ResearcherIstituto PasteurActive in the last 2 years
Mara Cirone
Matched on ORCID iD 0000-0002-2207-9624; OpenAlex author ID A5003089486. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 1, last author 8)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 5
2017: 1 paper · 2018: none · 2019: 1 paper · 2020: none · 2021: 1 paper · 2022: 2 papers · 2023: none · 2024: 1 paper · 2025: 2 papers · 2026: 1 paper
Papers 9
- Correction: Di Crosta et al. Valemetostat-SAHA-Driven Acetylation of p53 via SET/TAF-Iβ Displacement and p300 Activation Modulates Cell Cycle Regulators in Pan… Last author DOI 10.3390/biomedicines14051159↗
- Mechanistic insights and therapeutic strategies for targeting autophagy in pancreatic ductal adenocarcinoma. First author DOI 10.1007/s12672-025-02400-x↗
- Valemetostat-SAHA-Driven Acetylation of p53 via SET/TAF-Iβ Displacement and p300 Activation Modulates Cell Cycle Regulators in Pancreatic Cancer Cells. Last author DOI 10.3390/biomedicines13092279↗
- SAHA/5-AZA Enhances Acetylation and Degradation of mutp53, Upregulates p21 and Downregulates c-Myc and BRCA-1 in Pancreatic Cancer Cells. Last author DOI 10.3390/ijms25137020↗
- c-Myc Sustains Pancreatic Cancer Cell Survival and mutp53 Stability through the Mevalonate Pathway. Last author DOI 10.3390/biomedicines10102489↗
- VPA and TSA Interrupt the Interplay between mutp53 and HSP70, Leading to CHK1 and RAD51 Down-Regulation and Sensitizing Pancreatic Cancer Cells to AZD2461 PARP… Last author DOI 10.3390/ijms23042268↗
- PGE2 Released by Pancreatic Cancer Cells Undergoing ER Stress Transfers the Stress to DCs Impairing Their Immune Function. Last author DOI 10.1158/1535-7163.mct-20-0699↗
- Mutant p53, Stabilized by Its Interplay with HSP90, Activates a Positive Feed-Back Loop Between NRF2 and p62 that Induces Chemo-Resistance to Apigenin in Pancr… Last author DOI 10.3390/cancers11050703↗
- Histone deacetylase inhibitors VPA and TSA induce apoptosis and autophagy in pancreatic cancer cells. Last author DOI 10.1007/s13402-017-0314-z↗
Closest collaborators
The 5 strongest of 5 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Maria Saveria Gilardini Montani · 8 shared papers, 2017–2026 · strength 1.54
- Paper Correction: Di Crosta et al. Valemetostat-SAHA-Driven Acetylation of p53 via SET/TAF-Iβ Displacement and p300 Activatio… DOI 10.3390/biomedicines14051159↗
- Paper Valemetostat-SAHA-Driven Acetylation of p53 via SET/TAF-Iβ Displacement and p300 Activation Modulates Cell Cycle Regula… DOI 10.3390/biomedicines13092279↗
- Paper SAHA/5-AZA Enhances Acetylation and Degradation of mutp53, Upregulates p21 and Downregulates c-Myc and BRCA-1 in Pancre… DOI 10.3390/ijms25137020↗
- Michele Di Crosta · 3 shared papers, 2024–2026 · strength 0.87
- Paper Correction: Di Crosta et al. Valemetostat-SAHA-Driven Acetylation of p53 via SET/TAF-Iβ Displacement and p300 Activatio… DOI 10.3390/biomedicines14051159↗
- Paper Valemetostat-SAHA-Driven Acetylation of p53 via SET/TAF-Iβ Displacement and p300 Activation Modulates Cell Cycle Regula… DOI 10.3390/biomedicines13092279↗
- Paper SAHA/5-AZA Enhances Acetylation and Degradation of mutp53, Upregulates p21 and Downregulates c-Myc and BRCA-1 in Pancre… DOI 10.3390/ijms25137020↗
- Maria Anele Romeo · 4 shared papers, 2019–2022 · strength 0.59
- Paper c-Myc Sustains Pancreatic Cancer Cell Survival and mutp53 Stability through the Mevalonate Pathway. DOI 10.3390/biomedicines10102489↗
- Paper VPA and TSA Interrupt the Interplay between mutp53 and HSP70, Leading to CHK1 and RAD51 Down-Regulation and Sensitizing… DOI 10.3390/ijms23042268↗
- Paper PGE2 Released by Pancreatic Cancer Cells Undergoing ER Stress Transfers the Stress to DCs Impairing Their Immune Functi… DOI 10.1158/1535-7163.mct-20-0699↗
- Marco Cordani · 1 shared paper, 2025 · strength 0.33
- Federica Michetti · 1 shared paper, 2025 · strength 0.33
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.