ResearcherVita-Salute San Raffaele UniversityActive in the last 2 years
Jakob Robert Izbicki
Matched on ORCID iD 0000-0001-6807-8224; OpenAlex author ID A5039983309, A5109329116. Sources: ClinicalTrials.gov.
Their work
- Papers
- 70 (first author 1, last author 3)
- Trials
- 1 (principal investigator on 1)
- Grants
- 0
- Labs led
- 1
- Collaborators
- 160
2011: 5 papers · 2012: 4 papers · 2013: 2 papers · 2014: 9 papers, 1 trial · 2015: 3 papers · 2016: 4 papers · 2017: 2 papers · 2018: 2 papers · 2019: 6 papers · 2020: 2 papers · 2021: 11 papers · 2022: 4 papers · 2023: 8 papers · 2024: 3 papers · 2025: 5 papers
Papers 50 of 70
- Routine Imaging or Symptomatic Follow-Up After Resection of Pancreatic Adenocarcinoma. Co-author DOI 10.1001/jamasurg.2024.5024↗
- Polymorphisms within autophagy-related genes as susceptibility biomarkers for pancreatic cancer: A meta-analysis of three large European cohorts and functional… Co-author DOI 10.1002/ijc.35196↗
- Patient-derived tumor organoids highlight the potential of precision medicine in managing pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.35443↗
- Author Correction: Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. Co-author DOI 10.1038/s41586-025-08979-z↗
- Identification and characterization of tumor and stromal derived liquid biopsy analytes in pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s13046-024-03262-x↗
- Potential association between PSCA rs2976395 functional variant and pancreatic cancer risk. Co-author DOI 10.1002/ijc.35046↗
- REDISCOVER International Guidelines on the Perioperative Care of Surgical Patients With Borderline-resectable and Locally Advanced Pancreatic Cancer. Co-author DOI 10.1097/sla.0000000000006248↗
- Polymorphisms in transcription factor binding sites and enhancer regions and pancreatic ductal adenocarcinoma risk. Co-author DOI 10.1186/s40246-024-00576-x↗
- Association between a polymorphic variant in the CDKN2B-AS1/ANRIL gene and pancreatic cancer risk. Co-author DOI 10.1002/ijc.34383↗
- The PANcreatic Disease ReseArch (PANDoRA) consortium: Ten years' experience of association studies to understand the genetic architecture of pancreatic cancer. Co-author DOI 10.1016/j.critrevonc.2023.104020↗
- Genetic and non-genetic risk factors for early-onset pancreatic cancer. Co-author DOI 10.1016/j.dld.2023.02.023↗
- Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. Co-author DOI 10.1038/s41586-023-05728-y↗
- A scan of all coding region variants of the human genome, identifies 13q12.2-rs9579139 and 15q24.1-rs2277598 as novel risk loci for pancreatic ductal adenocarc… Co-author DOI 10.1093/carcin/bgad056↗
- Peripheral and Portal Venous KRAS ctDNA Detection as Independent Prognostic Markers of Early Tumor Recurrence in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1093/clinchem/hvac214↗
- Polymorphic variants involved in methylation regulation: a strategy to discover risk loci for pancreatic ductal adenocarcinoma. Co-author DOI 10.1136/jmg-2022-108910↗
- Exploring the Neandertal legacy of pancreatic ductal adenocarcinoma risk in Eurasians. Co-author DOI 10.1186/s40659-023-00457-y↗
- Common variability in oestrogen-related genes and pancreatic ductal adenocarcinoma risk in women. Co-author DOI 10.1038/s41598-022-22973-9↗
- Mucin 5AC expression is common but unrelated to tumor progression in pancreatic adenocarcinoma. Co-author DOI 10.1177/03946320221106504↗
- Circulating Cancer Associated Macrophage-like Cells as a Potential New Prognostic Marker in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3390/biomedicines10112955↗
