ResearcherBaylor College of MedicineNo recent work listed

Sarah H. ELSEA

ORCID-verified (high confidence, 0.95), identity_conf@1As of 7 November 2026

Matched on ORCID iD 0000-0002-1400-8519; OpenAlex author ID A5013465101; NIH RePORTER profile ID 1896431. Sources: OpenAlex.

Their work

Papers: 1
Trials: 0
Grants: 1
Labs led: 1
Collaborators: 4

Papers, trials and grants by year

2020: 1 paper · 2021: 1 grant

Papers 1

Differential expression of MAGEA6 toggles autophagy to promote pancreatic cancer progression. Co-author DOI 10.7554/elife.48963 ↗

Grants 1

Preclinical Analyses of NAD Kinase as a Redox Vulnerability for the Treatment of Pancreatic Cancer R01CA211176 ↗

Closest collaborators

The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Gordon Brent Mills · 1 shared paper, 2020 · strength 0.10
- Paper Differential expression of MAGEA6 toggles autophagy to promote pancreatic cancer progression. DOI 10.7554/elife.48963 ↗
Kenneth L. Scott · 1 shared paper, 2020 · strength 0.10
- Paper Differential expression of MAGEA6 toggles autophagy to promote pancreatic cancer progression. DOI 10.7554/elife.48963 ↗
Yiu Huen Tsang · 1 shared paper, 2020 · strength 0.10
- Paper Differential expression of MAGEA6 toggles autophagy to promote pancreatic cancer progression. DOI 10.7554/elife.48963 ↗
Yumeng Wang · 1 shared paper, 2020 · strength 0.10
- Paper Differential expression of MAGEA6 toggles autophagy to promote pancreatic cancer progression. DOI 10.7554/elife.48963 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.