ResearcherNew York UniversityActive in the last 2 years
Laura DeLong Wood
Matched on ORCID iD 0000-0003-3096-652X; OpenAlex author ID A5038534642; NIH RePORTER profile ID 11268904. Sources: OpenAlex.
Their work
- Papers
- 72 (first author 8, last author 12)
- Trials
- 0
- Grants
- 6
- Labs led
- 3
- Collaborators
- 305
2013: 4 papers · 2014: none · 2015: 4 papers · 2016: 6 papers · 2017: 6 papers · 2018: 4 papers · 2019: 10 papers · 2020: 5 papers, 1 grant · 2021: 5 papers · 2022: 4 papers, 1 grant · 2023: 4 papers · 2024: 5 papers · 2025: 5 papers, 1 grant · 2026: 10 papers, 3 grants
Papers 50 of 72
- Pancreatic ductal adenocarcinoma: The Vision of Heracles. Co-author DOI 10.1016/j.ccell.2026.07.014↗
- SPINK1-COL18A1 crosstalk shapes epigenome and drives cancer stemness in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.celrep.2026.117806↗
- Quantitative 3D histology reveals localized immune remodeling during early pancreatic cancer progression. Co-author DOI 10.1016/j.cpblue.2026.100008↗
- The want for Wnt: Pancreatic cancer assembles a self-sustaining Wnt-signaling ecosystem. First author DOI 10.1016/j.devcel.2026.04.010↗
- Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. Co-author DOI 10.1038/s41586-026-10995-6↗
- Inactivation of CDKN2AARF Promotes p53-Independent Remodeling of the PDAC Tumor Microenvironment. Co-author DOI 10.1158/0008-5472.can-25-1969↗
- Convergence for Inactivation of TGFβ Signaling Is a Common Feature of Advanced Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-24-0772↗
- Targeting RAS for Pancreatic Cancer Interception. Last author DOI 10.1158/2159-8290.cd-26-1179↗
- Computational Modeling of Cellular Influence Delineates Functionally Relevant and Distinct Cellular Neighborhoods in Primary and Metastatic Pancreatic Ductal A… Co-author DOI 10.1158/2326-6066.cir-25-0844↗
- Deep Learning Enabled 3D Multi-Omic Analysis Reveals Molecular Signatures of Heterogeneous Response to Chemotherapy in Pancreatic Cancer Last author DOI 10.64898/2026.03.03.709150↗
- Mucinous cystic neoplasms and simple mucinous cysts are two distinct precursors of pancreatic cancer: clinicopathological, genomic, and transcriptomic characte… Co-author DOI 10.1002/path.6437↗
- Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance in a novel pancreatic cancer mouse model Co-author DOI 10.1101/2025.07.14.664767↗
- Spatial profiling of human pancreatic ductal adenocarcinoma reveals molecular alterations associated with venous invasion. Last author DOI 10.1126/scitranslmed.ady7524↗
- The Evolutionary Forest of Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-23-1541↗
- P4HA1 Mediates Hypoxia-Induced Invasion in Human Pancreatic Cancer Organoids. Last author DOI 10.1158/2767-9764.crc-24-0025↗
- Understanding familial risk of pancreatic ductal adenocarcinoma. First author DOI 10.1007/s10689-024-00383-2↗
- Author Correction: Genotype tunes pancreatic ductal adenocarcinoma tissue tension to induce matricellular fibrosis and tumor progression. Co-author DOI 10.1038/s41591-023-02694-w↗
- Clinical Relevance of Cancerization of Ducts in Resected Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1097/mpa.0000000000002326↗
- Power-law growth models explain incidences and sizes of pancreatic cancer precursor lesions and confirm spatial genomic findings. Co-author DOI 10.1126/sciadv.ado5103↗
- Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial-Mesenchymal Transition and Inflammation in Cancer Cells in Pancreatic Du… Co-author DOI 10.1158/0008-5472.can-23-1660↗
- Tissue clearing and 3D reconstruction of digitized, serially sectioned slides provide novel insights into pancreatic cancer. Co-author DOI 10.1016/j.medj.2022.11.009↗
- Multifaceted role for p53 in pancreatic cancer suppression. Co-author DOI 10.1073/pnas.2211937120↗
- Intraparenchymal metastases as a cause for local recurrence of pancreatic cancer. Co-author DOI 10.1111/his.14839↗
- Morphology-guided transcriptomic analysis of human pancreatic cancer organoids reveals microenvironmental signals that enhance invasion. Last author DOI 10.1172/jci162054↗
- CODA: quantitative 3D reconstruction of large tissues at cellular resolution Co-author DOI 10.1038/s41592-022-01650-9↗
- Pancreatic Cancer: Pathogenesis, Screening, Diagnosis, and Treatment. Co-author DOI 10.1053/j.gastro.2022.03.056↗
- Organoids: A Promising Preclinical Model for Pancreatic Cancer Research. First author DOI 10.1097/mpa.0000000000002084↗
- Inflammatory Signaling in Pancreatic Cancer Transfers Between a Single-cell RNA Sequencing Atlas and Co-Culture Co-author DOI 10.1101/2022.07.14.500096↗
- Pancreatic cancer pathology viewed in the light of evolution. Co-author DOI 10.1007/s10555-020-09953-z↗
- Early detection of pancreatic cancer using DNA-based molecular approaches. First author DOI 10.1038/s41575-021-00470-0↗
- Methylation-based Cell-free DNA Signature for Early Detection of Pancreatic Cancer. Co-author DOI 10.1097/mpa.0000000000001919↗
- Comprehensive characterisation of pancreatic ductal adenocarcinoma with microsatellite instability: histology, molecular pathology and clinical implications. Co-author DOI 10.1136/gutjnl-2020-320726↗
- Cell of Origin Influences Pancreatic Cancer Subtype. Co-author DOI 10.1158/2159-8290.cd-20-0633↗
- Intraductal pancreatic cancer is less responsive than cancer in the stroma to neoadjuvant chemotherapy. Last author DOI 10.1038/s41379-020-0572-6↗
- A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s43018-019-0010-1↗
- Pattern of Invasion in Human Pancreatic Cancer Organoids Is Associated with Loss of SMAD4 and Clinical Outcome. Last author DOI 10.1158/0008-5472.can-19-1523↗
- The Evolutionary Origins of Recurrent Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-19-1508↗
- Intraductal Transplantation Models of Human Pancreatic Ductal Adenocarcinoma Reveal Progressive Transition of Molecular Subtypes. Co-author DOI 10.1158/2159-8290.cd-20-0133↗
- Single-cell sequencing defines genetic heterogeneity in pancreatic cancer precursor lesions. Last author DOI 10.1002/path.5194↗
- Why is pancreatic cancer so deadly? The pathologist's view. Last author DOI 10.1002/path.5260↗
- Tumor Microbiome Diversity and Composition Influence Pancreatic Cancer Outcomes. Co-author DOI 10.1016/j.cell.2019.07.008↗
- Pancreatic cancer arising in the remnant pancreas is not always a relapse of the preceding primary. Co-author DOI 10.1038/s41379-018-0183-7↗
- Genetics of Familial and Sporadic Pancreatic Cancer. Co-author DOI 10.1053/j.gastro.2018.12.039↗
- Biphenotypic Differentiation of Pancreatic Cancer in 3-Dimensional Culture. Co-author DOI 10.1097/mpa.0000000000001390↗
- The Genetic Basis of Transcriptional and Spatial Heterogeneity of Squamous Features in Pancreatic Ductal Adenocarcinoma Co-author DOI 10.1101/548354↗
