ResearcherInstitute for Biomedical Research and InnovationNo recent work listed
Melchiorre Cervello
Matched on ORCID iD 0000-0003-4923-7220; OpenAlex author ID A5070243247. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 0, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 8
2015: 1 paper · 2016: none · 2017: none · 2018: 2 papers · 2019: 3 papers · 2020: 1 paper · 2021: 1 paper · 2022: 3 papers · 2023: 1 paper
Papers 12
- Effects of chloroquine and hydroxychloroquine on the sensitivity of pancreatic cancer cells to targeted therapies. Last author DOI 10.1016/j.jbior.2022.100917↗
- Effects of the MDM2 inhibitor Nutlin-3a on sensitivity of pancreatic cancer cells to berberine and modified berberines in the presence and absence of WT-TP53. Co-author DOI 10.1016/j.jbior.2021.100840↗
- Effects of the Mutant TP53 Reactivator APR-246 on Therapeutic Sensitivity of Pancreatic Cancer Cells in the Presence and Absence of WT-TP53. Co-author DOI 10.3390/cells11050794↗
- Effects of TP53 Mutations and miRs on Immune Responses in the Tumor Microenvironment Important in Pancreatic Cancer Progression. Last author DOI 10.3390/cells11142155↗
- Sensitivity of pancreatic cancer cells to chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals can be regulated by WT-TP53. Co-author DOI 10.1016/j.jbior.2020.100780↗
- Abilities of β-Estradiol to interact with chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals and alter the proliferation of pancreatic c… Co-author DOI 10.1016/j.jbior.2019.100672↗
- Abilities of berberine and chemically modified berberines to inhibit proliferation of pancreatic cancer cells. Co-author DOI 10.1016/j.jbior.2018.10.003↗
- Effects of the MDM-2 inhibitor Nutlin-3a on PDAC cells containing and lacking WT-TP53 on sensitivity to chemotherapy, signal transduction inhibitors and nutrac… Co-author DOI 10.1016/j.jbior.2019.03.002↗
- Abilities of berberine and chemically modified berberines to interact with metformin and inhibit proliferation of pancreatic cancer cells. Co-author DOI 10.1016/j.jbior.2019.04.003↗
- Metformin influences drug sensitivity in pancreatic cancer cells. Co-author DOI 10.1016/j.jbior.2018.02.002↗
- Introduction of WT-TP53 into pancreatic cancer cells alters sensitivity to chemotherapeutic drugs, targeted therapeutics and nutraceuticals. Co-author DOI 10.1016/j.jbior.2018.06.002↗
- Roles of EGFR and KRAS and their downstream signaling pathways in pancreatic cancer and pancreatic cancer stem cells. Co-author DOI 10.1016/j.jbior.2015.06.003↗
Closest collaborators
The 8 strongest of 8 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Stephen Lee Abrams · 12 shared papers, 2015–2023 · strength 1.22
- Paper Effects of chloroquine and hydroxychloroquine on the sensitivity of pancreatic cancer cells to targeted therapies. DOI 10.1016/j.jbior.2022.100917↗
- Paper Effects of TP53 Mutations and miRs on Immune Responses in the Tumor Microenvironment Important in Pancreatic Cancer Pro… DOI 10.3390/cells11142155↗
- Paper Effects of the MDM2 inhibitor Nutlin-3a on sensitivity of pancreatic cancer cells to berberine and modified berberines … DOI 10.1016/j.jbior.2021.100840↗
- James Andrew McCubrey · 12 shared papers, 2015–2023 · strength 1.22
- Paper Effects of chloroquine and hydroxychloroquine on the sensitivity of pancreatic cancer cells to targeted therapies. DOI 10.1016/j.jbior.2022.100917↗
- Paper Effects of TP53 Mutations and miRs on Immune Responses in the Tumor Microenvironment Important in Pancreatic Cancer Pro… DOI 10.3390/cells11142155↗
- Paper Effects of the MDM2 inhibitor Nutlin-3a on sensitivity of pancreatic cancer cells to berberine and modified berberines … DOI 10.1016/j.jbior.2021.100840↗
- Saverio Candido · 9 shared papers, 2015–2022 · strength 0.73
- Paper Effects of the MDM2 inhibitor Nutlin-3a on sensitivity of pancreatic cancer cells to berberine and modified berberines … DOI 10.1016/j.jbior.2021.100840↗
- Paper Sensitivity of pancreatic cancer cells to chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals can… DOI 10.1016/j.jbior.2020.100780↗
- Paper Abilities of β-Estradiol to interact with chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals and… DOI 10.1016/j.jbior.2019.100672↗
- Agnieszka Gizak · 7 shared papers, 2018–2022 · strength 0.61
- Paper Effects of the Mutant TP53 Reactivator APR-246 on Therapeutic Sensitivity of Pancreatic Cancer Cells in the Presence an… DOI 10.3390/cells11050794↗
- Paper Sensitivity of pancreatic cancer cells to chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals can… DOI 10.1016/j.jbior.2020.100780↗
- Paper Abilities of β-Estradiol to interact with chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals and… DOI 10.1016/j.jbior.2019.100672↗
- Shaw M. Akula · 6 shared papers, 2019–2022 · strength 0.61
- Paper Effects of the MDM2 inhibitor Nutlin-3a on sensitivity of pancreatic cancer cells to berberine and modified berberines … DOI 10.1016/j.jbior.2021.100840↗
- Paper Effects of the Mutant TP53 Reactivator APR-246 on Therapeutic Sensitivity of Pancreatic Cancer Cells in the Presence an… DOI 10.3390/cells11050794↗
- Paper Sensitivity of pancreatic cancer cells to chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals can… DOI 10.1016/j.jbior.2020.100780↗
- Marco Falasca · 2 shared papers, 2018–2020 · strength 0.15
- Paper Abilities of β-Estradiol to interact with chemotherapeutic drugs, signal transduction inhibitors and nutraceuticals and… DOI 10.1016/j.jbior.2019.100672↗
- Paper Introduction of WT-TP53 into pancreatic cancer cells alters sensitivity to chemotherapeutic drugs, targeted therapeutic… DOI 10.1016/j.jbior.2018.06.002↗
- Przemysław Duda · 1 shared paper, 2022 · strength 0.13
- Timothy Louis Fitzgerald · 1 shared paper, 2015 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.