ResearcherUniversity of PisaActive in the last 2 years
Maarten F. Bijlsma
Matched on ORCID iD 0000-0001-6627-3229; OpenAlex author ID A5000969662, A5086619378. Sources: OpenAlex.
Their work
- Papers
- 71 (first author 5, last author 16)
- Trials
- 0
- Grants
- 0
- Labs led
- 2
- Collaborators
- 149
2013: 1 paper · 2014: none · 2015: 5 papers · 2016: 2 papers · 2017: 2 papers · 2018: 5 papers · 2019: 4 papers · 2020: 11 papers · 2021: 9 papers · 2022: 6 papers · 2023: 6 papers · 2024: 7 papers · 2025: 9 papers · 2026: 4 papers
Papers 50 of 71
- Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy of Overall Survival Following Adjuvant T… Co-author DOI 10.1002/ijc.70519↗
- Precision targeting of stromal states in pancreatic cancer: a clinical perspective. First author DOI 10.1016/j.trecan.2026.07.003↗
- Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective multicenter study across expert centers. Co-author DOI 10.1093/oncolo/oyag373↗
- Estrogen Production in Pancreatic Cancer Shapes a Tumor-Suppressive Stromal Microenvironment. Last author DOI 10.1158/0008-5472.can-24-4707↗
- Association of epigenetic landscapes with heterogeneity and plasticity in pancreatic cancer. Last author DOI 10.1016/j.critrevonc.2024.104573↗
- KRAS mutation status integrated with RNA subtyping improves prognostic modeling in FOLFIRINOX-treated metastatic pancreatic cancer. Last author DOI 10.1016/j.medj.2025.100601↗
- Organoids, tissue slices and organotypic cultures: Advancing our understanding of pancreatic ductal adenocarcinoma through in vitro and ex vivo models. Last author DOI 10.1016/j.semcancer.2024.12.003↗
- CRISPR/Cas9 genome engineering in PDAC: From preclinical studies to translation and clinical research. Co-author DOI 10.1016/j.semcancer.2025.07.004↗
- Tumor gene expression is associated with venous thromboembolism in patients with ductal pancreatic adenocarcinoma. Co-author DOI 10.1016/j.thromres.2024.109240↗
- Resectability stages of pancreatic cancer: nationwide reference. Co-author DOI 10.1093/bjs/znaf120↗
- A pleiotropy scan to discover new susceptibility loci for pancreatic ductal adenocarcinoma. Co-author DOI 10.1093/mutage/geae012↗
- Patient preferences for pancreatic cancer treatment (PERSEUS): a multicenter discrete choice experiment. Co-author DOI 10.1186/s12955-025-02440-5↗
- Stromal Hedgehog Signaling Is Associated with Favorable Outcomes in Pancreatic Cancer. Last author DOI 10.3390/ijms26115200↗
- Kinase activities in pancreatic ductal adenocarcinoma with prognostic and therapeutic avenues. Last author DOI 10.1002/1878-0261.13625↗
- CAPN2-responsive mesoporous silica nanoparticles: A promising nanocarrier for targeted therapy of pancreatic cancer. Co-author DOI 10.1016/j.canlet.2024.216845↗
- Transcriptome-based classification to predict FOLFIRINOX response in a real-world metastatic pancreatic cancer cohort. Last author DOI 10.1016/j.trsl.2024.08.002↗
- Identification of pancreatic cancer-specific protease substrates for protease-dependent targeted delivery. Co-author DOI 10.1038/s41389-024-00542-1↗
- Pancreatic cancer-associated fibroblasts modulate macrophage differentiation via sialic acid-Siglec interactions. Co-author DOI 10.1038/s42003-024-06087-8↗
- Polymorphisms in transcription factor binding sites and enhancer regions and pancreatic ductal adenocarcinoma risk. Co-author DOI 10.1186/s40246-024-00576-x↗
- Hypoxia Abrogates Tumor-Suppressive Activities of C/EBPδ in Pancreatic Cancer. Co-author DOI 10.3390/ijms25179449↗
- Serum levels of iCAF-derived osteoglycin predict favorable outcome in pancreatic cancer. Last author DOI 10.1002/ijc.34276↗
- Phosphoproteomics guides effective low-dose drug combinations against pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.celrep.2023.112581↗
- The PANcreatic Disease ReseArch (PANDoRA) consortium: Ten years' experience of association studies to understand the genetic architecture of pancreatic cancer. Co-author DOI 10.1016/j.critrevonc.2023.104020↗
- Genetic and non-genetic risk factors for early-onset pancreatic cancer. Co-author DOI 10.1016/j.dld.2023.02.023↗