- Promoter methylation of ADAMTS1 and BNC1 as potential biomarkers for early detection of pancreatic cancer in blood. Co-author DOI 10.1186/s13148-019-0650-0↗
- Prognostic Role of High-Grade Tumor Budding in Pancreatic Ductal Adenocarcinoma: A Systematic Review and Meta-Analysis with a Focus on Epithelial to Mesenchyma… Co-author DOI 10.3390/cancers11010113↗
- Liquid Biopsy as Surrogate for Tissue for Molecular Profiling in Pancreatic Cancer: A Meta-Analysis Towards Precision Medicine. Co-author DOI 10.3390/cancers11081152↗
- From somatic mutation to early detection: insights from molecular characterization of pancreatic cancer precursor lesions. First author DOI 10.1002/path.5154↗
- Cancerization of the Pancreatic Ducts: Demonstration of a Common and Under-recognized Process Using Immunolabeling of Paired Duct Lesions and Invasive Pancreat… Co-author DOI 10.1097/pas.0000000000001148↗
Grants 6
- Interrogation of the Impact of Selection on the Evolution of Human Pancreatic Cancer Precursor Lesions U01CA271273↗
- RTB 1 U54CA268083↗
- TBEL Project 3 U54CA274371↗
- An atlas of pancreatic tumorigenesis in the context of altered DNA repair occurring in high-risk individuals U01CA294548↗
- Early Events in Pancreatic Cancer Grant: Laura Wood AWARD lustgarten:2022-laura-wood-md-phd-early-events-in-pancreatic-cancer-grant-
- Characterization of the Molecular Determinants of High-Grade Dysplasia in Pancreatic Cancer Precursor Lesions K08DK107781↗
Closest collaborators
The 25 strongest of 305 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Elana Judith Fertig · 6 shared papers, 3 shared grants, 2022–2026 · strength 3.61
- Ralph H. Hruban · 30 shared papers, 2013–2026 · strength 2.68
- Paper Quantitative 3D histology reveals localized immune remodeling during early pancreatic cancer progression. DOI 10.1016/j.cpblue.2026.100008↗
- Paper Power-law growth models explain incidences and sizes of pancreatic cancer precursor lesions and confirm spatial genomic… DOI 10.1126/sciadv.ado5103↗
- Paper Clinical Relevance of Cancerization of Ducts in Resected Pancreatic Ductal Adenocarcinoma. DOI 10.1097/mpa.0000000000002326↗
- Denis Wirtz · 7 shared papers, 1 shared grant, 2017–2026 · strength 2.21
- Grant RTB 1 U54CA268083↗
- Paper Quantitative 3D histology reveals localized immune remodeling during early pancreatic cancer progression. DOI 10.1016/j.cpblue.2026.100008↗
- Paper Power-law growth models explain incidences and sizes of pancreatic cancer precursor lesions and confirm spatial genomic… DOI 10.1126/sciadv.ado5103↗
- Pei-Hsun wu · 3 shared papers, 1 shared grant, 2023–2026 · strength 1.56
- Grant RTB 1 U54CA268083↗
- Paper Quantitative 3D histology reveals localized immune remodeling during early pancreatic cancer progression. DOI 10.1016/j.cpblue.2026.100008↗
- Paper Tissue clearing and 3D reconstruction of digitized, serially sectioned slides provide novel insights into pancreatic ca… DOI 10.1016/j.medj.2022.11.009↗
- Anirban Maitra · 7 shared papers, 1 shared grant, 2015–2026 · strength 1.52
- Ashley L. Kiemen · 8 shared papers, 2022–2026 · strength 1.50
- Paper Quantitative 3D histology reveals localized immune remodeling during early pancreatic cancer progression. DOI 10.1016/j.cpblue.2026.100008↗
- Paper Power-law growth models explain incidences and sizes of pancreatic cancer precursor lesions and confirm spatial genomic… DOI 10.1126/sciadv.ado5103↗
