ResearcherGuiyang Medical UniversityActive in the last 2 years
Hao Hua
Matched on ORCID iD 0000-0003-4560-9656; OpenAlex author ID A5024735033. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 2, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 20
2019: 1 paper · 2020: 1 paper · 2021: 1 paper · 2022: 1 paper · 2023: 2 papers · 2024: none · 2025: 3 papers
Papers 9
- Identification of exosome-related genes associated with prognosis and immune infiltration features in pancreatic cancer. Last author DOI 10.1007/s12672-025-01961-1↗
- Causal association between rheumatoid arthritis and pancreatic cancer: a bidirectional two-sample Mendelian randomization study. First author DOI 10.1007/s12672-025-03919-9↗
- Senescence-related circRNA circHIF-1α is associated with pancreatic cancer progression. Co-author DOI 10.1186/s12935-025-03645-w↗
- FOXQ1 promotes pancreatic cancer cell proliferation, tumor stemness, invasion and metastasis through regulation of LDHA-mediated aerobic glycolysis. Co-author DOI 10.1038/s41419-023-06207-y↗
- RNF7 Facilitated the Tumorigenesis of Pancreatic Cancer by Activating PI3K/Akt Signaling Pathway. Co-author DOI 10.1155/2023/1728463↗
- The Senescence-Related Signature Predicts Prognosis and Characterization of Tumor Microenvironment Infiltration in Pancreatic Cancer. Co-author DOI 10.1155/2022/1916787↗
- ZC3HAV1 promotes the proliferation and metastasis via regulating KRAS in pancreatic cancer. First author DOI 10.18632/aging.203296↗
- Long noncoding RNA LINC01559 promotes pancreatic cancer progression by acting as a competing endogenous RNA of miR-1343-3p to upregulate RAF1 expression. Co-author DOI 10.18632/aging.103487↗
- MiR-345-5p functions as a tumor suppressor in pancreatic cancer by directly targeting CCL8. Co-author DOI 10.1016/j.biopha.2018.12.121↗
Closest collaborators
The 20 strongest of 20 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Chao Yu · 3 shared papers, 2022–2023 · strength 0.60
- Paper RNF7 Facilitated the Tumorigenesis of Pancreatic Cancer by Activating PI3K/Akt Signaling Pathway. DOI 10.1155/2023/1728463↗
- Paper FOXQ1 promotes pancreatic cancer cell proliferation, tumor stemness, invasion and metastasis through regulation of LDHA… DOI 10.1038/s41419-023-06207-y↗
- Paper The Senescence-Related Signature Predicts Prognosis and Characterization of Tumor Microenvironment Infiltration in Panc… DOI 10.1155/2022/1916787↗
- Zhiwei He · 2 shared papers, 2023–2025 · strength 0.47
- Changhao Wu · 2 shared papers, 2023–2025 · strength 0.47
- Jie Wang · 2 shared papers, 2019–2025 · strength 0.44
- Lingyu Fu · 1 shared paper, 2025 · strength 0.42
- Jujie Li · 1 shared paper, 2025 · strength 0.42
- Qin Yang · 3 shared papers, 2019–2021 · strength 0.38
- Paper ZC3HAV1 promotes the proliferation and metastasis via regulating KRAS in pancreatic cancer. DOI 10.18632/aging.203296↗
- Paper Long noncoding RNA LINC01559 promotes pancreatic cancer progression by acting as a competing endogenous RNA of miR-1343… DOI 10.18632/aging.103487↗
- Paper MiR-345-5p functions as a tumor suppressor in pancreatic cancer by directly targeting CCL8. DOI 10.1016/j.biopha.2018.12.121↗
- Dijie Zheng · 1 shared paper, 2025 · strength 0.27
- Chao Yuan Yu · 1 shared paper, 2025 · strength 0.27
- Fei Xie · 2 shared papers, 2019–2020 · strength 0.23
- Tinggang Mou · 2 shared papers, 2019–2020 · strength 0.23
- Jian Zheng · 1 shared paper, 2023 · strength 0.22
- Shiyu Chen · 1 shared paper, 2023 · strength 0.20
- Cheng-Yi Sun · 1 shared paper, 2023 · strength 0.20
- Xiushen Li · 1 shared paper, 2022 · strength 0.17
- Wei Huang · 1 shared paper, 2021 · strength 0.16
- Lü Jin · 1 shared paper, 2021 · strength 0.16
- Jie Wang · 1 shared paper, 2020 · strength 0.12
- Xiao Chen · 1 shared paper, 2020 · strength 0.12
- Liang Lai · 1 shared paper, 2019 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.