- Characterization of RARRES1 Expression on Circulating Tumor Cells as Unfavorable Prognostic Marker in Resected Pancreatic Ductal Adenocarcinoma Patients. Co-author DOI 10.3390/cancers14184405↗
- Genome-wide scan of long noncoding RNA single nucleotide polymorphisms and pancreatic cancer susceptibility. Co-author DOI 10.1002/ijc.33475↗
- Mesothelin is Commonly Expressed in Pancreatic Adenocarcinoma but Unrelated to Cancer Aggressiveness. Co-author DOI 10.1080/07357907.2021.1943747↗
- Associations between pancreatic expression quantitative traits and risk of pancreatic ductal adenocarcinoma. Co-author DOI 10.1093/carcin/bgab057↗
- Truncated O-GalNAc glycans impact on fundamental signaling pathways in pancreatic cancer. Co-author DOI 10.1093/glycob/cwab088↗
- Polygenic and multifactorial scores for pancreatic ductal adenocarcinoma risk prediction. Co-author DOI 10.1136/jmedgenet-2020-106961↗
- Genetic Polymorphisms Involved in Mitochondrial Metabolism and Pancreatic Cancer Risk. Co-author DOI 10.1158/1055-9965.epi-21-0353↗
- Integrin alpha-V is an important driver in pancreatic adenocarcinoma progression. Co-author DOI 10.1186/s13046-021-01946-2↗
- Association of Genetic Variants Affecting microRNAs and Pancreatic Cancer Risk. Co-author DOI 10.3389/fgene.2021.693933↗
- Identification of Recessively Inherited Genetic Variants Potentially Linked to Pancreatic Cancer Risk. Co-author DOI 10.3389/fonc.2021.771312↗
- Evaluation of the Hamburg-Glasgow Classification in Pancreatic Cancer: Preoperative Staging by Combining Disseminated Tumor Load and Systemic Inflammation. Co-author DOI 10.3390/cancers13235942↗
- DOG1 is commonly expressed in pancreatic adenocarcinoma but unrelated to cancer aggressiveness. Co-author DOI 10.7717/peerj.11905↗
- Biperiden and mepazine effectively inhibit MALT1 activity and tumor growth in pancreatic cancer. Co-author DOI 10.1002/ijc.32567↗
- Genome-wide association study identifies an early onset pancreatic cancer risk locus. Co-author DOI 10.1002/ijc.33004↗
- Patterns of Recurrence After Resection of Pancreatic Ductal Adenocarcinoma: A Secondary Analysis of the ESPAC-4 Randomized Adjuvant Chemotherapy Trial. Co-author DOI 10.1001/jamasurg.2019.3337↗
- Oligometastases in pancreatic cancer (Synchronous resections of hepatic oligometastatic pancreatic cancer: Disputing a principle in a time of safe pancreatic o… First author DOI 10.1002/ags3.12255↗
- Genetic determinants of telomere length and risk of pancreatic cancer: A PANDoRA study. Co-author DOI 10.1002/ijc.31928↗
- Germline BRCA2 K3326X and CHEK2 I157T mutations increase risk for sporadic pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.32127↗
- Evaluation of the MDACC clinical classification system for pancreatic cancer patients in an European multicenter cohort. Co-author DOI 10.1016/j.ejso.2018.12.012↗
- The Impact of Positive Resection Margins on Survival and Recurrence Following Resection and Adjuvant Chemotherapy for Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1097/sla.0000000000002557↗
- Do pancreatic cancer and chronic pancreatitis share the same genetic risk factors? A PANcreatic Disease ReseArch (PANDoRA) consortium investigation. Co-author DOI 10.1002/ijc.31047↗
- Kidney Transplantation after Extended Multivisceral Resection for Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1155/2018/3757382↗
- HSP90 is a promising target in gemcitabine and 5-fluorouracil resistant pancreatic cancer. Co-author DOI 10.1007/s10495-016-1332-4↗
