ResearcherNebraska Medical CenterActive in the last 2 years
Rakesh Bhatia
Matched on ORCID iD 0000-0001-6428-1022; OpenAlex author ID A5039090834, A5124913936. Sources: OpenAlex.
Their work
- Papers
- 14 (first author 3, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 42
2017: 1 paper · 2018: 1 paper · 2019: none · 2020: 1 paper · 2021: 5 papers · 2022: 3 papers · 2023: 1 paper · 2024: none · 2025: 1 paper · 2026: 1 paper
Papers 14
- Acinar cell plasticity and subtype specification during acinar-to-ductal metaplasia and pancreatic cancer progression. Last author DOI 10.1016/j.bbcan.2026.189549↗
- Ribosome biogenesis, altered metabolism and ribotoxic stress response in pancreatic ductal adenocarcinoma tumor microenvironment. Last author DOI 10.1016/j.canlet.2025.217484↗
- Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. Co-author DOI 10.1038/s41388-022-02587-1↗
- Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.canlet.2022.215922↗
- Cytokines chattering in pancreatic ductal adenocarcinoma tumor microenvironment. Co-author DOI 10.1016/j.semcancer.2022.03.021↗
- Mucin 5AC Serves as the Nexus for β-Catenin/c-Myc Interplay to Promote Glutamine Dependency During Pancreatic Cancer Chemoresistance. First author DOI 10.1053/j.gastro.2021.09.017↗
- Molecular mechanisms of pancreatic myofibroblast activation in chronic pancreatitis and pancreatic ductal adenocarcinoma. Co-author DOI 10.1007/s00535-021-01800-4↗
- SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1128/mcb.00135-21↗
- Mucin Expression and Splicing Determine Novel Subtypes and Patient Mortality in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-21-1591↗
- Repurposing Niclosamide for Targeting Pancreatic Cancer by Inhibiting Hh/Gli Non-Canonical Axis of Gsk3β. First author DOI 10.3390/cancers13133105↗
- Correction: Kaushal et al. Repurposing Niclosamide for Targeting Pancreatic Cancer by Inhibiting Hh/Gli Non-Canonical Axis of Gsk3β. Cancers 2021, 13, 3105. First author DOI 10.3390/cancers13225591↗
- CXCR3 and Cognate Ligands are Associated with Immune Cell Alteration and Aggressiveness of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1078-0432.ccr-20-1359↗
- Emerging trends in the immunotherapy of pancreatic cancer. Co-author DOI 10.1016/j.canlet.2017.12.012↗
- MUC4 mucin- a therapeutic target for pancreatic ductal adenocarcinoma. Co-author DOI 10.1080/14728222.2017.1323880↗
Closest collaborators
The 25 strongest of 42 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Surinder K. Batra · 12 shared papers, 2017–2023 · strength 1.50
- Paper Cytokines chattering in pancreatic ductal adenocarcinoma tumor microenvironment. DOI 10.1016/j.semcancer.2022.03.021↗
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Molecular mechanisms of pancreatic myofibroblast activation in chronic pancreatitis and pancreatic ductal adenocarcinom… DOI 10.1007/s00535-021-01800-4↗
- Sushil Kumar K · 8 shared papers, 2017–2023 · strength 0.99
- Paper Cytokines chattering in pancreatic ductal adenocarcinoma tumor microenvironment. DOI 10.1016/j.semcancer.2022.03.021↗
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Molecular mechanisms of pancreatic myofibroblast activation in chronic pancreatitis and pancreatic ductal adenocarcinom… DOI 10.1007/s00535-021-01800-4↗
- Sanjib Chaudhary · 2 shared papers, 2025–2026 · strength 0.70
- Paper Ribosome biogenesis, altered metabolism and ribotoxic stress response in pancreatic ductal adenocarcinoma tumor microen… DOI 10.1016/j.canlet.2025.217484↗
- Paper Acinar cell plasticity and subtype specification during acinar-to-ductal metaplasia and pancreatic cancer progression. DOI 10.1016/j.bbcan.2026.189549↗
- Ramesh Pothuraju · 2 shared papers, 2025–2026 · strength 0.70
- Paper Ribosome biogenesis, altered metabolism and ribotoxic stress response in pancreatic ductal adenocarcinoma tumor microen… DOI 10.1016/j.canlet.2025.217484↗
- Paper Acinar cell plasticity and subtype specification during acinar-to-ductal metaplasia and pancreatic cancer progression. DOI 10.1016/j.bbcan.2026.189549↗
- Christopher M. Thompson · 5 shared papers, 2020–2023 · strength 0.66
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Molecular mechanisms of pancreatic myofibroblast activation in chronic pancreatitis and pancreatic ductal adenocarcinom… DOI 10.1007/s00535-021-01800-4↗
- Paper Mucin 5AC Serves as the Nexus for β-Catenin/c-Myc Interplay to Promote Glutamine Dependency During Pancreatic Cancer Ch… DOI 10.1053/j.gastro.2021.09.017↗
- Andrew C. Cannon · 5 shared papers, 2017–2023 · strength 0.60