- Paper Deep Learning Enabled 3D Multi-Omic Analysis Reveals Molecular Signatures of Heterogeneous Response to Chemotherapy in … DOI 10.64898/2026.03.03.709150↗
- Christine A. Iacobuzio–Donahue · 15 shared papers, 2013–2026 · strength 1.49
- Paper Convergence for Inactivation of TGFβ Signaling Is a Common Feature of Advanced Pancreatic Cancer. DOI 10.1158/2159-8290.cd-24-0772↗
- Paper SPINK1-COL18A1 crosstalk shapes epigenome and drives cancer stemness in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2026.117806↗
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Christopher Lee Wolfgang · 19 shared papers, 2013–2026 · strength 1.31
- Paper SPINK1-COL18A1 crosstalk shapes epigenome and drives cancer stemness in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2026.117806↗
- Paper Methylation-based Cell-free DNA Signature for Early Detection of Pancreatic Cancer. DOI 10.1097/mpa.0000000000001919↗
- Paper Cancerization of the Pancreatic Ducts: Demonstration of a Common and Under-recognized Process Using Immunolabeling of P… DOI 10.1097/pas.0000000000001148↗
- Jin Peng He · 12 shared papers, 2016–2024 · strength 1.16
- Paper Clinical Relevance of Cancerization of Ducts in Resected Pancreatic Ductal Adenocarcinoma. DOI 10.1097/mpa.0000000000002326↗
- Paper Tissue clearing and 3D reconstruction of digitized, serially sectioned slides provide novel insights into pancreatic ca… DOI 10.1016/j.medj.2022.11.009↗
- Paper Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial-Mesenchymal Transition and Infla… DOI 10.1158/0008-5472.can-23-1660↗
- Elizabeth M. Jaffee · 5 shared papers, 2022–2026 · strength 1.14
- Paper Targeting RAS for Pancreatic Cancer Interception. DOI 10.1158/2159-8290.cd-26-1179↗
- Paper Computational Modeling of Cellular Influence Delineates Functionally Relevant and Distinct Cellular Neighborhoods in Pr… DOI 10.1158/2326-6066.cir-25-0844↗
- Paper Pancreatic Cancer: Pathogenesis, Screening, Diagnosis, and Treatment. DOI 10.1053/j.gastro.2022.03.056↗
- Rachel Karchin · 2 shared papers, 1 shared grant, 2016–2026 · strength 1.11
- Grant Interrogation of the Impact of Selection on the Evolution of Human Pancreatic Cancer Precursor Lesions U01CA271273↗
- Paper Single-cell sequencing defines genetic heterogeneity in pancreatic cancer precursor lesions. DOI 10.1002/path.5194↗
- Paper Whole Genome Sequencing Defines the Genetic Heterogeneity of Familial Pancreatic Cancer. DOI 10.1158/2159-8290.cd-15-0402↗
- Marina Pasca di Magliano · 1 shared paper, 1 shared grant, 2026 · strength 1.05
- Michael Goggins · 12 shared papers, 2015–2026 · strength 0.99
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genetics of Familial and Sporadic Pancreatic Cancer. DOI 10.1053/j.gastro.2018.12.039↗
- Paper Pancreatic cancer pathology viewed in the light of evolution. DOI 10.1007/s10555-020-09953-z↗
- Nicholas Jason Roberts · 8 shared papers, 2016–2024 · strength 0.95
- Paper Understanding familial risk of pancreatic ductal adenocarcinoma. DOI 10.1007/s10689-024-00383-2↗
- Paper Intraparenchymal metastases as a cause for local recurrence of pancreatic cancer. DOI 10.1111/his.14839↗
- Paper Pancreatic cancer pathology viewed in the light of evolution. DOI 10.1007/s10555-020-09953-z↗
- Rosalie C. Sears · 1 shared paper, 1 shared grant, 2025 · strength 0.90
