ResearcherChinese Academy of Medical Sciences & Peking Union Medical CollegeActive in the last 2 years
Wenming Wu
Matched on ORCID iD 0000-0003-0691-8333; OpenAlex author ID A5101755677. Sources: OpenAlex.
Their work
- Papers
- 23 (first author 0, last author 11)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 62
2012: 1 paper · 2013: none · 2014: 2 papers · 2015: 1 paper · 2016: 1 paper · 2017: none · 2018: none · 2019: 3 papers · 2020: 1 paper · 2021: 3 papers · 2022: 1 paper · 2023: 1 paper · 2024: 2 papers · 2025: 2 papers · 2026: 5 papers
Papers 23
- Tumor-educated adipocytes promote muscle wasting via EV-packaged miR-221-3p targeting IRS1 in pancreatic cancer cachexia. Last author DOI 10.1016/j.canlet.2026.218795↗
- Bioengineering advances in pancreatic cancer organoids for reproducible tumor microenvironment modeling. Last author DOI 10.1016/j.isci.2026.116487↗
- Drug-associated pancreatic cancer: insights from real-world pharmacovigilance and network pharmacology. Last author DOI 10.1080/14740338.2025.2469273↗
- Neutrophil extracellular traps induced by tumour-derived NGAL reprogram CAF subtypes via ITGA5 to promote tumour progression in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2025-337720↗
- Clinical quality of large language model answers to questions about pancreatic cancer Last author DOI 10.21203/rs.3.rs-10624412/v1↗
- Pancreatic cancer cachexia: A systemic consequence of multi-organ interactions. Last author DOI 10.1016/j.hlife.2025.05.002↗
- Oncogenic KRAS Promotes Ferroptosis in Pancreatic Cancer Through Regulation of the Fosl1-Tfrc Axis. Last author DOI 10.1097/mpa.0000000000002426↗
- Gut resistome profiling reveals high diversity and fluctuations in pancreatic cancer cohorts. Last author DOI 10.3389/fcimb.2024.1354234↗
- Role and functional mechanisms of IL‑17/IL‑17R signaling in pancreatic cancer (Review). Last author DOI 10.3892/or.2024.8803↗
- Prognostic value of preoperative [68 Ga]Ga-FAPI-04 PET/CT in patients with resectable pancreatic ductal adenocarcinoma in correlation with immunohistological c… Co-author DOI 10.1007/s00259-022-06100-4↗
- Early Detection of Pancreatic Cancer: Are We Ready for Prime Time? Co-author DOI 10.1053/j.gastro.2022.07.072↗
- Comparison of minimal invasive versus open radical antegrade modular pancreatosplenectomy (RAMPS) for pancreatic ductal adenocarcinoma: a single center retrosp… Co-author DOI 10.1007/s00464-020-07938-1↗
- Interleukin-17 activates and synergizes with the notch signaling pathway in the progression of pancreatic ductal adenocarcinoma. Last author DOI 10.1016/j.canlet.2021.03.003↗
- Mutant Kras co-opts a proto-oncogenic enhancer network in inflammation-induced metaplastic progenitor cells to initiate pancreatic cancer. Co-author DOI 10.1038/s43018-020-00134-z↗
- Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor microenvironment. Co-author DOI 10.1007/s00262-020-02542-7↗
- Single-cell RNA-seq highlights intra-tumoral heterogeneity and malignant progression in pancreatic ductal adenocarcinoma. Last author DOI 10.1038/s41422-019-0195-y↗
- Author Correction: Single-cell RNA-seq highlights intra-tumoral heterogeneity and malignant progression in pancreatic ductal adenocarcinoma. Last author DOI 10.1038/s41422-019-0212-1↗
- [The effect of para-aortic lymph node metastasis on the resectability of pancreatic cancer]. Co-author DOI 10.3760/cma.j.issn.0529-5815.2019.01.014↗
- Advances in understanding the molecular mechanism of pancreatic cancer metastasis. Co-author DOI 10.1016/s1499-3872(15)60033-9↗
- Catechol-O-methyltransferase, a new target for pancreatic cancer therapy. Co-author DOI 10.1111/cas.12648↗
- The prognostic value of Cyclin B1 in pancreatic cancer. Co-author DOI 10.1007/s12032-014-0107-4↗
- Prognostic significance of epidermal growth factor-like domain 7 in pancreatic cancer. Co-author DOI 10.1016/s1499-3872(14)60272-1↗
