ResearcherThe Cancer Institute HospitalActive in the last 2 years
Tomotaka Kato
Matched on ORCID iD 0009-0005-7651-4774; OpenAlex author ID A5112368580. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 38
2021: 2 papers · 2022: 2 papers · 2023: 3 papers · 2024: none · 2025: none · 2026: 2 papers
Papers 9
- Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. Co-author DOI 10.1245/s10434-025-18806-4↗
- ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. Co-author DOI 10.1245/s10434-025-19046-2↗
- Feasibility of laparoscopic radical antegrade modular pancreatosplenectomy (RAMPS) as a standard treatment for distal resectable pancreatic cancer. Co-author DOI 10.1007/s00423-023-02942-0↗
- New approach of circumferential lymph node dissection around the superior mesenteric artery for pancreatic cancer during pancreaticoduodenectomy (with video). Co-author DOI 10.1007/s00423-023-03159-x↗
- RRM1 is mediated by histone acetylation through gemcitabine resistance and contributes to invasiveness and ECM remodeling in pancreatic cancer. Co-author DOI 10.3892/ijo.2023.5499↗
- KRAS variant allele frequency, but not mutation positivity, associates with survival of patients with pancreatic cancer. Co-author DOI 10.1111/cas.15398↗
- Laparoscopic Radical Antegrade Modular Pancreatosplenectomy with Anterocranial Splenic Artery-First Approach for Left-Sided Resectable Pancreatic Cancer (with … Co-author DOI 10.1245/s10434-022-11382-x↗
- C646 inhibits G2/M cell cycle-related proteins and potentiates anti-tumor effects in pancreatic cancer. First author DOI 10.1038/s41598-021-89530-8↗
- Cytoplasmic RRM1 activation as an acute response to gemcitabine treatment is involved in drug resistance of pancreatic cancer cells. Co-author DOI 10.1371/journal.pone.0252917↗
Closest collaborators
The 25 strongest of 38 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Atsushi Oba · 5 shared papers, 2022–2026 · strength 1.02
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Paper New approach of circumferential lymph node dissection around the superior mesenteric artery for pancreatic cancer durin… DOI 10.1007/s00423-023-03159-x↗
- Yosuke Inoue · 5 shared papers, 2022–2026 · strength 0.89
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Paper New approach of circumferential lymph node dissection around the superior mesenteric artery for pancreatic cancer durin… DOI 10.1007/s00423-023-03159-x↗
- Paper Feasibility of laparoscopic radical antegrade modular pancreatosplenectomy (RAMPS) as a standard treatment for distal r… DOI 10.1007/s00423-023-02942-0↗
- Yu Takahashi · 5 shared papers, 2022–2026 · strength 0.89
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Paper New approach of circumferential lymph node dissection around the superior mesenteric artery for pancreatic cancer durin… DOI 10.1007/s00423-023-03159-x↗
- Paper Feasibility of laparoscopic radical antegrade modular pancreatosplenectomy (RAMPS) as a standard treatment for distal r… DOI 10.1007/s00423-023-02942-0↗
- Hiromichi Ito · 5 shared papers, 2021–2026 · strength 0.89
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper New approach of circumferential lymph node dissection around the superior mesenteric artery for pancreatic cancer durin… DOI 10.1007/s00423-023-03159-x↗
- Paper Feasibility of laparoscopic radical antegrade modular pancreatosplenectomy (RAMPS) as a standard treatment for distal r… DOI 10.1007/s00423-023-02942-0↗
- Yoshihiro Ono · 4 shared papers, 2022–2026 · strength 0.83
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Paper Feasibility of laparoscopic radical antegrade modular pancreatosplenectomy (RAMPS) as a standard treatment for distal r… DOI 10.1007/s00423-023-02942-0↗
- Naoki Sasahira · 3 shared papers, 2022–2026 · strength 0.59
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Paper KRAS variant allele frequency, but not mutation positivity, associates with survival of patients with pancreatic cancer. DOI 10.1111/cas.15398↗
- Takashi Sasaki · 2 shared papers, 2026 · strength 0.48
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Aya Maekawa · 2 shared papers, 2026 · strength 0.48
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Masato Ozaka · 2 shared papers, 2026 · strength 0.48
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Tatsunori Miyata · 2 shared papers, 2026 · strength 0.48