- Jonathan Robert Brody · 1 shared paper, 1 shared grant, 2025 · strength 0.90
- Alison P. Klein · 4 shared papers, 2016–2026 · strength 0.86
- Jungeui Hong · 6 shared papers, 2017–2026 · strength 0.76
- Paper Convergence for Inactivation of TGFβ Signaling Is a Common Feature of Advanced Pancreatic Cancer. DOI 10.1158/2159-8290.cd-24-0772↗
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper The Evolutionary Origins of Recurrent Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1508↗
- Bernat Navarro-Serer · 4 shared papers, 2020–2025 · strength 0.74
- Paper Organoids: A Promising Preclinical Model for Pancreatic Cancer Research. DOI 10.1097/mpa.0000000000002084↗
- Paper P4HA1 Mediates Hypoxia-Induced Invasion in Human Pancreatic Cancer Organoids. DOI 10.1158/2767-9764.crc-24-0025↗
- Paper Morphology-guided transcriptomic analysis of human pancreatic cancer organoids reveals microenvironmental signals that … DOI 10.1172/jci162054↗
- Won Jin Ho · 3 shared papers, 2026 · strength 0.73
- Paper Computational Modeling of Cellular Influence Delineates Functionally Relevant and Distinct Cellular Neighborhoods in Pr… DOI 10.1158/2326-6066.cir-25-0844↗
- Paper Quantitative 3D histology reveals localized immune remodeling during early pancreatic cancer progression. DOI 10.1016/j.cpblue.2026.100008↗
- Paper Deep Learning Enabled 3D Multi-Omic Analysis Reveals Molecular Signatures of Heterogeneous Response to Chemotherapy in … DOI 10.64898/2026.03.03.709150↗
- Luciane Tsukamoto Kagohara · 4 shared papers, 2022–2026 · strength 0.68
- Paper Deep Learning Enabled 3D Multi-Omic Analysis Reveals Molecular Signatures of Heterogeneous Response to Chemotherapy in … DOI 10.64898/2026.03.03.709150↗
- Paper Spatial profiling of human pancreatic ductal adenocarcinoma reveals molecular alterations associated with venous invasi… DOI 10.1126/scitranslmed.ady7524↗
- Paper Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial-Mesenchymal Transition and Infla… DOI 10.1158/0008-5472.can-23-1660↗
- Zachary A. Kohutek · 5 shared papers, 2017–2026 · strength 0.67
- Paper Convergence for Inactivation of TGFβ Signaling Is a Common Feature of Advanced Pancreatic Cancer. DOI 10.1158/2159-8290.cd-24-0772↗
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-019-0010-1↗
- Pei-Hsun Wu · 4 shared papers, 2017–2026 · strength 0.65
- Paper Power-law growth models explain incidences and sizes of pancreatic cancer precursor lesions and confirm spatial genomic… DOI 10.1126/sciadv.ado5103↗
- Paper Deep Learning Enabled 3D Multi-Omic Analysis Reveals Molecular Signatures of Heterogeneous Response to Chemotherapy in … DOI 10.64898/2026.03.03.709150↗
- Paper CODA: quantitative 3D reconstruction of large tissues at cellular resolution DOI 10.1038/s41592-022-01650-9↗
- Richard Andrew Burkhart · 6 shared papers, 2018–2024 · strength 0.64
- Paper Clinical Relevance of Cancerization of Ducts in Resected Pancreatic Ductal Adenocarcinoma. DOI 10.1097/mpa.0000000000002326↗
- Paper Transfer Learning Reveals Cancer-Associated Fibroblasts Are Associated with Epithelial-Mesenchymal Transition and Infla… DOI 10.1158/0008-5472.can-23-1660↗
- Paper Inflammatory Signaling in Pancreatic Cancer Transfers Between a Single-cell RNA Sequencing Atlas and Co-Culture DOI 10.1101/2022.07.14.500096↗
- Nikki K. Lytle · 1 shared paper, 2026 · strength 0.63
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.