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Molecular mechanisms of pancreatic myofibroblast activation in chronic pancreatitis and pancreatic ductal adenocarcinom… DOI 10.1007/s00535-021-01800-4↗
- Paper Mucin Expression and Splicing Determine Novel Subtypes and Patient Mortality in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-21-1591↗
- Pranita Atri · 4 shared papers, 2020–2022 · strength 0.48
- Paper Mucin 5AC Serves as the Nexus for β-Catenin/c-Myc Interplay to Promote Glutamine Dependency During Pancreatic Cancer Ch… DOI 10.1053/j.gastro.2021.09.017↗
- Paper Mucin Expression and Splicing Determine Novel Subtypes and Patient Mortality in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-21-1591↗
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Shailendra Kumar Gautam · 3 shared papers, 2017–2026 · strength 0.47
- Paper Acinar cell plasticity and subtype specification during acinar-to-ductal metaplasia and pancreatic cancer progression. DOI 10.1016/j.bbcan.2026.189549↗
- Paper Emerging trends in the immunotherapy of pancreatic cancer. DOI 10.1016/j.canlet.2017.12.012↗
- Paper MUC4 mucin- a therapeutic target for pancreatic ductal adenocarcinoma. DOI 10.1080/14728222.2017.1323880↗
- Satyanarayana Rachagani · 3 shared papers, 2021–2023 · strength 0.43
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Repurposing Niclosamide for Targeting Pancreatic Cancer by Inhibiting Hh/Gli Non-Canonical Axis of Gsk3β. DOI 10.3390/cancers13133105↗
- Paper Correction: Kaushal et al. Repurposing Niclosamide for Targeting Pancreatic Cancer by Inhibiting Hh/Gli Non-Canonical A… DOI 10.3390/cancers13225591↗
- Jesse L. Cox · 3 shared papers, 2022–2023 · strength 0.40
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Mucin 5AC Serves as the Nexus for β-Catenin/c-Myc Interplay to Promote Glutamine Dependency During Pancreatic Cancer Ch… DOI 10.1053/j.gastro.2021.09.017↗
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Koelina Ganguly · 3 shared papers, 2021–2023 · strength 0.39
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Mucin 5AC Serves as the Nexus for β-Catenin/c-Myc Interplay to Promote Glutamine Dependency During Pancreatic Cancer Ch… DOI 10.1053/j.gastro.2021.09.017↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Sanchita Rauth · 3 shared papers, 2021–2022 · strength 0.36
- Paper Mucin 5AC Serves as the Nexus for β-Catenin/c-Myc Interplay to Promote Glutamine Dependency During Pancreatic Cancer Ch… DOI 10.1053/j.gastro.2021.09.017↗
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Xiaoqi Li · 2 shared papers, 2022–2023 · strength 0.34
- Bharti Gautam · 1 shared paper, 2026 · strength 0.33
- Maneesh Jain · 3 shared papers, 2017–2023 · strength 0.29
- Paper Muc4 loss mitigates epidermal growth factor receptor activity essential for PDAC tumorigenesis. DOI 10.1038/s41388-022-02587-1↗
- Paper Emerging trends in the immunotherapy of pancreatic cancer. DOI 10.1016/j.canlet.2017.12.012↗
- Paper MUC4 mucin- a therapeutic target for pancreatic ductal adenocarcinoma. DOI 10.1080/14728222.2017.1323880↗
- Jyoti Kaushal · 2 shared papers, 2021 · strength 0.28
- Lynette M. Smith · 2 shared papers, 2021–2022 · strength 0.24
- Paper Mucin Expression and Splicing Determine Novel Subtypes and Patient Mortality in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-21-1591↗
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Sushil Kumar · 2 shared papers, 2021 · strength 0.24
- Paper Mucin Expression and Splicing Determine Novel Subtypes and Patient Mortality in Pancreatic Ductal Adenocarcinoma. DOI 10.1158/1078-0432.ccr-21-1591↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Parthasarathy Seshacharyulu · 2 shared papers, 2021–2022 · strength 0.23
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Ashu Shah · 2 shared papers, 2021–2022 · strength 0.23
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Rama Krishna Nimmakayala · 2 shared papers, 2021–2022 · strength 0.23
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Moorthy Palanimuthu Ponnusamy · 2 shared papers, 2021–2022 · strength 0.23
- Paper Small molecule inhibitor against onco-mucins disrupts Src/FosL1 axis to enhance gemcitabine efficacy in pancreatic duct… DOI 10.1016/j.canlet.2022.215922↗
- Paper SUMO Modification of PAF1/PD2 Enables PML Interaction and Promotes Radiation Resistance in Pancreatic Ductal Adenocarci… DOI 10.1128/mcb.00135-21↗
- Joyce C. Solheim · 2 shared papers, 2018–2021 · strength 0.22
- Namita Bhyravbhatla · 1 shared paper, 2022 · strength 0.19
- Naveenkumar Perumal · 1 shared paper, 2023 · strength 0.15
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.