- Comparison of adjuvant gemcitabine and capecitabine with gemcitabine monotherapy in patients with resected pancreatic cancer (ESPAC-4): a multicentre, open-lab… Co-author DOI 10.1016/s0140-6736(16)32409-6↗
- Pancreatic cancer and liver metastases: state of the art. Co-author DOI 10.1007/s13304-016-0407-7↗
- Synchronous resections of hepatic oligometastatic pancreatic cancer: Disputing a principle in a time of safe pancreatic operations in a retrospective multicent… Co-author DOI 10.1016/j.surg.2016.02.019↗
- Relevance of Sp Binding Site Polymorphism in WWOX for Treatment Outcome in Pancreatic Cancer. Co-author DOI 10.1093/jnci/djv387↗
- Functional single nucleotide polymorphisms within the cyclin-dependent kinase inhibitor 2A/2B region affect pancreatic cancer risk. Co-author DOI 10.18632/oncotarget.10935↗
- Cyclin D1 is a strong prognostic factor for survival in pancreatic cancer: analysis of CD G870A polymorphism, FISH and immunohistochemistry. Co-author DOI 10.1002/jso.23826↗
- Resection margin clearance in pancreatic cancer after implementation of the Leeds Pathology Protocol (LEEPP): clinically relevant or just academic? Co-author DOI 10.1007/s00268-014-2808-4↗
- COSMC knockdown mediated aberrant O-glycosylation promotes oncogenic properties in pancreatic cancer. Co-author DOI 10.1186/s12943-015-0386-1↗
Trials 1
- Neoadjuvant Treatment in Resectable Pancreatic Cancer Principal investigator NCT01900327↗
Closest collaborators
The 25 strongest of 160 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Faik Güntac Uzunoglu · 13 shared papers, 2014–2025 · strength 1.57
- Paper Patient-derived tumor organoids highlight the potential of precision medicine in managing pancreatic ductal adenocarcin… DOI 10.1002/ijc.35443↗
- Paper Identification and characterization of tumor and stromal derived liquid biopsy analytes in pancreatic ductal adenocarci… DOI 10.1186/s13046-024-03262-x↗
- Paper Author Correction: Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-025-08979-z↗
- Marianne Sinn · 7 shared papers, 2022–2025 · strength 1.10
- Paper Patient-derived tumor organoids highlight the potential of precision medicine in managing pancreatic ductal adenocarcin… DOI 10.1002/ijc.35443↗
- Paper Identification and characterization of tumor and stromal derived liquid biopsy analytes in pancreatic ductal adenocarci… DOI 10.1186/s13046-024-03262-x↗
- Paper Author Correction: Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-025-08979-z↗
- Maximilian Bockhorn · 24 shared papers, 2011–2021 · strength 1.09
- Paper Truncated O-GalNAc glycans impact on fundamental signaling pathways in pancreatic cancer. DOI 10.1093/glycob/cwab088↗
- Paper Biperiden and mepazine effectively inhibit MALT1 activity and tumor growth in pancreatic cancer. DOI 10.1002/ijc.32567↗
- Paper Evaluation of the MDACC clinical classification system for pancreatic cancer patients in an European multicenter cohort. DOI 10.1016/j.ejso.2018.12.012↗
- Cenap Güngör · 11 shared papers, 2011–2025 · strength 0.88
- Paper Author Correction: Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-025-08979-z↗
- Paper Characterization of RARRES1 Expression on Circulating Tumor Cells as Unfavorable Prognostic Marker in Resected Pancreat… DOI 10.3390/cancers14184405↗
- Paper Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-023-05728-y↗
- Harriet Wikman · 5 shared papers, 2022–2025 · strength 0.81
- Paper Patient-derived tumor organoids highlight the potential of precision medicine in managing pancreatic ductal adenocarcin… DOI 10.1002/ijc.35443↗
- Paper Identification and characterization of tumor and stromal derived liquid biopsy analytes in pancreatic ductal adenocarci… DOI 10.1186/s13046-024-03262-x↗
