ResearcherShanghai Medical College of Fudan UniversityNo recent work listed
Ye Li
Matched on ORCID iD 0009-0002-5110-5454; OpenAlex author ID A5100339238. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 31
2016: 1 paper · 2017: 1 paper · 2018: none · 2019: 1 paper · 2020: 2 papers · 2021: 2 papers · 2022: 1 paper
Papers 8
- Development and validation of a tissue-based DNA methylation risk-score model to predict the prognosis of surgically resected pancreatic cancer patients. Co-author DOI 10.21037/gs-22-517↗
- Fam83D promotes tumorigenesis and gemcitabine resistance of pancreatic adenocarcinoma through the Wnt/β-catenin pathway. Co-author DOI 10.1016/j.lfs.2021.119205↗
- Development and Validation of a Nomogram Based on Nutritional Indicators and Tumor Markers for Prognosis Prediction of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3389/fonc.2021.682969↗
- CXCL9 chemokine promotes the progression of human pancreatic adenocarcinoma through STAT3-dependent cytotoxic T lymphocyte suppression. Co-author DOI 10.18632/aging.102638↗
- miR-939-5p Contributes to the Migration and Invasion of Pancreatic Cancer by Targeting ARHGAP4. Co-author DOI 10.2147/ott.s227644↗
- Selective activation of tumor-suppressive MAPKP signaling pathway by triptonide effectively inhibits pancreatic cancer cell tumorigenicity and tumor growth. Co-author DOI 10.1016/j.bcp.2019.05.010↗
- Methylation of BNIP3 in pancreatic cancer inhibits the induction of mitochondrial-mediated tumor cell apoptosis. Co-author DOI 10.18632/oncotarget.18736↗
- HIF-2α promotes epithelial-mesenchymal transition through regulating Twist2 binding to the promoter of E-cadherin in pancreatic cancer. Co-author DOI 10.1186/s13046-016-0298-y↗
Closest collaborators
The 25 strongest of 31 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Haoran Li · 2 shared papers, 2021–2022 · strength 0.28
- Paper Development and validation of a tissue-based DNA methylation risk-score model to predict the prognosis of surgically re… DOI 10.21037/gs-22-517↗
- Paper Development and Validation of a Nomogram Based on Nutritional Indicators and Tumor Markers for Prognosis Prediction of … DOI 10.3389/fonc.2021.682969↗
- Yuchen Tang · 2 shared papers, 2021–2022 · strength 0.28
- Paper Development and validation of a tissue-based DNA methylation risk-score model to predict the prognosis of surgically re… DOI 10.21037/gs-22-517↗
- Paper Development and Validation of a Nomogram Based on Nutritional Indicators and Tumor Markers for Prognosis Prediction of … DOI 10.3389/fonc.2021.682969↗
- Bin Yi · 2 shared papers, 2021–2022 · strength 0.28
- Paper Development and validation of a tissue-based DNA methylation risk-score model to predict the prognosis of surgically re… DOI 10.21037/gs-22-517↗
- Paper Development and Validation of a Nomogram Based on Nutritional Indicators and Tumor Markers for Prognosis Prediction of … DOI 10.3389/fonc.2021.682969↗
- Luming Liu · 2 shared papers, 2020–2021 · strength 0.27
- Jian Yang · 3 shared papers, 2016–2021 · strength 0.25
- Paper Development and Validation of a Nomogram Based on Nutritional Indicators and Tumor Markers for Prognosis Prediction of … DOI 10.3389/fonc.2021.682969↗
- Paper Methylation of BNIP3 in pancreatic cancer inhibits the induction of mitochondrial-mediated tumor cell apoptosis. DOI 10.18632/oncotarget.18736↗
- Paper HIF-2α promotes epithelial-mesenchymal transition through regulating Twist2 binding to the promoter of E-cadherin in pa… DOI 10.1186/s13046-016-0298-y↗
- Jian Zhou · 3 shared papers, 2016–2021 · strength 0.25
- Paper Development and Validation of a Nomogram Based on Nutritional Indicators and Tumor Markers for Prognosis Prediction of … DOI 10.3389/fonc.2021.682969↗
- Paper Methylation of BNIP3 in pancreatic cancer inhibits the induction of mitochondrial-mediated tumor cell apoptosis. DOI 10.18632/oncotarget.18736↗
- Paper HIF-2α promotes epithelial-mesenchymal transition through regulating Twist2 binding to the promoter of E-cadherin in pa… DOI 10.1186/s13046-016-0298-y↗
- Huifeng Gao · 2 shared papers, 2020 · strength 0.24
- Zhiqiang Meng · 2 shared papers, 2020 · strength 0.24
- Jie Wang · 1 shared paper, 2022 · strength 0.16
- Jian Yang · 1 shared paper, 2022 · strength 0.16
- Zixiang Zhang · 1 shared paper, 2022 · strength 0.16
- Yongqiang Hua · 1 shared paper, 2021 · strength 0.15
- Zhouyu Ning · 1 shared paper, 2021 · strength 0.15
- Weidong Shi · 1 shared paper, 2021 · strength 0.15
- Ke Zhang · 1 shared paper, 2021 · strength 0.15
- Dechun Li · 2 shared papers, 2016–2017 · strength 0.13
- Paper Methylation of BNIP3 in pancreatic cancer inhibits the induction of mitochondrial-mediated tumor cell apoptosis. DOI 10.18632/oncotarget.18736↗
- Paper HIF-2α promotes epithelial-mesenchymal transition through regulating Twist2 binding to the promoter of E-cadherin in pa… DOI 10.1186/s13046-016-0298-y↗
- Dongming Zhu · 2 shared papers, 2016–2017 · strength 0.13
- Paper Methylation of BNIP3 in pancreatic cancer inhibits the induction of mitochondrial-mediated tumor cell apoptosis. DOI 10.18632/oncotarget.18736↗
- Paper HIF-2α promotes epithelial-mesenchymal transition through regulating Twist2 binding to the promoter of E-cadherin in pa… DOI 10.1186/s13046-016-0298-y↗
- Yi Zhang · 2 shared papers, 2016–2017 · strength 0.13
- Paper Methylation of BNIP3 in pancreatic cancer inhibits the induction of mitochondrial-mediated tumor cell apoptosis. DOI 10.18632/oncotarget.18736↗
- Paper HIF-2α promotes epithelial-mesenchymal transition through regulating Twist2 binding to the promoter of E-cadherin in pa… DOI 10.1186/s13046-016-0298-y↗
- Yehua Shen · 1 shared paper, 2020 · strength 0.13
- Gang Chen · 1 shared paper, 2020 · strength 0.13
- Liping Zhuang · 1 shared paper, 2020 · strength 0.13
- Peng Du · 1 shared paper, 2021 · strength 0.12
- Dongming Zhu · 1 shared paper, 2021 · strength 0.12
- Yujie Huang · 1 shared paper, 2021 · strength 0.12
- Hao Chen · 1 shared paper, 2020 · strength 0.12
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.