ResearcherChinese Academy of Medical Sciences & Peking Union Medical CollegeActive in the last 2 years
Xinpeng Yin
Matched on ORCID iD 0009-0003-4512-1287; OpenAlex author ID A5047465002. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 11
2022: 4 papers · 2023: 2 papers · 2024: 2 papers
Papers 8
- Enhanced cytokine signaling and ferroptosis defense interplay initiates obesity-associated pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.canlet.2024.217162↗
- Immune-related signature identifies IL1R2 as an immunological and prognostic biomarker in pancreatic cancer. Co-author DOI 10.1097/jp9.0000000000000175↗
- SQLE promotes pancreatic cancer growth by attenuating ER stress and activating lipid rafts-regulated Src/PI3K/Akt signaling pathway. Co-author DOI 10.1038/s41419-023-05987-7↗
- Cell-in-Cell-Mediated Entosis Reveals a Progressive Mechanism in Pancreatic Cancer. Co-author DOI 10.1053/j.gastro.2023.08.035↗
- Lipid metabolism in pancreatic cancer: emerging roles and potential targets. Co-author DOI 10.1002/cac2.12360↗
- Construction of immune-related signature and identification of S100A14 determining immune-suppressive microenvironment in pancreatic cancer. Co-author DOI 10.1186/s12885-022-09927-0↗
- Construction of a novel model based on cell-in-cell-related genes and validation of KRT7 as a biomarker for predicting survival and immune microenvironment in … Co-author DOI 10.1186/s12885-022-09983-6↗
- Prognostic value of glycolysis markers in pancreatic cancer: A systematic review and meta-analysis. Co-author DOI 10.3389/fonc.2022.1004850↗
Closest collaborators
The 11 strongest of 11 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jianlu Song · 8 shared papers, 2022–2024 · strength 1.52
- Paper Enhanced cytokine signaling and ferroptosis defense interplay initiates obesity-associated pancreatic ductal adenocarci… DOI 10.1016/j.canlet.2024.217162↗
- Paper Immune-related signature identifies IL1R2 as an immunological and prognostic biomarker in pancreatic cancer. DOI 10.1097/jp9.0000000000000175↗
- Paper SQLE promotes pancreatic cancer growth by attenuating ER stress and activating lipid rafts-regulated Src/PI3K/Akt signa… DOI 10.1038/s41419-023-05987-7↗
- Rexiati Ruze · 8 shared papers, 2022–2024 · strength 1.52
- Paper Enhanced cytokine signaling and ferroptosis defense interplay initiates obesity-associated pancreatic ductal adenocarci… DOI 10.1016/j.canlet.2024.217162↗
- Paper Immune-related signature identifies IL1R2 as an immunological and prognostic biomarker in pancreatic cancer. DOI 10.1097/jp9.0000000000000175↗
- Paper SQLE promotes pancreatic cancer growth by attenuating ER stress and activating lipid rafts-regulated Src/PI3K/Akt signa… DOI 10.1038/s41419-023-05987-7↗
- Ruiyuan Xu · 8 shared papers, 2022–2024 · strength 1.52
- Paper Enhanced cytokine signaling and ferroptosis defense interplay initiates obesity-associated pancreatic ductal adenocarci… DOI 10.1016/j.canlet.2024.217162↗
- Paper Immune-related signature identifies IL1R2 as an immunological and prognostic biomarker in pancreatic cancer. DOI 10.1097/jp9.0000000000000175↗
- Paper SQLE promotes pancreatic cancer growth by attenuating ER stress and activating lipid rafts-regulated Src/PI3K/Akt signa… DOI 10.1038/s41419-023-05987-7↗
- Yupei Zhao · 6 shared papers, 2022–2024 · strength 1.17
- Paper Enhanced cytokine signaling and ferroptosis defense interplay initiates obesity-associated pancreatic ductal adenocarci… DOI 10.1016/j.canlet.2024.217162↗
- Paper Immune-related signature identifies IL1R2 as an immunological and prognostic biomarker in pancreatic cancer. DOI 10.1097/jp9.0000000000000175↗
- Paper SQLE promotes pancreatic cancer growth by attenuating ER stress and activating lipid rafts-regulated Src/PI3K/Akt signa… DOI 10.1038/s41419-023-05987-7↗
- Yuan Chen · 6 shared papers, 2022–2024 · strength 1.14
- Paper Enhanced cytokine signaling and ferroptosis defense interplay initiates obesity-associated pancreatic ductal adenocarci… DOI 10.1016/j.canlet.2024.217162↗
- Paper Immune-related signature identifies IL1R2 as an immunological and prognostic biomarker in pancreatic cancer. DOI 10.1097/jp9.0000000000000175↗
- Paper Cell-in-Cell-Mediated Entosis Reveals a Progressive Mechanism in Pancreatic Cancer. DOI 10.1053/j.gastro.2023.08.035↗
- Chengcheng Wang · 3 shared papers, 2022–2024 · strength 0.57
- Paper Immune-related signature identifies IL1R2 as an immunological and prognostic biomarker in pancreatic cancer. DOI 10.1097/jp9.0000000000000175↗
- Paper Prognostic value of glycolysis markers in pancreatic cancer: A systematic review and meta-analysis. DOI 10.3389/fonc.2022.1004850↗
- Paper Construction of a novel model based on cell-in-cell-related genes and validation of KRT7 as a biomarker for predicting … DOI 10.1186/s12885-022-09983-6↗
- Qiang Xu · 3 shared papers, 2022 · strength 0.50
- Paper Prognostic value of glycolysis markers in pancreatic cancer: A systematic review and meta-analysis. DOI 10.3389/fonc.2022.1004850↗
- Paper Construction of immune-related signature and identification of S100A14 determining immune-suppressive microenvironment … DOI 10.1186/s12885-022-09927-0↗
- Paper Construction of a novel model based on cell-in-cell-related genes and validation of KRT7 as a biomarker for predicting … DOI 10.1186/s12885-022-09983-6↗
- Chengcheng Wang · 2 shared papers, 2023–2024 · strength 0.43
- Paper Enhanced cytokine signaling and ferroptosis defense interplay initiates obesity-associated pancreatic ductal adenocarci… DOI 10.1016/j.canlet.2024.217162↗
- Paper SQLE promotes pancreatic cancer growth by attenuating ER stress and activating lipid rafts-regulated Src/PI3K/Akt signa… DOI 10.1038/s41419-023-05987-7↗
- Chengcheng Wang · 2 shared papers, 2022–2023 · strength 0.35
- Hui Zhang · 1 shared paper, 2023 · strength 0.18
- Qiang Xu · 1 shared paper, 2022 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.