ResearcherShanghai Medical College of Fudan UniversityActive in the last 2 years
Ying-Yi Li
Matched on ORCID iD 0000-0002-6269-2992; OpenAlex author ID A5101536986. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 13
2013: 1 paper · 2014: none · 2015: 1 paper · 2016: 1 paper · 2017: none · 2018: 1 paper · 2019: none · 2020: none · 2021: none · 2022: none · 2023: none · 2024: none · 2025: 1 paper
Papers 5
- Correction to: Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Src/MAPK/ERK pathways. Co-author DOI 10.1038/s41419-025-07495-2↗
- Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Src/MAPK/ERK pathways. Co-author DOI 10.1038/s41419-018-0574-1↗
- Bufalin suppresses cancer stem-like cells in gemcitabine-resistant pancreatic cancer cells via Hedgehog signaling. Co-author DOI 10.3892/mmr.2016.5471↗
- miR-33a suppresses the nuclear translocation of β-catenin to enhance gemcitabine sensitivity in human pancreatic cancer cells. Co-author DOI 10.1007/s13277-015-3679-5↗
- Pim-3 promotes the growth of human pancreatic cancer in the orthotopic nude mouse model through vascular endothelium growth factor. Last author DOI 10.1016/j.jss.2013.06.004↗
Closest collaborators
The 13 strongest of 13 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Chen Liang · 3 shared papers, 2015–2025 · strength 0.38
- Paper Correction to: Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Sr… DOI 10.1038/s41419-025-07495-2↗
- Paper Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Src/MAPK/ERK path… DOI 10.1038/s41419-018-0574-1↗
- Paper miR-33a suppresses the nuclear translocation of β-catenin to enhance gemcitabine sensitivity in human pancreatic cancer… DOI 10.1007/s13277-015-3679-5↗
- Hong‐Xi Xu · 2 shared papers, 2018–2025 · strength 0.32
- Paper Correction to: Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Sr… DOI 10.1038/s41419-025-07495-2↗
- Paper Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Src/MAPK/ERK path… DOI 10.1038/s41419-018-0574-1↗
- Yang Li · 2 shared papers, 2018–2025 · strength 0.32
- Paper Correction to: Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Sr… DOI 10.1038/s41419-025-07495-2↗
- Paper Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Src/MAPK/ERK path… DOI 10.1038/s41419-018-0574-1↗
- Zhen Wang · 2 shared papers, 2018–2025 · strength 0.32
- Paper Correction to: Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Sr… DOI 10.1038/s41419-025-07495-2↗
- Paper Natural compound Oblongifolin C confers gemcitabine resistance in pancreatic cancer by downregulating Src/MAPK/ERK path… DOI 10.1038/s41419-018-0574-1↗
- Hongyu Li · 2 shared papers, 2013–2015 · strength 0.10
- Paper miR-33a suppresses the nuclear translocation of β-catenin to enhance gemcitabine sensitivity in human pancreatic cancer… DOI 10.1007/s13277-015-3679-5↗
- Paper Pim-3 promotes the growth of human pancreatic cancer in the orthotopic nude mouse model through vascular endothelium gr… DOI 10.1016/j.jss.2013.06.004↗
- Zhouyu Ning · 1 shared paper, 2016 · strength 0.08
- Haiyong Wang · 1 shared paper, 2016 · strength 0.08
- Xiaoyan Zhu · 1 shared paper, 2016 · strength 0.08
- Zhiqiang Meng · 1 shared paper, 2016 · strength 0.08
- Fei Zhang · 1 shared paper, 2015 · strength 0.06
- Bin Liu · 1 shared paper, 2013 · strength 0.04
- Li Wu · 1 shared paper, 2013 · strength 0.04
- Chen Wang · 1 shared paper, 2013 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.