ResearcherNorthwestern UniversityNo recent work listed

Beatriz Sosa-Pineda

ORCID-verified (high confidence, 0.95), identity_conf@1As of 8 November 2026

Matched on ORCID iD 0000-0001-6872-5444; OpenAlex author ID A5085915684; NIH RePORTER profile ID 6796053. Sources: OpenAlex.

Their work

Papers: 2 (first author 0, last author 1)
Trials: 0
Grants: 1
Labs led: 2
Collaborators: 4

Papers, trials and grants by year

2014: 1 paper · 2015: none · 2016: none · 2017: 1 paper · 2018: 1 grant

Papers 2

Grants 1

Development, cellular plasticity and homeostasis of the exocrine pancreas R01DK106266 ↗

Closest collaborators

The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Yiannis Drosos · 2 shared papers, 2014–2017 · strength 0.10
- Paper ATM-deficiency increases genomic instability and metastatic potential in a mouse model of pancreatic cancer. DOI 10.1038/s41598-017-11661-8 ↗
- Paper Fibrogenesis in pancreatic cancer is a dynamic process regulated by macrophage-stellate cell interaction. DOI 10.1038/labinvest.2014.10 ↗
Vaibhav Sahai · 1 shared paper, 2017 · strength 0.06
- Paper ATM-deficiency increases genomic instability and metastatic potential in a mouse model of pancreatic cancer. DOI 10.1038/s41598-017-11661-8 ↗
Chanjuan Shi · 1 shared paper, 2014 · strength 0.04
- Paper Fibrogenesis in pancreatic cancer is a dynamic process regulated by macrophage-stellate cell interaction. DOI 10.1038/labinvest.2014.10 ↗
Anna L. Means · 1 shared paper, 2014 · strength 0.04
- Paper Fibrogenesis in pancreatic cancer is a dynamic process regulated by macrophage-stellate cell interaction. DOI 10.1038/labinvest.2014.10 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.