ResearcherUniversity of MichiganActive in the last 2 years
Yaqing Zhang
Matched on ORCID iD 0000-0002-9185-4364; OpenAlex author ID A5037667722; NIH RePORTER profile ID 10840179. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 2
- Labs led
- 1
- Collaborators
- 34
2020: 1 paper · 2021: none · 2022: 1 grant · 2023: 1 paper · 2024: 1 paper · 2025: none · 2026: 5 papers, 1 grant
Papers 8
- A stromal PAI1-tPA axis orchestrates immunosuppression in pancreatic cancer. Co-author DOI 10.1126/sciadv.aea6734↗
- Wilms Tumor 1-Expressing Stromal Cells Promote Pancreatic Cancer Progression. Co-author DOI 10.1158/0008-5472.can-25-1014↗
- Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization. Co-author DOI 10.1158/2159-8290.cd-25-0377↗
- Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-25-2001↗
- Targeting CCR1 Remodels the Tumor Microenvironment and Relieves Immunosuppression in Pancreatic Cancer. Co-author DOI 10.1158/2326-6066.cir-25-1300↗
- Notch Signaling Regulates Immunosuppressive Tumor-Associated Macrophage Function in Pancreatic Cancer. Co-author DOI 10.1158/2326-6066.cir-23-0037↗
- WNT signaling in the tumor microenvironment promotes immunosuppression in murine pancreatic cancer. Co-author DOI 10.1084/jem.20220503↗
- Differential Contribution of Pancreatic Fibroblast Subsets to the Pancreatic Cancer Stroma. Co-author DOI 10.1016/j.jcmgh.2020.05.004↗
Grants 2
Closest collaborators
The 25 strongest of 34 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Marina Pasca di Magliano · 8 shared papers, 2020–2026 · strength 1.58
- Paper A stromal PAI1-tPA axis orchestrates immunosuppression in pancreatic cancer. DOI 10.1126/sciadv.aea6734↗
- Paper Wilms Tumor 1-Expressing Stromal Cells Promote Pancreatic Cancer Progression. DOI 10.1158/0008-5472.can-25-1014↗
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Bin Zheng · 1 shared grant, 2026 · strength 1.26
- Eileen S. Carpenter · 6 shared papers, 2023–2026 · strength 1.21
- Paper Wilms Tumor 1-Expressing Stromal Cells Promote Pancreatic Cancer Progression. DOI 10.1158/0008-5472.can-25-1014↗
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Paper Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. DOI 10.1158/2159-8290.cd-25-2001↗
- Filip Bednar · 6 shared papers, 2020–2026 · strength 1.14
- Paper Wilms Tumor 1-Expressing Stromal Cells Promote Pancreatic Cancer Progression. DOI 10.1158/0008-5472.can-25-1014↗
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Paper Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. DOI 10.1158/2159-8290.cd-25-2001↗
- Timothy L. Frankel · 4 shared papers, 2026 · strength 0.86
- Paper Wilms Tumor 1-Expressing Stromal Cells Promote Pancreatic Cancer Progression. DOI 10.1158/0008-5472.can-25-1014↗
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Paper Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. DOI 10.1158/2159-8290.cd-25-2001↗
- Padma Kadiyala · 4 shared papers, 2023–2026 · strength 0.79
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Paper Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. DOI 10.1158/2159-8290.cd-25-2001↗
- Paper Targeting CCR1 Remodels the Tumor Microenvironment and Relieves Immunosuppression in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-25-1300↗
- Katelyn L. Donahue · 4 shared papers, 2023–2026 · strength 0.76
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Paper Targeting CCR1 Remodels the Tumor Microenvironment and Relieves Immunosuppression in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-25-1300↗
- Paper Notch Signaling Regulates Immunosuppressive Tumor-Associated Macrophage Function in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-23-0037↗
- Jiaqi Shi · 3 shared papers, 2026 · strength 0.72
- Paper A stromal PAI1-tPA axis orchestrates immunosuppression in pancreatic cancer. DOI 10.1126/sciadv.aea6734↗
- Paper Wilms Tumor 1-Expressing Stromal Cells Promote Pancreatic Cancer Progression. DOI 10.1158/0008-5472.can-25-1014↗
- Paper Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. DOI 10.1158/2159-8290.cd-25-2001↗
- Ahmed M. Elhossiny · 3 shared papers, 2026 · strength 0.63
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Paper Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. DOI 10.1158/2159-8290.cd-25-2001↗
- Paper Targeting CCR1 Remodels the Tumor Microenvironment and Relieves Immunosuppression in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-25-1300↗
- Wei Yan · 3 shared papers, 2024–2026 · strength 0.61
- Paper Wilms Tumor 1-Expressing Stromal Cells Promote Pancreatic Cancer Progression. DOI 10.1158/0008-5472.can-25-1014↗
- Paper Targeting CCR1 Remodels the Tumor Microenvironment and Relieves Immunosuppression in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-25-1300↗
- Paper Notch Signaling Regulates Immunosuppressive Tumor-Associated Macrophage Function in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-23-0037↗
- Rosa Elena Menjivar · 3 shared papers, 2023–2026 · strength 0.54
- Paper Targeting CCR1 Remodels the Tumor Microenvironment and Relieves Immunosuppression in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-25-1300↗
- Paper Notch Signaling Regulates Immunosuppressive Tumor-Associated Macrophage Function in Pancreatic Cancer. DOI 10.1158/2326-6066.cir-23-0037↗
- Paper WNT signaling in the tumor microenvironment promotes immunosuppression in murine pancreatic cancer. DOI 10.1084/jem.20220503↗
- Brian D. Griffith · 2 shared papers, 2026 · strength 0.43
- Paper Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Pola… DOI 10.1158/2159-8290.cd-25-0377↗
- Paper Asynchronous Evolution of Epithelium and Stroma Differentiates Precursor Lesions from Pancreatic Cancer. DOI 10.1158/2159-8290.cd-25-2001↗
- Emily L. Lasse Opsahl · 2 shared papers, 2026 · strength 0.43
- Costas Andreas Lyssiotis · 2 shared papers, 2026 · strength 0.40
- Wenting Du · 2 shared papers, 2023–2024 · strength 0.34
- Kyoung Eun Lee · 1 shared paper, 2026 · strength 0.28
- Ashley M. Mello · 1 shared paper, 2026 · strength 0.28
- Tenzin Ngodup · 1 shared paper, 2026 · strength 0.28
- Sean Matthew Hannifin · 1 shared paper, 2026 · strength 0.28
- Allison Bischoff · 1 shared paper, 2026 · strength 0.24
- Holly Attebury · 1 shared paper, 2026 · strength 0.22
- Elana Judith Fertig · 1 shared paper, 2026 · strength 0.21
- Julianne M. Szczepanski · 1 shared paper, 2026 · strength 0.21
- Mariana T. Ruckert · 1 shared paper, 2026 · strength 0.21
- Anirban Maitra · 1 shared paper, 2026 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.