ResearcherHarbin Medical UniversityActive in the last 2 years
Jisheng Hu
Matched on ORCID iD 0000-0003-4550-4897; OpenAlex author ID A5101534899, A5101534902. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 31
2017: 1 paper · 2018: 1 paper · 2019: 2 papers · 2020: 1 paper · 2021: 1 paper · 2022: 1 paper · 2023: 3 papers · 2024: none · 2025: none · 2026: 2 papers
Papers 12
- Tumour-infiltrating adipocyte-derived 12,13-DiHOME subverts CD8+ T cell immunity in pancreatic ductal adenocarcinoma by promoting PPARγ-mediated ferritinophagy… Co-author DOI 10.1136/gutjnl-2025-337119↗
- Spatial Multiomics Analyses Reveal That Diabetes Promotes Pancreatic Cancer Progression by Stimulating Cholesterol-Induced Neutrophil Extracellular Trap Format… Co-author DOI 10.1158/0008-5472.can-25-2854↗
- Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarcinoma Progression through Extracellular … Co-author DOI 10.1002/advs.202203324↗
- circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activation of MMPs in pancreatic cancer. Co-author DOI 10.1186/s12943-023-01847-2↗
- Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. Co-author DOI 10.1186/s12967-023-04693-4↗
- Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. Co-author DOI 10.1111/cas.15388↗
- Cancer-Associated Fibroblast (CAF) Heterogeneity and Targeting Therapy of CAFs in Pancreatic Cancer. Co-author DOI 10.3389/fcell.2021.655152↗
- Small Molecule Inhibitor C188-9 Synergistically Enhances the Demethylated Activity of Low-Dose 5-Aza-2'-Deoxycytidine Against Pancreatic Cancer. Last author DOI 10.3389/fonc.2020.00612↗
- UBL4A inhibits autophagy-mediated proliferation and metastasis of pancreatic ductal adenocarcinoma via targeting LAMP1. Co-author DOI 10.1186/s13046-019-1278-9↗
- Overexpression of KLF5 is associated with poor survival and G1/S progression in pancreatic cancer. Co-author DOI 10.18632/aging.102096↗
- MiR-361-3p regulates ERK1/2-induced EMT via DUSP2 mRNA degradation in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41419-018-0839-8↗
- Silencing IGFBP-2 decreases pancreatic cancer metastasis and enhances chemotherapeutic sensitivity. Co-author DOI 10.18632/oncotarget.18669↗
Closest collaborators
The 25 strongest of 31 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Rui Kong · 9 shared papers, 2017–2026 · strength 1.21
- Paper Spatial Multiomics Analyses Reveal That Diabetes Promotes Pancreatic Cancer Progression by Stimulating Cholesterol-Indu… DOI 10.1158/0008-5472.can-25-2854↗
- Paper Tumour-infiltrating adipocyte-derived 12,13-DiHOME subverts CD8+ T cell immunity in pancreatic ductal adenocarcinoma by… DOI 10.1136/gutjnl-2025-337119↗
- Paper Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. DOI 10.1186/s12967-023-04693-4↗
- Guanqun Li · 7 shared papers, 2018–2026 · strength 1.09
- Paper Spatial Multiomics Analyses Reveal That Diabetes Promotes Pancreatic Cancer Progression by Stimulating Cholesterol-Indu… DOI 10.1158/0008-5472.can-25-2854↗
- Paper Tumour-infiltrating adipocyte-derived 12,13-DiHOME subverts CD8+ T cell immunity in pancreatic ductal adenocarcinoma by… DOI 10.1136/gutjnl-2025-337119↗
- Paper Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. DOI 10.1186/s12967-023-04693-4↗
- Hongze Chen · 8 shared papers, 2018–2023 · strength 0.93
- Paper Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. DOI 10.1186/s12967-023-04693-4↗
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Yongwei Wang · 7 shared papers, 2017–2026 · strength 0.90
- Paper Spatial Multiomics Analyses Reveal That Diabetes Promotes Pancreatic Cancer Progression by Stimulating Cholesterol-Indu… DOI 10.1158/0008-5472.can-25-2854↗
- Paper Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. DOI 10.1186/s12967-023-04693-4↗
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Zhongjie Zhao · 6 shared papers, 2019–2023 · strength 0.70
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Paper Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. DOI 10.1111/cas.15388↗
- Bei Sun · 7 shared papers, 2017–2023 · strength 0.70
- Paper Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. DOI 10.1186/s12967-023-04693-4↗
- Paper Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. DOI 10.1111/cas.15388↗
