrosine Kinase. Last author DOI 10.1021/acs.molpharmaceut.8b00298↗
- Therapeutic evaluation of monoclonal antibody-maytansinoid conjugate as a model of RON-targeted drug delivery for pancreatic cancer treatment. Last author PMID 27293990↗
- Synergistic activities of MET/RON inhibitor BMS-777607 and mTOR inhibitor AZD8055 to polyploid cells derived from pancreatic cancer and cancer stem cells. Last author DOI 10.1158/1535-7163.mct-13-0242↗
- KCN1, a novel synthetic sulfonamide anticancer agent: in vitro and in vivo anti-pancreatic cancer activities and preclinical pharmacology. Co-author DOI 10.1371/journal.pone.0044883↗
- Sustained expression of the RON receptor tyrosine kinase by pancreatic cancer stem cells as a potential targeting moiety for antibody-directed chemotherapeutic… Last author DOI 10.1021/mp200193u↗
Closest collaborators
The 12 strongest of 12 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hangping Yao · 6 shared papers, 2011–2020 · strength 0.51
- Paper RON receptor tyrosine kinase in pancreatic ductal adenocarcinoma: Pathogenic mechanism in malignancy and pharmaceutical… DOI 10.1016/j.bbcan.2020.188360↗
- Paper Aberrant RON and MET Co-overexpression as Novel Prognostic Biomarkers of Shortened Patient Survival and Therapeutic Tar… DOI 10.3389/fonc.2019.01377↗
- Paper Preclinical Efficacy of Anti-RON Antibody-Drug Conjugate Zt/g4-MMAE for Targeted Therapy of Pancreatic Cancer Overexpre… DOI 10.1021/acs.molpharmaceut.8b00298↗
- Chenyu W Hu · 2 shared papers, 2018–2019 · strength 0.17
- Paper Aberrant RON and MET Co-overexpression as Novel Prognostic Biomarkers of Shortened Patient Survival and Therapeutic Tar… DOI 10.3389/fonc.2019.01377↗
- Paper Preclinical Efficacy of Anti-RON Antibody-Drug Conjugate Zt/g4-MMAE for Targeted Therapy of Pancreatic Cancer Overexpre… DOI 10.1021/acs.molpharmaceut.8b00298↗
- Ruiwen Zhang · 3 shared papers, 2011–2016 · strength 0.14
- Paper Therapeutic evaluation of monoclonal antibody-maytansinoid conjugate as a model of RON-targeted drug delivery for pancr… PMID 27293990↗
- Paper Sustained expression of the RON receptor tyrosine kinase by pancreatic cancer stem cells as a potential targeting moiet… DOI 10.1021/mp200193u↗
- Paper KCN1, a novel synthetic sulfonamide anticancer agent: in vitro and in vivo anti-pancreatic cancer activities and precli… DOI 10.1371/journal.pone.0044883↗
- Xiangming Xu · 1 shared paper, 2019 · strength 0.09
- Bo Hong · 1 shared paper, 2019 · strength 0.09
- Yun Qian · 1 shared paper, 2019 · strength 0.09
- Linghui Chen · 1 shared paper, 2018 · strength 0.08
- Weilin Wang · 1 shared paper, 2018 · strength 0.08
- Jun-Ying Zeng · 1 shared paper, 2014 · strength 0.05
- Snehal Padhye · 1 shared paper, 2011 · strength 0.03
- Hongxia Xu · 1 shared paper, 2012 · strength 0.03
- Wei Wang · 1 shared paper, 2012 · strength 0.03
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.