ResearcherNew York UniversityActive in the last 2 years
Cosimo Commisso
Matched on ORCID iD 0000-0002-7884-9210; OpenAlex author ID A5080326390; NIH RePORTER profile ID 10376567. Sources: funder_awards.
Their work
- Papers
- 13 (first author 1, last author 6)
- Trials
- 0
- Grants
- 9
- Labs led
- 1
- Collaborators
- 41
2011: 1 grant · 2012: none · 2013: none · 2014: none · 2015: 1 paper, 1 grant · 2016: none · 2017: none · 2018: none · 2019: none · 2020: 4 papers · 2021: 1 grant · 2022: none · 2023: none · 2024: 2 papers · 2025: 4 papers, 3 grants · 2026: 2 papers, 3 grants
Papers 13
- The Aging Microenvironment Is a Determinant of Immune Exclusion and Metastatic Fate in Pancreatic Cancer. Last author DOI 10.1158/0008-5472.can-25-1904↗
- Tumor-derived SAA1-TLR4 signaling drives tumor-to-muscle communication in pancreatic cancer cachexia Co-author DOI 10.64898/2026.09.04.748998↗
- Macropinocytosis maintains CAF subtype identity under metabolic stress in pancreatic cancer. Last author DOI 10.1016/j.ccell.2025.06.021↗
- PI5P4Kα promotes glucose and iron acquisition to support metabolic fitness in pancreatic cancer. Co-author DOI 10.1016/j.celrep.2025.116199↗
- Disabling PSGL-1 abrogates immune suppression and resistance to PD-1 blockade in pancreatic cancer Co-author DOI 10.1101/2025.05.22.655365↗
- MICAL2 Promotes Pancreatic Cancer Growth and Metastasis. Co-author DOI 10.1158/0008-5472.can-24-0744↗
- Glutamine mimicry suppresses tumor progression through asparagine metabolism in pancreatic ductal adenocarcinoma. Last author DOI 10.1038/s43018-023-00649-1↗
- Macropinocytosis controls metabolic stress-driven CAF subtype identity in pancreatic cancer Last author DOI 10.1101/2024.11.29.625709↗
- Metabolic regulation of EGFR effector and feedback signaling in pancreatic cancer cells requires K-Ras. First author DOI 10.1016/j.bbrc.2020.09.029↗
- Loss of HIF1A From Pancreatic Cancer Cells Increases Expression of PPP1R1B and Degradation of p53 to Promote Invasion and Metastasis. Co-author DOI 10.1053/j.gastro.2020.07.046↗
- Glutamine depletion regulates Slug to promote EMT and metastasis in pancreatic cancer. Last author DOI 10.1084/jem.20200388↗
- Golgi Acidification by NHE7 Regulates Cytosolic pH Homeostasis in Pancreatic Cancer Cells. Last author DOI 10.1158/2159-8290.cd-19-1007↗
- Human pancreatic cancer tumors are nutrient poor and tumor cells actively scavenge extracellular protein. Co-author DOI 10.1158/0008-5472.can-14-2211↗
Grants 9
- Macropinocytosis: A Paradigm Shift from Metabolic Inhibition to Lethal Hyper-Activation in PDAC AWARD lustgarten:2026-cosimo-commisso-phd-macropinocytosis-a-paradigm-shift-from-m
- Macropinocytosis Inhibition as a Glutamine Mimetic Sensitization Strategy in Pancreatic Cancer R01CA272433↗
- Discovery of Novel Inhibitors Targeting trans-Golgi Network Acidification in Pancreatic Cancer R01CA276193↗
- Regulation of Nutrient Stress-Induced Macropinocytosis in Pancreatic Ductal Adenocarcinoma R01CA207189↗
- Regulation and Function of Stromal Macropinocytosis in Pancreatic Ductal Adenocarcinoma R01CA254806↗
- The Aging Pancreas as a Pro-tumorigenic Niche R21AG087544↗
- The Macropinosome: Uncovering the Molecular Anatomy of an Oncogene-driven Organelle R21CA243701↗
- Targeting macropinocytosis via Na+/H+ exchanger inhibition in PDAC AWARD pancan:2015-cosimo-commisso-phd-targeting-macropinocytosis-via-na-h-exch
- Pancreatic Cancer, Macropinocytosis and Nutrient Internalization AWARD pancan:2011-cosimo-commisso-phd-pancreatic-cancer-macropinocytosis-and-n
Closest collaborators
The 25 strongest of 41 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Yijuan Zhang · 8 shared papers, 2020–2026 · strength 1.50
- Paper The Aging Microenvironment Is a Determinant of Immune Exclusion and Metastatic Fate in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1904↗
- Paper Tumor-derived SAA1-TLR4 signaling drives tumor-to-muscle communication in pancreatic cancer cachexia DOI 10.64898/2026.09.04.748998↗