- Paper Small Molecule Inhibitor C188-9 Synergistically Enhances the Demethylated Activity of Low-Dose 5-Aza-2'-Deoxycytidine A… DOI 10.3389/fonc.2020.00612↗
- Le Li · 6 shared papers, 2018–2023 · strength 0.67
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Paper Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. DOI 10.1111/cas.15388↗
- Bei Sun · 4 shared papers, 2021–2026 · strength 0.64
- Paper Spatial Multiomics Analyses Reveal That Diabetes Promotes Pancreatic Cancer Progression by Stimulating Cholesterol-Indu… DOI 10.1158/0008-5472.can-25-2854↗
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Chundong Cheng · 5 shared papers, 2019–2023 · strength 0.62
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Paper Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. DOI 10.1111/cas.15388↗
- Gang Wang · 4 shared papers, 2019–2026 · strength 0.61
- Paper Spatial Multiomics Analyses Reveal That Diabetes Promotes Pancreatic Cancer Progression by Stimulating Cholesterol-Indu… DOI 10.1158/0008-5472.can-25-2854↗
- Paper Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. DOI 10.1186/s12967-023-04693-4↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Yan Luo · 3 shared papers, 2023–2026 · strength 0.60
- Paper Spatial Multiomics Analyses Reveal That Diabetes Promotes Pancreatic Cancer Progression by Stimulating Cholesterol-Indu… DOI 10.1158/0008-5472.can-25-2854↗
- Paper Tumour-infiltrating adipocyte-derived 12,13-DiHOME subverts CD8+ T cell immunity in pancreatic ductal adenocarcinoma by… DOI 10.1136/gutjnl-2025-337119↗
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Wenbo Yang · 4 shared papers, 2021–2023 · strength 0.56
- Paper Caprin-1 influences autophagy-induced tumor growth and immune modulation in pancreatic cancer. DOI 10.1186/s12967-023-04693-4↗
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Yilong Li · 6 shared papers, 2017–2022 · strength 0.54
- Paper Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. DOI 10.1111/cas.15388↗
- Paper Small Molecule Inhibitor C188-9 Synergistically Enhances the Demethylated Activity of Low-Dose 5-Aza-2'-Deoxycytidine A… DOI 10.3389/fonc.2020.00612↗
- Paper UBL4A inhibits autophagy-mediated proliferation and metastasis of pancreatic ductal adenocarcinoma via targeting LAMP1. DOI 10.1186/s13046-019-1278-9↗
- Shangha Pan · 4 shared papers, 2017–2023 · strength 0.42
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. DOI 10.1111/cas.15388↗
- Paper Overexpression of KLF5 is associated with poor survival and G1/S progression in pancreatic cancer. DOI 10.18632/aging.102096↗
- Danxi Liu · 3 shared papers, 2022–2023 · strength 0.41
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Paper Positive feedback regulation of lncRNA TPT1-AS1 and ITGB3 promotes cell growth and metastasis in pancreatic cancer. DOI 10.1111/cas.15388↗
- Xinglong Geng · 3 shared papers, 2021–2023 · strength 0.41
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Paper Cancer-Associated Fibroblast (CAF) Heterogeneity and Targeting Therapy of CAFs in Pancreatic Cancer. DOI 10.3389/fcell.2021.655152↗
- Le Li · 2 shared papers, 2023–2026 · strength 0.39
- Yangyang Zhang · 2 shared papers, 2023 · strength 0.28
- Paper circEIF3I facilitates the recruitment of SMAD3 to early endosomes to promote TGF-β signalling pathway-mediated activati… DOI 10.1186/s12943-023-01847-2↗
- Paper Tumor Cell Derived Lnc-FSD2-31:1 Contributes to Cancer-Associated Fibroblasts Activation in Pancreatic Ductal Adenocarc… DOI 10.1002/advs.202203324↗
- Le Li · 3 shared papers, 2017–2021 · strength 0.28
- Paper Cancer-Associated Fibroblast (CAF) Heterogeneity and Targeting Therapy of CAFs in Pancreatic Cancer. DOI 10.3389/fcell.2021.655152↗
- Paper UBL4A inhibits autophagy-mediated proliferation and metastasis of pancreatic ductal adenocarcinoma via targeting LAMP1. DOI 10.1186/s13046-019-1278-9↗
- Paper Silencing IGFBP-2 decreases pancreatic cancer metastasis and enhances chemotherapeutic sensitivity. DOI 10.18632/oncotarget.18669↗
- Tao Zhang · 2 shared papers, 2022–2023 · strength 0.27
- X W Bai · 1 shared paper, 2026 · strength 0.22
- Xinyuan Liu · 1 shared paper, 2026 · strength 0.22
- Yingmei Zhang · 1 shared paper, 2026 · strength 0.22
- Bo Sun · 1 shared paper, 2026 · strength 0.22
- Liwei Liu · 1 shared paper, 2023 · strength 0.15
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.