- Increased COMT expression in pancreatic cancer and correlation with clinicopathologic parameters. Co-author DOI 10.1007/s11427-012-4375-y↗
Closest collaborators
The 25 strongest of 62 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Yupei Zhao · 9 shared papers, 2012–2026 · strength 0.95
- Paper Neutrophil extracellular traps induced by tumour-derived NGAL reprogram CAF subtypes via ITGA5 to promote tumour progre… DOI 10.1136/gutjnl-2025-337720↗
- Paper [The effect of para-aortic lymph node metastasis on the resectability of pancreatic cancer]. DOI 10.3760/cma.j.issn.0529-5815.2019.01.014↗
- Paper Comparison of minimal invasive versus open radical antegrade modular pancreatosplenectomy (RAMPS) for pancreatic ductal… DOI 10.1007/s00464-020-07938-1↗
- Aoyi Xiao · 3 shared papers, 2025–2026 · strength 0.78
- Paper Pancreatic cancer cachexia: A systemic consequence of multi-organ interactions. DOI 10.1016/j.hlife.2025.05.002↗
- Paper Tumor-educated adipocytes promote muscle wasting via EV-packaged miR-221-3p targeting IRS1 in pancreatic cancer cachexi… DOI 10.1016/j.canlet.2026.218795↗
- Paper Neutrophil extracellular traps induced by tumour-derived NGAL reprogram CAF subtypes via ITGA5 to promote tumour progre… DOI 10.1136/gutjnl-2025-337720↗
- Xudong Liu · 3 shared papers, 2024–2026 · strength 0.76
- Paper Pancreatic cancer cachexia: A systemic consequence of multi-organ interactions. DOI 10.1016/j.hlife.2025.05.002↗
- Paper Neutrophil extracellular traps induced by tumour-derived NGAL reprogram CAF subtypes via ITGA5 to promote tumour progre… DOI 10.1136/gutjnl-2025-337720↗
- Paper Gut resistome profiling reveals high diversity and fluctuations in pancreatic cancer cohorts. DOI 10.3389/fcimb.2024.1354234↗
- Xianze Wang · 3 shared papers, 2021–2026 · strength 0.72
- Paper Role and functional mechanisms of IL‑17/IL‑17R signaling in pancreatic cancer (Review). DOI 10.3892/or.2024.8803↗
- Paper Neutrophil extracellular traps induced by tumour-derived NGAL reprogram CAF subtypes via ITGA5 to promote tumour progre… DOI 10.1136/gutjnl-2025-337720↗
- Paper Interleukin-17 activates and synergizes with the notch signaling pathway in the progression of pancreatic ductal adenoc… DOI 10.1016/j.canlet.2021.03.003↗
- Junya Peng · 5 shared papers, 2019–2026 · strength 0.71
- Paper Bioengineering advances in pancreatic cancer organoids for reproducible tumor microenvironment modeling. DOI 10.1016/j.isci.2026.116487↗
- Paper Interleukin-17 activates and synergizes with the notch signaling pathway in the progression of pancreatic ductal adenoc… DOI 10.1016/j.canlet.2021.03.003↗
- Paper Mutant Kras co-opts a proto-oncogenic enhancer network in inflammation-induced metaplastic progenitor cells to initiate… DOI 10.1038/s43018-020-00134-z↗
- Renyu Chen · 2 shared papers, 2026 · strength 0.61
- Paper Bioengineering advances in pancreatic cancer organoids for reproducible tumor microenvironment modeling. DOI 10.1016/j.isci.2026.116487↗
- Paper Neutrophil extracellular traps induced by tumour-derived NGAL reprogram CAF subtypes via ITGA5 to promote tumour progre… DOI 10.1136/gutjnl-2025-337720↗
- Wenze Wang · 5 shared papers, 2015–2026 · strength 0.58
- Paper Neutrophil extracellular traps induced by tumour-derived NGAL reprogram CAF subtypes via ITGA5 to promote tumour progre… DOI 10.1136/gutjnl-2025-337720↗
- Paper Interleukin-17 activates and synergizes with the notch signaling pathway in the progression of pancreatic ductal adenoc… DOI 10.1016/j.canlet.2021.03.003↗
- Paper Mutant Kras co-opts a proto-oncogenic enhancer network in inflammation-induced metaplastic progenitor cells to initiate… DOI 10.1038/s43018-020-00134-z↗
- Xianlin Han · 2 shared papers, 2026 · strength 0.55
- Taiping Zhang · 6 shared papers, 2014–2023 · strength 0.53