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Masaaki Iwatsuki · 2 shared papers, 2026 · strength 0.48
- Paper Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Resection. DOI 10.1245/s10434-025-18806-4↗
- Paper ASO Visual Abstract: Surgical Challenges Associated with Collateral Veins Formation in Pancreatic Cancer with Vein Rese… DOI 10.1245/s10434-025-19046-2↗
- Hiroaki Ono · 3 shared papers, 2021–2023 · strength 0.40
- Paper RRM1 is mediated by histone acetylation through gemcitabine resistance and contributes to invasiveness and ECM remodeli… DOI 10.3892/ijo.2023.5499↗
- Paper Cytoplasmic RRM1 activation as an acute response to gemcitabine treatment is involved in drug resistance of pancreatic … DOI 10.1371/journal.pone.0252917↗
- Paper C646 inhibits G2/M cell cycle-related proteins and potentiates anti-tumor effects in pancreatic cancer. DOI 10.1038/s41598-021-89530-8↗
- Kosuke Ogawa · 3 shared papers, 2021–2023 · strength 0.40
- Paper RRM1 is mediated by histone acetylation through gemcitabine resistance and contributes to invasiveness and ECM remodeli… DOI 10.3892/ijo.2023.5499↗
- Paper Cytoplasmic RRM1 activation as an acute response to gemcitabine treatment is involved in drug resistance of pancreatic … DOI 10.1371/journal.pone.0252917↗
- Paper C646 inhibits G2/M cell cycle-related proteins and potentiates anti-tumor effects in pancreatic cancer. DOI 10.1038/s41598-021-89530-8↗
- Minoru Tanabe · 3 shared papers, 2021–2023 · strength 0.40
- Paper RRM1 is mediated by histone acetylation through gemcitabine resistance and contributes to invasiveness and ECM remodeli… DOI 10.3892/ijo.2023.5499↗
- Paper Cytoplasmic RRM1 activation as an acute response to gemcitabine treatment is involved in drug resistance of pancreatic … DOI 10.1371/journal.pone.0252917↗
- Paper C646 inhibits G2/M cell cycle-related proteins and potentiates anti-tumor effects in pancreatic cancer. DOI 10.1038/s41598-021-89530-8↗
- Shinji Tanaka · 3 shared papers, 2021–2023 · strength 0.40
- Paper RRM1 is mediated by histone acetylation through gemcitabine resistance and contributes to invasiveness and ECM remodeli… DOI 10.3892/ijo.2023.5499↗
- Paper Cytoplasmic RRM1 activation as an acute response to gemcitabine treatment is involved in drug resistance of pancreatic … DOI 10.1371/journal.pone.0252917↗
- Paper C646 inhibits G2/M cell cycle-related proteins and potentiates anti-tumor effects in pancreatic cancer. DOI 10.1038/s41598-021-89530-8↗
- Yoshihiro Ono · 2 shared papers, 2022–2023 · strength 0.30
- Paper New approach of circumferential lymph node dissection around the superior mesenteric artery for pancreatic cancer durin… DOI 10.1007/s00423-023-03159-x↗
- Paper KRAS variant allele frequency, but not mutation positivity, associates with survival of patients with pancreatic cancer. DOI 10.1111/cas.15398↗
- Manabu Takamatsu · 2 shared papers, 2022–2023 · strength 0.30
- Paper New approach of circumferential lymph node dissection around the superior mesenteric artery for pancreatic cancer durin… DOI 10.1007/s00423-023-03159-x↗
- Paper KRAS variant allele frequency, but not mutation positivity, associates with survival of patients with pancreatic cancer. DOI 10.1111/cas.15398↗
- Yoshiki Murase · 2 shared papers, 2021–2023 · strength 0.27
- Shuichi Watanabe · 2 shared papers, 2021–2023 · strength 0.27
- Yu Takahashi · 1 shared paper, 2026 · strength 0.24
- Yosuke Inoue · 1 shared paper, 2026 · strength 0.24
- Toshiro Ogura · 2 shared papers, 2021 · strength 0.24
- Paper Cytoplasmic RRM1 activation as an acute response to gemcitabine treatment is involved in drug resistance of pancreatic … DOI 10.1371/journal.pone.0252917↗
- Paper C646 inhibits G2/M cell cycle-related proteins and potentiates anti-tumor effects in pancreatic cancer. DOI 10.1038/s41598-021-89530-8↗
- Daisuke Ban · 2 shared papers, 2021 · strength 0.24
- Paper Cytoplasmic RRM1 activation as an acute response to gemcitabine treatment is involved in drug resistance of pancreatic … DOI 10.1371/journal.pone.0252917↗
- Paper C646 inhibits G2/M cell cycle-related proteins and potentiates anti-tumor effects in pancreatic cancer. DOI 10.1038/s41598-021-89530-8↗
- Y. H. Andrew Wu · 1 shared paper, 2023 · strength 0.18
- Shoki Sato · 1 shared paper, 2023 · strength 0.18
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.