- Paper Peripheral and Portal Venous KRAS ctDNA Detection as Independent Prognostic Markers of Early Tumor Recurrence in Pancre… DOI 10.1093/clinchem/hvac214↗
- Christine Nitschke · 5 shared papers, 2022–2025 · strength 0.81
- Paper Patient-derived tumor organoids highlight the potential of precision medicine in managing pancreatic ductal adenocarcin… DOI 10.1002/ijc.35443↗
- Paper Identification and characterization of tumor and stromal derived liquid biopsy analytes in pancreatic ductal adenocarci… DOI 10.1186/s13046-024-03262-x↗
- Paper Peripheral and Portal Venous KRAS ctDNA Detection as Independent Prognostic Markers of Early Tumor Recurrence in Pancre… DOI 10.1093/clinchem/hvac214↗
- Thilo Hackert · 6 shared papers, 2016–2025 · strength 0.66
- Paper Patient-derived tumor organoids highlight the potential of precision medicine in managing pancreatic ductal adenocarcin… DOI 10.1002/ijc.35443↗
- Paper Identification and characterization of tumor and stromal derived liquid biopsy analytes in pancreatic ductal adenocarci… DOI 10.1186/s13046-024-03262-x↗
- Paper Association between a polymorphic variant in the CDKN2B-AS1/ANRIL gene and pancreatic cancer risk. DOI 10.1002/ijc.34383↗
- Florian Gebauer · 14 shared papers, 2011–2021 · strength 0.63
- Paper Integrin alpha-V is an important driver in pancreatic adenocarcinoma progression. DOI 10.1186/s13046-021-01946-2↗
- Paper Pancreatic cancer and liver metastases: state of the art. DOI 10.1007/s13304-016-0407-7↗
- Paper Resection margin clearance in pancreatic cancer after implementation of the Leeds Pathology Protocol (LEEPP): clinicall… DOI 10.1007/s00268-014-2808-4↗
- Michael Tachezy · 13 shared papers, 2011–2021 · strength 0.60
- Paper Integrin alpha-V is an important driver in pancreatic adenocarcinoma progression. DOI 10.1186/s13046-021-01946-2↗
- Paper Pancreatic cancer and liver metastases: state of the art. DOI 10.1007/s13304-016-0407-7↗
- Paper Resection margin clearance in pancreatic cancer after implementation of the Leeds Pathology Protocol (LEEPP): clinicall… DOI 10.1007/s00268-014-2808-4↗
- Yogesh Kumar Vashist · 14 shared papers, 2011–2018 · strength 0.57
- Paper Kidney Transplantation after Extended Multivisceral Resection for Pancreatic Ductal Adenocarcinoma. DOI 10.1155/2018/3757382↗
- Paper HSP90 is a promising target in gemcitabine and 5-fluorouracil resistant pancreatic cancer. DOI 10.1007/s10495-016-1332-4↗
- Paper Resection margin clearance in pancreatic cancer after implementation of the Leeds Pathology Protocol (LEEPP): clinicall… DOI 10.1007/s00268-014-2808-4↗
- Leonie Konczalla · 4 shared papers, 2020–2025 · strength 0.51
- Paper Author Correction: Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-025-08979-z↗
- Paper Circulating Cancer Associated Macrophage-like Cells as a Potential New Prognostic Marker in Pancreatic Ductal Adenocarc… DOI 10.3390/biomedicines10112955↗
- Paper Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-023-05728-y↗
- John P. Neoptolemos · 8 shared papers, 2014–2023 · strength 0.46
- Paper Association between a polymorphic variant in the CDKN2B-AS1/ANRIL gene and pancreatic cancer risk. DOI 10.1002/ijc.34383↗
- Paper Common variability in oestrogen-related genes and pancreatic ductal adenocarcinoma risk in women. DOI 10.1038/s41598-022-22973-9↗
- Paper The Impact of Positive Resection Margins on Survival and Recurrence Following Resection and Adjuvant Chemotherapy for P… DOI 10.1097/sla.0000000000002557↗
- Joseph Tintelnot · 3 shared papers, 2023–2025 · strength 0.43