- Paper Macropinocytosis maintains CAF subtype identity under metabolic stress in pancreatic cancer. DOI 10.1016/j.ccell.2025.06.021↗
- Susanne Heynen-Genel · 1 shared grant, 2026 · strength 1.26
- Cheska Marie Galapate · 5 shared papers, 2024–2026 · strength 1.07
- Paper The Aging Microenvironment Is a Determinant of Immune Exclusion and Metastatic Fate in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1904↗
- Paper Macropinocytosis maintains CAF subtype identity under metabolic stress in pancreatic cancer. DOI 10.1016/j.ccell.2025.06.021↗
- Paper PI5P4Kα promotes glucose and iron acquisition to support metabolic fitness in pancreatic cancer. DOI 10.1016/j.celrep.2025.116199↗
- Andrew M. Lowy · 5 shared papers, 2020–2025 · strength 0.86
- Paper Macropinocytosis maintains CAF subtype identity under metabolic stress in pancreatic cancer. DOI 10.1016/j.ccell.2025.06.021↗
- Paper Macropinocytosis controls metabolic stress-driven CAF subtype identity in pancreatic cancer DOI 10.1101/2024.11.29.625709↗
- Paper Glutamine mimicry suppresses tumor progression through asparagine metabolism in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-023-00649-1↗
- Linda M. Bradley · 3 shared papers, 2024–2025 · strength 0.60
- Paper Macropinocytosis maintains CAF subtype identity under metabolic stress in pancreatic cancer. DOI 10.1016/j.ccell.2025.06.021↗
- Paper Disabling PSGL-1 abrogates immune suppression and resistance to PD-1 blockade in pancreatic cancer DOI 10.1101/2025.05.22.655365↗
- Paper Macropinocytosis controls metabolic stress-driven CAF subtype identity in pancreatic cancer DOI 10.1101/2024.11.29.625709↗
- Jennifer L. Hope · 3 shared papers, 2024–2025 · strength 0.60
- Paper Macropinocytosis maintains CAF subtype identity under metabolic stress in pancreatic cancer. DOI 10.1016/j.ccell.2025.06.021↗
- Paper Disabling PSGL-1 abrogates immune suppression and resistance to PD-1 blockade in pancreatic cancer DOI 10.1101/2025.05.22.655365↗
- Paper Macropinocytosis controls metabolic stress-driven CAF subtype identity in pancreatic cancer DOI 10.1101/2024.11.29.625709↗
- Anindya Bagchi · 4 shared papers, 2020–2025 · strength 0.59
- Paper Macropinocytosis maintains CAF subtype identity under metabolic stress in pancreatic cancer. DOI 10.1016/j.ccell.2025.06.021↗
- Paper Macropinocytosis controls metabolic stress-driven CAF subtype identity in pancreatic cancer DOI 10.1101/2024.11.29.625709↗
- Paper Glutamine depletion regulates Slug to promote EMT and metastasis in pancreatic cancer. DOI 10.1084/jem.20200388↗
- Maria Victoria Recouvreux · 3 shared papers, 2020–2024 · strength 0.41
- Paper Glutamine mimicry suppresses tumor progression through asparagine metabolism in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-023-00649-1↗
- Paper Glutamine depletion regulates Slug to promote EMT and metastasis in pancreatic cancer. DOI 10.1084/jem.20200388↗
- Paper Golgi Acidification by NHE7 Regulates Cytosolic pH Homeostasis in Pancreatic Cancer Cells. DOI 10.1158/2159-8290.cd-19-1007↗
- Yoav Altman · 2 shared papers, 2025 · strength 0.40
- Ling Li · 2 shared papers, 2025 · strength 0.40
- Hervé Tiriac · 2 shared papers, 2024–2025 · strength 0.35
- Priyanka Gupta · 1 shared paper, 2026 · strength 0.29
- Szu-Wei Lee · 1 shared paper, 2020 · strength 0.24
- Koen M.O. Galenkamp · 2 shared papers, 2020 · strength 0.23
- Gabriele Guarnaccia · 1 shared paper, 2026 · strength 0.21
- Alessandra Sacco · 1 shared paper, 2026 · strength 0.21
- Chantal Pauli · 1 shared paper, 2025 · strength 0.20
- Brooke M. Emerling · 1 shared paper, 2025 · strength 0.20
- Gurpreet K. Arora · 1 shared paper, 2025 · strength 0.20
- Katelyn T. Byrne · 1 shared paper, 2025 · strength 0.20
- Divya Sood · 1 shared paper, 2025 · strength 0.17
- Bharti Garg · 1 shared paper, 2025 · strength 0.17
- Nirakar Rajbhandari · 1 shared paper, 2025 · strength 0.17
- Pnina Brodt · 1 shared paper, 2025 · strength 0.17
- Kevin Christian Montecillo Gulay · 1 shared paper, 2025 · strength 0.17
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.