Pancreatic Cancer Research

ResearcherShanghai Medical College of Fudan UniversityActive in the last 2 years

Zhengjie Dai

ORCID-verified (high confidence, 0.95), identity_conf@1As of 8 November 2026

Matched on ORCID iD 0009-0000-4886-587X; OpenAlex author ID A5048394658. Sources: OpenAlex.

Their work

Papers
11 (first author 2, last author 0)
Trials
0
Grants
0
Labs led
0
Collaborators
24
Papers, trials and grants by year

2022: 2 papers · 2023: 4 papers · 2024: 5 papers

Papers 11

  1. Ectopic CXCR2 expression cells improve the anti-tumor efficiency of CAR-T cells and remodel the immune microenvironment of pancreatic ductal adenocarcinoma. Co-author DOI 10.1007/s00262-024-03648-y↗
  2. DNA hypo-methylation and expression of GBP4 induces T cell exhaustion in pancreatic cancer. Co-author DOI 10.1007/s00262-024-03786-3↗
  3. BCL6 overexpression in CD4+ T cells induces Tfh-like transdifferentiation and enhances antitumor efficiency of CAR-T therapy in pancreatic cancer. First author DOI 10.1016/j.bbadis.2024.167346↗
  4. Antigen/HLA-agnostic strategies for Characterizing Tumor-responsive T cell receptors in PDAC patients via single-cell sequencing and autologous organoid applic… First author DOI 10.1016/j.canlet.2024.216741↗
  5. Efficacy of Hyperthermic Intraperitoneal Chemotherapy Alone for Diffuse Peritoneal Carcinomatosis from Pancreatic Adenocarcinoma: A Single-Centre Retrospective… Co-author DOI 10.21203/rs.3.rs-4011964/v1↗
  6. Characterization of intratumoral tertiary lymphoid structures in pancreatic ductal adenocarcinoma: cellular properties and prognostic significance. Co-author DOI 10.1136/jitc-2023-006698↗
  7. Characterization of Estrogen Receptors in Pancreatic Adenocarcinoma with Tertiary Lymphoid Structures. Co-author DOI 10.3390/cancers15030828↗
  8. Expression Profiles of Cuproptosis-Related Genes Determine Distinct Subtypes of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3390/curroncol30020126↗
  9. Glutamine deprivation induces ferroptosis in pancreatic cancer cells. Co-author DOI 10.3724/abbs.2023029↗
  10. Aberrant APOBEC3C expression induces characteristic genomic instability in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41389-022-00411-9↗
  11. Atypical Mucin Expression Predicts Worse Overall Survival in Resectable Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1155/2022/7353572↗

Closest collaborators

The 24 strongest of 24 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Xianjun Yu · 11 shared papers, 2022–2024 · strength 1.89
  2. Xuan Lin · 10 shared papers, 2022–2024 · strength 1.72
  3. Yusheng Chen · 9 shared papers, 2022–2024 · strength 1.56
  4. Xuan Zou · 9 shared papers, 2022–2024 · strength 1.53
  5. Yesiboli Tasiheng · 9 shared papers, 2022–2024 · strength 1.52
  6. Mingjian Ma · 7 shared papers, 2023–2024 · strength 1.25
  7. Xu Wang · 7 shared papers, 2022–2024 · strength 1.14
  8. He Cheng · 6 shared papers, 2022–2024 · strength 0.97
  9. Chen Liu · 6 shared papers, 2022–2024 · strength 0.97
  10. Chen Liu · 4 shared papers, 2023–2024 · strength 0.72
  11. Yu Yan · 3 shared papers, 2023–2024 · strength 0.51
  12. Guopei Luo · 3 shared papers, 2022–2023 · strength 0.44
  13. He Cheng · 2 shared papers, 2024 · strength 0.39
  14. Yitao Gong · 2 shared papers, 2022 · strength 0.27
  15. Yunzhen Qian · 2 shared papers, 2022 · strength 0.27
  16. Yu Liu · 1 shared paper, 2024 · strength 0.20
  17. Chen Liu · 1 shared paper, 2024 · strength 0.20
  18. Jia Dong · 1 shared paper, 2024 · strength 0.18
  19. Quanxing Ni · 1 shared paper, 2023 · strength 0.17
  20. Zhiwen Xiao · 1 shared paper, 2023 · strength 0.17
  21. Wenchao Gu · 1 shared paper, 2023 · strength 0.17
  22. Yusheng Chen · 1 shared paper, 2023 · strength 0.16
  23. Cheng He · 1 shared paper, 2023 · strength 0.16
  24. Fei Ding · 1 shared paper, 2023 · strength 0.15

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.