ResearcherPhilipps University of MarburgActive in the last 2 years
Günter Klöppel
Matched on ORCID iD 0000-0002-6688-086X; OpenAlex author ID A5089124733. Sources: OpenAlex.
Their work
- Papers
- 15 (first author 1, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 79
2011: 1 paper · 2012: 3 papers · 2013: 2 papers · 2014: 1 paper · 2015: none · 2016: 2 papers · 2017: none · 2018: 3 papers · 2019: none · 2020: 1 paper · 2021: none · 2022: none · 2023: 1 paper · 2024: none · 2025: none · 2026: 1 paper
Papers 15
- Pancreatic Neuroendocrine Tumor with Acinar Pattern Mimicking Pancreatic Ductal Adenocarcinoma: A Case Report. Co-author DOI 10.70352/scrj.cr.26-0184↗
- Molecular heterogeneity and commonalities in pancreatic cancer precursors with gastric and intestinal phenotype. Co-author DOI 10.1136/gutjnl-2021-326550↗
- Utility of pVHL, maspin, IMP3, S100P and Ki67 in the distinction of autoimmune pancreatitis from pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.prp.2020.152925↗
- Evolutionary routes and KRAS dosage define pancreatic cancer phenotypes. Co-author DOI 10.1038/nature25459↗
- Pancreatic Ductal Adenocarcinoma Subtyping Using the Biomarkers Hepatocyte Nuclear Factor-1A and Cytokeratin-81 Correlates with Outcome and Treatment Response. Co-author DOI 10.1158/1078-0432.ccr-17-2180↗
- The Combination of MiRNA-196b, LCN2, and TIMP1 is a Potential Set of Circulating Biomarkers for Screening Individuals at Risk for Familial Pancreatic Cancer. Co-author DOI 10.3390/jcm7100295↗
- Refinement of screening for familial pancreatic cancer. Co-author DOI 10.1136/gutjnl-2015-311098↗
- Benefit of Surveillance for Pancreatic Cancer in High-Risk Individuals: Outcome of Long-Term Prospective Follow-Up Studies From Three European Expert Centers. Co-author DOI 10.1200/jco.2015.64.0730↗
- MicroRNA-196a and -196b as Potential Biomarkers for the Early Detection of Familial Pancreatic Cancer. Co-author DOI 10.1016/j.tranon.2014.05.007↗
- [IgG4-related systemic disease with autoimmune pancreatitis and lung involvement primarily presenting as pancreatic cancer with pulmonary metastases]. First author DOI 10.1055/s-0032-1330533↗
- Variation in precursor lesions of pancreatic cancer among high-risk groups. Co-author DOI 10.1158/1078-0432.ccr-12-2730↗
- Lack of CCR7 expression is rate limiting for lymphatic spread of pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.26502↗
- Origin of pancreatic ductal adenocarcinoma from atypical flat lesions: a comparative study in transgenic mice and human tissues. Co-author DOI 10.1002/path.3017↗
- [New insights into the origin of pancreatic cancer. Role of atypical flat lesions in pancreatic carcinogenesis]. Last author DOI 10.1007/s00292-012-1673-x↗
- Stat3/Socs3 activation by IL-6 transsignaling promotes progression of pancreatic intraepithelial neoplasia and development of pancreatic cancer. Co-author DOI 10.1016/j.ccr.2011.03.009↗
Closest collaborators
The 25 strongest of 79 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Iréne Esposito · 6 shared papers, 2012–2023 · strength 0.36
- Paper Molecular heterogeneity and commonalities in pancreatic cancer precursors with gastric and intestinal phenotype. DOI 10.1136/gutjnl-2021-326550↗
- Paper Pancreatic Ductal Adenocarcinoma Subtyping Using the Biomarkers Hepatocyte Nuclear Factor-1A and Cytokeratin-81 Correla… DOI 10.1158/1078-0432.ccr-17-2180↗
- Paper [New insights into the origin of pancreatic cancer. Role of atypical flat lesions in pancreatic carcinogenesis]. DOI 10.1007/s00292-012-1673-x↗
- Anna Melissa Schlitter · 3 shared papers, 2012–2023 · strength 0.26
- Paper Molecular heterogeneity and commonalities in pancreatic cancer precursors with gastric and intestinal phenotype. DOI 10.1136/gutjnl-2021-326550↗
- Paper Pancreatic Ductal Adenocarcinoma Subtyping Using the Biomarkers Hepatocyte Nuclear Factor-1A and Cytokeratin-81 Correla… DOI 10.1158/1078-0432.ccr-17-2180↗
