ResearcherHokkaido UniversityNo recent work listed
Hisataka Sabe
Matched on ORCID iD 0000-0002-6261-7095; OpenAlex author ID A5000830028. Sources: OpenAlex.
Their work
- Papers
- 4 (first author 0, last author 4)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 7
2019: 1 paper · 2020: 1 paper · 2021: 1 paper · 2022: 1 paper
Papers 4
- ARF6-AMAP1 pathway induces pancreatic cancer cells to express immunosuppressive chemokines Last author DOI 10.21203/rs.3.rs-1897319/v1↗
- Inhibition of mutant KRAS-driven overexpression of ARF6 and MYC by an eIF4A inhibitor drug improves the effects of anti-PD-1 immunotherapy for pancreatic cance… Last author DOI 10.1186/s12964-021-00733-y↗
- High expression of AMAP1, an ARF6 effector, is associated with elevated levels of PD-L1 and fibrosis of pancreatic cancer. Last author DOI 10.1186/s12964-020-00608-8↗
- ARF6 and AMAP1 are major targets of KRAS and TP53 mutations to promote invasion, PD-L1 dynamics, and immune evasion of pancreatic cancer. Last author DOI 10.1073/pnas.1901765116↗
Closest collaborators
The 7 strongest of 7 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Akio Tsutaho · 4 shared papers, 2019–2022 · strength 0.48
- Paper ARF6-AMAP1 pathway induces pancreatic cancer cells to express immunosuppressive chemokines DOI 10.21203/rs.3.rs-1897319/v1↗
- Paper Inhibition of mutant KRAS-driven overexpression of ARF6 and MYC by an eIF4A inhibitor drug improves the effects of anti… DOI 10.1186/s12964-021-00733-y↗
- Paper High expression of AMAP1, an ARF6 effector, is associated with elevated levels of PD-L1 and fibrosis of pancreatic canc… DOI 10.1186/s12964-020-00608-8↗
- Ari Hashimoto · 4 shared papers, 2019–2022 · strength 0.48
- Paper ARF6-AMAP1 pathway induces pancreatic cancer cells to express immunosuppressive chemokines DOI 10.21203/rs.3.rs-1897319/v1↗
- Paper Inhibition of mutant KRAS-driven overexpression of ARF6 and MYC by an eIF4A inhibitor drug improves the effects of anti… DOI 10.1186/s12964-021-00733-y↗
- Paper High expression of AMAP1, an ARF6 effector, is associated with elevated levels of PD-L1 and fibrosis of pancreatic canc… DOI 10.1186/s12964-020-00608-8↗
- Satoshi Hirano · 4 shared papers, 2019–2022 · strength 0.48
- Paper ARF6-AMAP1 pathway induces pancreatic cancer cells to express immunosuppressive chemokines DOI 10.21203/rs.3.rs-1897319/v1↗
- Paper Inhibition of mutant KRAS-driven overexpression of ARF6 and MYC by an eIF4A inhibitor drug improves the effects of anti… DOI 10.1186/s12964-021-00733-y↗
- Paper High expression of AMAP1, an ARF6 effector, is associated with elevated levels of PD-L1 and fibrosis of pancreatic canc… DOI 10.1186/s12964-020-00608-8↗
- Shigeru Hashimoto · 4 shared papers, 2019–2022 · strength 0.48
- Paper ARF6-AMAP1 pathway induces pancreatic cancer cells to express immunosuppressive chemokines DOI 10.21203/rs.3.rs-1897319/v1↗
- Paper Inhibition of mutant KRAS-driven overexpression of ARF6 and MYC by an eIF4A inhibitor drug improves the effects of anti… DOI 10.1186/s12964-021-00733-y↗
- Paper High expression of AMAP1, an ARF6 effector, is associated with elevated levels of PD-L1 and fibrosis of pancreatic canc… DOI 10.1186/s12964-020-00608-8↗
- Yoshihiro Nishikawa · 2 shared papers, 2019–2022 · strength 0.22
- Yuzo Kodama · 2 shared papers, 2019–2022 · strength 0.22
- Yusuke Mizukami · 1 shared paper, 2019 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.