ResearcherUniversity of FreiburgNo recent work listed
Andrea Schmidt
Matched on ORCID iD 0009-0009-8246-1030; OpenAlex author ID A5029860230. Sources: OpenAlex.
Their work
- Papers
- 3
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 9
2015: 1 paper · 2016: none · 2017: 1 paper · 2018: 1 paper
Papers 3
- Metformin Decreases the Incidence of Pancreatic Ductal Adenocarcinoma Promoted by Diet-induced Obesity in the Conditional KrasG12D Mouse Model. Co-author DOI 10.1038/s41598-018-24337-8↗
- Direct growth-inhibitory effects of prostaglandin E2 in pancreatic cancer cells in vitro through an EP4/PKA-mediated mechanism. Co-author DOI 10.1016/j.surg.2016.12.037↗
- E-cadherin expression in obesity-associated, Kras-initiated pancreatic ductal adenocarcinoma in mice. Co-author DOI 10.1016/j.surg.2015.07.023↗
Closest collaborators
The 9 strongest of 9 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hui-Hua Chang · 3 shared papers, 2015–2018 · strength 0.20
- Paper Metformin Decreases the Incidence of Pancreatic Ductal Adenocarcinoma Promoted by Diet-induced Obesity in the Condition… DOI 10.1038/s41598-018-24337-8↗
- Paper Direct growth-inhibitory effects of prostaglandin E2 in pancreatic cancer cells in vitro through an EP4/PKA-mediated me… DOI 10.1016/j.surg.2016.12.037↗
- Paper E-cadherin expression in obesity-associated, Kras-initiated pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.surg.2015.07.023↗
- Guido Eibl · 3 shared papers, 2015–2018 · strength 0.20
- Paper Metformin Decreases the Incidence of Pancreatic Ductal Adenocarcinoma Promoted by Diet-induced Obesity in the Condition… DOI 10.1038/s41598-018-24337-8↗
- Paper Direct growth-inhibitory effects of prostaglandin E2 in pancreatic cancer cells in vitro through an EP4/PKA-mediated me… DOI 10.1016/j.surg.2016.12.037↗
- Paper E-cadherin expression in obesity-associated, Kras-initiated pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.surg.2015.07.023↗
- Oscar Joe Hines · 3 shared papers, 2015–2018 · strength 0.20
- Paper Metformin Decreases the Incidence of Pancreatic Ductal Adenocarcinoma Promoted by Diet-induced Obesity in the Condition… DOI 10.1038/s41598-018-24337-8↗
- Paper Direct growth-inhibitory effects of prostaglandin E2 in pancreatic cancer cells in vitro through an EP4/PKA-mediated me… DOI 10.1016/j.surg.2016.12.037↗
- Paper E-cadherin expression in obesity-associated, Kras-initiated pancreatic ductal adenocarcinoma in mice. DOI 10.1016/j.surg.2015.07.023↗
- James W. Sinnett‐Smith · 2 shared papers, 2017–2018 · strength 0.15
- Paper Metformin Decreases the Incidence of Pancreatic Ductal Adenocarcinoma Promoted by Diet-induced Obesity in the Condition… DOI 10.1038/s41598-018-24337-8↗
- Paper Direct growth-inhibitory effects of prostaglandin E2 in pancreatic cancer cells in vitro through an EP4/PKA-mediated me… DOI 10.1016/j.surg.2016.12.037↗
- Enrique Rozengurt · 2 shared papers, 2017–2018 · strength 0.15
- Paper Metformin Decreases the Incidence of Pancreatic Ductal Adenocarcinoma Promoted by Diet-induced Obesity in the Condition… DOI 10.1038/s41598-018-24337-8↗
- Paper Direct growth-inhibitory effects of prostaglandin E2 in pancreatic cancer cells in vitro through an EP4/PKA-mediated me… DOI 10.1016/j.surg.2016.12.037↗
- David W. Dawson · 2 shared papers, 2017–2018 · strength 0.15
- Paper Metformin Decreases the Incidence of Pancreatic Ductal Adenocarcinoma Promoted by Diet-induced Obesity in the Condition… DOI 10.1038/s41598-018-24337-8↗
- Paper Direct growth-inhibitory effects of prostaglandin E2 in pancreatic cancer cells in vitro through an EP4/PKA-mediated me… DOI 10.1016/j.surg.2016.12.037↗
- Hao Fang · 1 shared paper, 2018 · strength 0.08
- Kathleen M. Hertzer · 1 shared paper, 2015 · strength 0.05
- Alexander P. Stark · 1 shared paper, 2015 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.