- Paper [New insights into the origin of pancreatic cancer. Role of atypical flat lesions in pancreatic carcinogenesis]. DOI 10.1007/s00292-012-1673-x↗
- Atsuko Kasajima · 1 shared paper, 2026 · strength 0.25
- Kei Yamane · 1 shared paper, 2026 · strength 0.25
- Kazuyuki Nagai · 1 shared paper, 2026 · strength 0.25
- Etsuro Hatano · 1 shared paper, 2026 · strength 0.25
- Yasunori Shirakawa · 1 shared paper, 2026 · strength 0.25
- Shinya Okumura · 1 shared paper, 2026 · strength 0.25
- Detlef Klaus Bartsch · 6 shared papers, 2012–2018 · strength 0.24
- Paper The Combination of MiRNA-196b, LCN2, and TIMP1 is a Potential Set of Circulating Biomarkers for Screening Individuals a… DOI 10.3390/jcm7100295↗
- Paper Refinement of screening for familial pancreatic cancer. DOI 10.1136/gutjnl-2015-311098↗
- Paper Benefit of Surveillance for Pancreatic Cancer in High-Risk Individuals: Outcome of Long-Term Prospective Follow-Up Stud… DOI 10.1200/jco.2015.64.0730↗
- Emily P. Slater · 5 shared papers, 2013–2018 · strength 0.22
- Paper The Combination of MiRNA-196b, LCN2, and TIMP1 is a Potential Set of Circulating Biomarkers for Screening Individuals a… DOI 10.3390/jcm7100295↗
- Paper Refinement of screening for familial pancreatic cancer. DOI 10.1136/gutjnl-2015-311098↗
- Paper Benefit of Surveillance for Pancreatic Cancer in High-Risk Individuals: Outcome of Long-Term Prospective Follow-Up Stud… DOI 10.1200/jco.2015.64.0730↗
- Andreas Trumpp · 2 shared papers, 2018–2023 · strength 0.21
- Paper Molecular heterogeneity and commonalities in pancreatic cancer precursors with gastric and intestinal phenotype. DOI 10.1136/gutjnl-2021-326550↗
- Paper Pancreatic Ductal Adenocarcinoma Subtyping Using the Biomarkers Hepatocyte Nuclear Factor-1A and Cytokeratin-81 Correla… DOI 10.1158/1078-0432.ccr-17-2180↗
- Peter Langer · 5 shared papers, 2012–2016 · strength 0.18
- Paper Refinement of screening for familial pancreatic cancer. DOI 10.1136/gutjnl-2015-311098↗
- Paper Benefit of Surveillance for Pancreatic Cancer in High-Risk Individuals: Outcome of Long-Term Prospective Follow-Up Stud… DOI 10.1200/jco.2015.64.0730↗
- Paper MicroRNA-196a and -196b as Potential Biomarkers for the Early Detection of Familial Pancreatic Cancer. DOI 10.1016/j.tranon.2014.05.007↗
- Jens Thomas Siveke · 2 shared papers, 2012–2023 · strength 0.18
- Paper Molecular heterogeneity and commonalities in pancreatic cancer precursors with gastric and intestinal phenotype. DOI 10.1136/gutjnl-2021-326550↗
- Paper Origin of pancreatic ductal adenocarcinoma from atypical flat lesions: a comparative study in transgenic mice and human… DOI 10.1002/path.3017↗
- Sven‐Thorsten Liffers · 1 shared paper, 2023 · strength 0.15
- Elisa Espinet · 1 shared paper, 2023 · strength 0.15
- Nikolas Hendrik Stoecklein · 1 shared paper, 2023 · strength 0.15
- Aslihan Yavas · 1 shared paper, 2023 · strength 0.15
- Lena Häberle · 1 shared paper, 2023 · strength 0.15
- Laura Godfrey · 1 shared paper, 2023 · strength 0.15
- Wolfram Trudo Knoefel · 1 shared paper, 2023 · strength 0.15
- Gitte Hedegaard Jensen · 1 shared paper, 2020 · strength 0.13
- Michael Friberg Bruun Nielsen · 1 shared paper, 2020 · strength 0.13
- Michael Bau Mortensen · 1 shared paper, 2020 · strength 0.13
- Sönke Detlefsen · 1 shared paper, 2020 · strength 0.13
- Volker Fendrich · 3 shared papers, 2014–2016 · strength 0.12
- Paper Refinement of screening for familial pancreatic cancer. DOI 10.1136/gutjnl-2015-311098↗
- Paper Benefit of Surveillance for Pancreatic Cancer in High-Risk Individuals: Outcome of Long-Term Prospective Follow-Up Stud… DOI 10.1200/jco.2015.64.0730↗
- Paper MicroRNA-196a and -196b as Potential Biomarkers for the Early Detection of Familial Pancreatic Cancer. DOI 10.1016/j.tranon.2014.05.007↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.