ResearcherUniversity of Milano-BicoccaActive in the last 2 years
Ferdinando Chiaradonna
Matched on ORCID iD 0000-0001-8529-2732; OpenAlex author ID A5065251555. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 0, last author 4)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 18
2020: 1 paper · 2021: 2 papers · 2022: none · 2023: 1 paper · 2024: none · 2025: none · 2026: 1 paper
Papers 5
- PGM3 inhibition rewires RUVBL2-dependent DNA repair and induces a BRCAness-like state in pancreatic cancer cells Last author DOI 10.64898/2026.08.31.746486↗
- PGM3 inhibition shows cooperative effects with erastin inducing pancreatic cancer cell death via activation of the unfolded protein response. Last author DOI 10.3389/fonc.2023.1125855↗
- Suppression of the HBP Function Increases Pancreatic Cancer Cell Sensitivity to a Pan-RAS Inhibitor. Last author DOI 10.3390/cells10020431↗
- The Role of Mitochondria in the Chemoresistance of Pancreatic Cancer Cells. Co-author DOI 10.3390/cells10030497↗
- Hexosamine pathway inhibition overcomes pancreatic cancer resistance to gemcitabine through unfolded protein response and EGFR-Akt pathway modulation. Last author DOI 10.1038/s41388-020-1260-1↗
Closest collaborators
The 18 strongest of 18 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Barbara Zerbato · 2 shared papers, 2023–2026 · strength 0.43
- Paper PGM3 inhibition rewires RUVBL2-dependent DNA repair and induces a BRCAness-like state in pancreatic cancer cells DOI 10.64898/2026.08.31.746486↗
- Paper PGM3 inhibition shows cooperative effects with erastin inducing pancreatic cancer cell death via activation of the unfo… DOI 10.3389/fonc.2023.1125855↗
- Francesca Ricciardiello · 3 shared papers, 2020–2021 · strength 0.36
- Paper Suppression of the HBP Function Increases Pancreatic Cancer Cell Sensitivity to a Pan-RAS Inhibitor. DOI 10.3390/cells10020431↗
- Paper The Role of Mitochondria in the Chemoresistance of Pancreatic Cancer Cells. DOI 10.3390/cells10030497↗
- Paper Hexosamine pathway inhibition overcomes pancreatic cancer resistance to gemcitabine through unfolded protein response a… DOI 10.1038/s41388-020-1260-1↗
- Gang Yang · 3 shared papers, 2020–2021 · strength 0.36
- Paper Suppression of the HBP Function Increases Pancreatic Cancer Cell Sensitivity to a Pan-RAS Inhibitor. DOI 10.3390/cells10020431↗
- Paper The Role of Mitochondria in the Chemoresistance of Pancreatic Cancer Cells. DOI 10.3390/cells10030497↗
- Paper Hexosamine pathway inhibition overcomes pancreatic cancer resistance to gemcitabine through unfolded protein response a… DOI 10.1038/s41388-020-1260-1↗
- Taiping Zhang · 3 shared papers, 2020–2021 · strength 0.36
- Paper Suppression of the HBP Function Increases Pancreatic Cancer Cell Sensitivity to a Pan-RAS Inhibitor. DOI 10.3390/cells10020431↗
- Paper The Role of Mitochondria in the Chemoresistance of Pancreatic Cancer Cells. DOI 10.3390/cells10030497↗
- Paper Hexosamine pathway inhibition overcomes pancreatic cancer resistance to gemcitabine through unfolded protein response a… DOI 10.1038/s41388-020-1260-1↗
- Fangyu Zhao · 2 shared papers, 2020–2021 · strength 0.21
- Lei You · 2 shared papers, 2020–2021 · strength 0.21
- Virginia Brancato · 1 shared paper, 2023 · strength 0.19
- Jianchun Xiao · 1 shared paper, 2021 · strength 0.11
- Yueze Liu · 1 shared paper, 2021 · strength 0.11
- Yibo Fu · 1 shared paper, 2021 · strength 0.11
- Wenhao Luo · 1 shared paper, 2021 · strength 0.11
- Hua Huang · 1 shared paper, 2021 · strength 0.11
- Zhe Cao · 1 shared paper, 2021 · strength 0.11
- Guangyu Chen · 1 shared paper, 2021 · strength 0.11
- Jiangdong Qiu · 1 shared paper, 2021 · strength 0.11
- Quanxiao Li · 1 shared paper, 2020 · strength 0.10
- Jin Gu · 1 shared paper, 2020 · strength 0.10
- Yupei Zhao · 1 shared paper, 2020 · strength 0.10
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.