- Paper Prognostic value of preoperative [68 Ga]Ga-FAPI-04 PET/CT in patients with resectable pancreatic ductal adenocarcinoma … DOI 10.1007/s00259-022-06100-4↗
- Paper Comparison of minimal invasive versus open radical antegrade modular pancreatosplenectomy (RAMPS) for pancreatic ductal… DOI 10.1007/s00464-020-07938-1↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Qiang Xu · 1 shared paper, 2026 · strength 0.43
- Hao Xie · 1 shared paper, 2026 · strength 0.43
- Huanwen Wu · 3 shared papers, 2015–2026 · strength 0.40
- Paper Tumor-educated adipocytes promote muscle wasting via EV-packaged miR-221-3p targeting IRS1 in pancreatic cancer cachexi… DOI 10.1016/j.canlet.2026.218795↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Paper Catechol-O-methyltransferase, a new target for pancreatic cancer therapy. DOI 10.1111/cas.12648↗
- Huijia Zhao · 1 shared paper, 2025 · strength 0.37
- Wanli Liu · 1 shared paper, 2024 · strength 0.36
- Junchao Guo · 5 shared papers, 2014–2021 · strength 0.36
- Paper Comparison of minimal invasive versus open radical antegrade modular pancreatosplenectomy (RAMPS) for pancreatic ductal… DOI 10.1007/s00464-020-07938-1↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Paper Single-cell RNA-seq highlights intra-tumoral heterogeneity and malignant progression in pancreatic ductal adenocarcinom… DOI 10.1038/s41422-019-0195-y↗
- Menghua Dai · 4 shared papers, 2019–2021 · strength 0.35
- Paper Comparison of minimal invasive versus open radical antegrade modular pancreatosplenectomy (RAMPS) for pancreatic ductal… DOI 10.1007/s00464-020-07938-1↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Paper Single-cell RNA-seq highlights intra-tumoral heterogeneity and malignant progression in pancreatic ductal adenocarcinom… DOI 10.1038/s41422-019-0195-y↗
- Ruohan Cui · 1 shared paper, 2026 · strength 0.29
- Hongmei Dai · 3 shared papers, 2015–2021 · strength 0.29
- Paper [The effect of para-aortic lymph node metastasis on the resectability of pancreatic cancer]. DOI 10.3760/cma.j.issn.0529-5815.2019.01.014↗
- Paper Interleukin-17 activates and synergizes with the notch signaling pathway in the progression of pancreatic ductal adenoc… DOI 10.1016/j.canlet.2021.03.003↗
- Paper Catechol-O-methyltransferase, a new target for pancreatic cancer therapy. DOI 10.1111/cas.12648↗
- Quan Liao · 3 shared papers, 2019–2021 · strength 0.28
- Paper Comparison of minimal invasive versus open radical antegrade modular pancreatosplenectomy (RAMPS) for pancreatic ductal… DOI 10.1007/s00464-020-07938-1↗
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Paper Single-cell RNA-seq highlights intra-tumoral heterogeneity and malignant progression in pancreatic ductal adenocarcinom… DOI 10.1038/s41422-019-0195-y↗
- Jingcheng Zhang · 1 shared paper, 2025 · strength 0.27
- Wanli Liu · 1 shared paper, 2026 · strength 0.26
- Li Zhou · 5 shared papers, 2012–2020 · strength 0.25
- Paper Combined blockade of TGf-β1 and GM-CSF improves chemotherapeutic effects for pancreatic cancer by modulating tumor micr… DOI 10.1007/s00262-020-02542-7↗
- Paper Catechol-O-methyltransferase, a new target for pancreatic cancer therapy. DOI 10.1111/cas.12648↗
- Paper The prognostic value of Cyclin B1 in pancreatic cancer. DOI 10.1007/s12032-014-0107-4↗
- W.T. Liu · 1 shared paper, 2026 · strength 0.25
- Mingyang Liu · 1 shared paper, 2026 · strength 0.25
- Lei You · 5 shared papers, 2012–2016 · strength 0.25
- Paper Advances in understanding the molecular mechanism of pancreatic cancer metastasis. DOI 10.1016/s1499-3872(15)60033-9↗
- Paper Catechol-O-methyltransferase, a new target for pancreatic cancer therapy. DOI 10.1111/cas.12648↗
- Paper The prognostic value of Cyclin B1 in pancreatic cancer. DOI 10.1007/s12032-014-0107-4↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.