ResearcherShanghai Medical College of Fudan UniversityActive in the last 2 years

Weiding Wu

Verified by OpenAlex author ID (good confidence, 0.85), identity_conf@1As of 8 November 2026

Matched on OpenAlex author ID A5107936006. Sources: OpenAlex.

Their work

Papers: 3 (first author 0, last author 2)
Trials: 0
Grants: 0
Labs led: 1
Collaborators: 13

Papers, trials and grants by year

2023: 1 paper · 2024: none · 2025: 1 paper · 2026: 1 paper

Papers 3

Overcoming multidimensional immunotherapy resistance in PDAC: from microenvironment to clinic. Last author DOI 10.3389/fimmu.2026.1782960 ↗
Neurogenic inducers inhibit the proliferation of pancreatic cancer by promoting tumor cell transdifferentiation. Last author DOI 10.1186/s13046-025-03563-9 ↗
Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreatic cancer. Co-author DOI 10.1007/s12672-023-00628-z ↗

Closest collaborators

The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Kaizhou Jin · 2 shared papers, 2025–2026 · strength 0.59
- Paper Overcoming multidimensional immunotherapy resistance in PDAC: from microenvironment to clinic. DOI 10.3389/fimmu.2026.1782960 ↗
- Paper Neurogenic inducers inhibit the proliferation of pancreatic cancer by promoting tumor cell transdifferentiation. DOI 10.1186/s13046-025-03563-9 ↗
Jin Yan · 1 shared paper, 2026 · strength 0.36
- Paper Overcoming multidimensional immunotherapy resistance in PDAC: from microenvironment to clinic. DOI 10.3389/fimmu.2026.1782960 ↗
Saimeng Shi · 1 shared paper, 2025 · strength 0.23
- Paper Neurogenic inducers inhibit the proliferation of pancreatic cancer by promoting tumor cell transdifferentiation. DOI 10.1186/s13046-025-03563-9 ↗
Qinglin Fei · 1 shared paper, 2025 · strength 0.23
- Paper Neurogenic inducers inhibit the proliferation of pancreatic cancer by promoting tumor cell transdifferentiation. DOI 10.1186/s13046-025-03563-9 ↗
Duancheng Guo · 1 shared paper, 2025 · strength 0.23
- Paper Neurogenic inducers inhibit the proliferation of pancreatic cancer by promoting tumor cell transdifferentiation. DOI 10.1186/s13046-025-03563-9 ↗
Jianhui Yang · 1 shared paper, 2025 · strength 0.23
- Paper Neurogenic inducers inhibit the proliferation of pancreatic cancer by promoting tumor cell transdifferentiation. DOI 10.1186/s13046-025-03563-9 ↗
Long‐Yun Ye · 1 shared paper, 2025 · strength 0.23
- Paper Neurogenic inducers inhibit the proliferation of pancreatic cancer by promoting tumor cell transdifferentiation. DOI 10.1186/s13046-025-03563-9 ↗
Quanxing Ni · 1 shared paper, 2023 · strength 0.17
- Paper Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreati… DOI 10.1007/s12672-023-00628-z ↗
Zhiwen Xiao · 1 shared paper, 2023 · strength 0.17
- Paper Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreati… DOI 10.1007/s12672-023-00628-z ↗
Cheng He · 1 shared paper, 2023 · strength 0.17
- Paper Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreati… DOI 10.1007/s12672-023-00628-z ↗
Xianjun Yu · 1 shared paper, 2023 · strength 0.17
- Paper Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreati… DOI 10.1007/s12672-023-00628-z ↗
Chen Liu · 1 shared paper, 2023 · strength 0.17
- Paper Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreati… DOI 10.1007/s12672-023-00628-z ↗
Guopei Luo · 1 shared paper, 2023 · strength 0.17
- Paper Glutamine is a substrate for glycosylation and CA19-9 biosynthesis through hexosamine biosynthetic pathway in pancreati… DOI 10.1007/s12672-023-00628-z ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.