- Paper Author Correction: Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-025-08979-z↗
- Paper Peripheral and Portal Venous KRAS ctDNA Detection as Independent Prognostic Markers of Early Tumor Recurrence in Pancre… DOI 10.1093/clinchem/hvac214↗
- Paper Microbiota-derived 3-IAA influences chemotherapy efficacy in pancreatic cancer. DOI 10.1038/s41586-023-05728-y↗
- Michael Fabian Nentwich · 5 shared papers, 2012–2019 · strength 0.35
- Paper Oligometastases in pancreatic cancer (Synchronous resections of hepatic oligometastatic pancreatic cancer: Disputing a … DOI 10.1002/ags3.12255↗
- Paper Limits of surgery for pancreatic cancer. PMID 25301342↗
- Paper Cyclin D1 is a strong prognostic factor for survival in pancreatic cancer: analysis of CD G870A polymorphism, FISH and … DOI 10.1002/jso.23826↗
- Matthias Reeh · 5 shared papers, 2011–2021 · strength 0.34
- Paper Evaluation of the Hamburg-Glasgow Classification in Pancreatic Cancer: Preoperative Staging by Combining Disseminated T… DOI 10.3390/cancers13235942↗
- Paper Evaluation of the MDACC clinical classification system for pancreatic cancer patients in an European multicenter cohort. DOI 10.1016/j.ejso.2018.12.012↗
- Paper HSP90 is a promising target in gemcitabine and 5-fluorouracil resistant pancreatic cancer. DOI 10.1007/s10495-016-1332-4↗
- Gerrit Wolters‐Eisfeld · 5 shared papers, 2014–2021 · strength 0.31
- Paper Truncated O-GalNAc glycans impact on fundamental signaling pathways in pancreatic cancer. DOI 10.1093/glycob/cwab088↗
- Paper Biperiden and mepazine effectively inhibit MALT1 activity and tumor growth in pancreatic cancer. DOI 10.1002/ijc.32567↗
- Paper The multifunctional growth factor midkine promotes proliferation and migration in pancreatic cancer. DOI 10.1158/1541-7786.mcr-13-0467↗
- Till Krech · 3 shared papers, 2021–2022 · strength 0.31
- Paper Mesothelin is Commonly Expressed in Pancreatic Adenocarcinoma but Unrelated to Cancer Aggressiveness. DOI 10.1080/07357907.2021.1943747↗
- Paper Mucin 5AC expression is common but unrelated to tumor progression in pancreatic adenocarcinoma. DOI 10.1177/03946320221106504↗
- Paper DOG1 is commonly expressed in pancreatic adenocarcinoma but unrelated to cancer aggressiveness. DOI 10.7717/peerj.11905↗
- Till Sebastian Clauditz · 3 shared papers, 2021–2022 · strength 0.31
- Paper Mesothelin is Commonly Expressed in Pancreatic Adenocarcinoma but Unrelated to Cancer Aggressiveness. DOI 10.1080/07357907.2021.1943747↗
- Paper Mucin 5AC expression is common but unrelated to tumor progression in pancreatic adenocarcinoma. DOI 10.1177/03946320221106504↗
- Paper DOG1 is commonly expressed in pancreatic adenocarcinoma but unrelated to cancer aggressiveness. DOI 10.7717/peerj.11905↗
- Udo Schumacher · 5 shared papers, 2013–2021 · strength 0.31
- Paper Integrin alpha-V is an important driver in pancreatic adenocarcinoma progression. DOI 10.1186/s13046-021-01946-2↗
- Paper Biperiden and mepazine effectively inhibit MALT1 activity and tumor growth in pancreatic cancer. DOI 10.1002/ijc.32567↗
- Paper Serum EpCAM expression in pancreatic cancer. PMID 25202052↗
- Danmei Zhang · 2 shared papers, 2023–2025 · strength 0.29
- Nicola Gagliani · 2 shared papers, 2023–2025 · strength 0.29
- Marija Trajkovic‐Arsic · 2 shared papers, 2023–2025 · strength 0.29
- Jens Thomas Siveke · 2 shared papers, 2023–2025 · strength 0.29
- Alec C. Kimmelman · 2 shared papers, 2023–2025 · strength 0.29
- Stefan Hubert Boeck · 2 shared papers, 2023–2025 · strength 0.29
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.