Pancreatic Cancer Research

ResearcherFirst Affiliated Hospital of Wenzhou Medical UniversityActive in the last 2 years

Linhua Lan

Matched on ORCID iD 0000-0002-6482-1508; OpenAlex author ID A5079654043. Sources: OpenAlex.

Their work

Papers
11 (first author 0, last author 8)
Trials
0
Grants
0
Labs led
1
Collaborators
20
Papers, trials and grants by year

2021: 4 papers · 2022: 4 papers · 2023: 1 paper · 2024: 1 paper · 2025: none · 2026: 1 paper

Papers 11

  1. BCAT1 inhibits glutamine-dependent Akt/mTOR signaling and nucleotide synthesis by initiating BTRC-mediated degradation of SLC3A2 in pancreatic adenocarcinoma. Last author DOI 10.1186/s40170-026-00436-3↗
  2. Lactate enhances NMNAT1 lactylation to sustain nuclear NAD+ salvage pathway and promote survival of pancreatic adenocarcinoma cells under glucose-deprived cond… Last author DOI 10.1016/j.canlet.2024.216806↗
  3. Construction of a metabolism-related gene prognostic model to predict survival of pancreatic cancer patients. Co-author DOI 10.1016/j.heliyon.2022.e12378↗
  4. Synthetic oleanane triterpenoid derivative CDDO-Me disrupts cellular bioenergetics to suppress pancreatic ductal adenocarcinoma via targeting SLC1A5. Co-author DOI 10.1002/jbt.23192↗
  5. 6-methoxydihydroavicine, the alkaloid extracted from Macleaya cordata (Willd.) R. Br. (Papaveraceae), triggers RIPK1/Caspase-dependent cell death in pancreatic… Last author DOI 10.1016/j.phymed.2022.154164↗
  6. A synthesized olean-28,13β-lactam targets YTHDF1-GLS1 axis to induce ROS-dependent metabolic crisis and cell death in pancreatic adenocarcinoma. Last author DOI 10.1186/s12935-022-02562-6↗
  7. Single-Cell Transcriptome Reveals the Metabolic and Clinical Features of a Highly Malignant Cell Subpopulation in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3389/fcell.2022.798165↗
  8. S100A16 promotes metastasis and progression of pancreatic cancer through FGF19-mediated AKT and ERK1/2 pathways. Last author DOI 10.1007/s10565-020-09574-w↗
  9. Centromere protein F is identified as a novel therapeutic target by genomics profile and contributing to the progression of pancreatic cancer. Last author DOI 10.1016/j.ygeno.2020.10.039↗
  10. RUNX2 and LAMC2: promising pancreatic cancer biomarkers identified by an integrative data mining of pancreatic adenocarcinoma tissues. Last author DOI 10.18632/aging.203589↗
  11. CXCL10 is a Tumor Microenvironment and Immune Infiltration Related Prognostic Biomarker in Pancreatic Adenocarcinoma. Last author DOI 10.3389/fmolb.2021.611508↗

Closest collaborators

The 20 strongest of 20 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Huimin Huang · 7 shared papers, 2021–2026 · strength 1.30
  2. Guihua Jin · 6 shared papers, 2021–2026 · strength 1.13
  3. Fugen Shangguan · 3 shared papers, 2021–2022 · strength 0.48
  4. Shijia Wu · 3 shared papers, 2022 · strength 0.43
  5. Hongping Xia · 3 shared papers, 2021–2022 · strength 0.39
  6. Xiaoqun Zheng · 2 shared papers, 2022–2024 · strength 0.36
  7. Percy David Papa Akuetteh · 2 shared papers, 2022 · strength 0.29
  8. Xiao Mo · 2 shared papers, 2021 · strength 0.25
  9. Liyi Li · 1 shared paper, 2023 · strength 0.20
  10. Qingqing Ruan · 1 shared paper, 2021 · strength 0.19
  11. Hongping Xia · 1 shared paper, 2024 · strength 0.19
  12. Yangyang Fang · 1 shared paper, 2022 · strength 0.17
  13. Hongbao Yang · 1 shared paper, 2022 · strength 0.15
  14. Qiyu Zhang · 1 shared paper, 2022 · strength 0.15
  15. Nengfang Ma · 1 shared paper, 2022 · strength 0.14
  16. Hailong Yang · 1 shared paper, 2022 · strength 0.14
  17. Hongjin Chen · 1 shared paper, 2021 · strength 0.13
  18. Xiaoming Wang · 1 shared paper, 2021 · strength 0.13
  19. Alaa Abdelatty · 1 shared paper, 2021 · strength 0.12
  20. Dan Fang · 1 shared paper, 2021 · strength 0.12

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.