- C/EBP-Family Redundancy Determines Patient Survival and Lymph Node Involvement in PDAC. Co-author DOI 10.3390/ijms24021537↗
- Preclinical Assessment of ADAM9-Responsive Mesoporous Silica Nanoparticles for the Treatment of Pancreatic Cancer. Co-author DOI 10.3390/ijms241310704↗
- Analysis of the glyco-code in pancreatic ductal adenocarcinoma identifies glycan-mediated immune regulatory circuits. Co-author DOI 10.1038/s42003-021-02934-0↗
- Impact of classical and basal-like molecular subtypes on overall survival in resected pancreatic cancer in the SPACIOUS-2 multicentre study. Co-author DOI 10.1093/bjs/znac272↗
- The contributions of hypoxia to poor outcomes in pancreatic cancer. Co-author DOI 10.21037/tcr-22-2167↗
- Editorial: Translational Insights Into Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3389/fcell.2022.875836↗
- C/EBPδ Suppresses Motility-Associated Gene Signatures and Reduces PDAC Cell Migration. Co-author DOI 10.3390/cells11213334↗
- Mesoporous Silica Nanoparticle-Based Drug Delivery Systems for the Treatment of Pancreatic Cancer: A Systematic Literature Overview. Co-author DOI 10.3390/pharmaceutics14020390↗
- Marker-free lineage tracing reveals an environment-instructed clonogenic hierarchy in pancreatic cancer. Co-author DOI 10.1016/j.celrep.2021.109852↗
- The case for a stratified application of targeted agents against pancreatic cancer. Co-author DOI 10.1016/j.ebiom.2021.103344↗
- Readily available biomarkers predict poor survival in metastatic pancreatic cancer. Co-author DOI 10.1080/1354750x.2021.1893814↗
- Polygenic and multifactorial scores for pancreatic ductal adenocarcinoma risk prediction. Co-author DOI 10.1136/jmedgenet-2020-106961↗
- Genetic Polymorphisms Involved in Mitochondrial Metabolism and Pancreatic Cancer Risk. Co-author DOI 10.1158/1055-9965.epi-21-0353↗
- Focal adhesion kinase inhibition synergizes with nab-paclitaxel to target pancreatic ductal adenocarcinoma. First author DOI 10.1186/s13046-021-01892-z↗
- The Cellular Origins of Cancer-Associated Fibroblasts and Their Opposing Contributions to Pancreatic Cancer Growth. First author DOI 10.3389/fcell.2021.743907↗
- Non-Tumor CCAAT/Enhancer-Binding Protein Delta Potentiates Tumor Cell Extravasation and Pancreatic Cancer Metastasis Formation. Co-author DOI 10.3390/biom11081079↗
- ADAM9-Responsive Mesoporous Silica Nanoparticles for Targeted Drug Delivery in Pancreatic Cancer. Co-author DOI 10.3390/cancers13133321↗
- Circulating tumor DNA quantity is related to tumor volume and both predict survival in metastatic pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.32586↗
- Co-expression analysis of pancreatic cancer proteome reveals biology and prognostic biomarkers. Co-author DOI 10.1007/s13402-020-00548-y↗
- Macrophage-secreted MMP9 induces mesenchymal transition in pancreatic cancer cells via PAR1 activation. Co-author DOI 10.1007/s13402-020-00549-x↗
- Unsupervised class discovery in pancreatic ductal adenocarcinoma reveals cell-intrinsic mesenchymal features and high concordance between existing classificati… Last author DOI 10.1038/s41598-019-56826-9↗
- Microdissected pancreatic cancer proteomes reveal tumor heterogeneity and therapeutic targets. Last author DOI 10.1172/jci.insight.138290↗
- Early macrophage infiltrates impair pancreatic cancer cell growth by TNF-α secretion. Co-author DOI 10.1186/s12885-020-07697-1↗
- High-grade mesenchymal pancreatic ductal adenocarcinoma drives stromal deactivation through CSF-1. Last author DOI 10.15252/embr.201948780↗
- Combined Expression of Plasma Thrombospondin-2 and CA19-9 for Diagnosis of Pancreatic Cancer and Distal Cholangiocarcinoma: A Proteome Approach. Co-author DOI 10.1634/theoncologist.2019-0680↗
- Matrix Metalloproteases in Pancreatic Ductal Adenocarcinoma: Key Drivers of Disease Progression? Co-author DOI 10.3390/biology9040080↗
Closest collaborators
The 25 strongest of 149 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hanneke W.M. van Laarhoven · 31 shared papers, 2015–2026 · strength 4.24
- Paper Association of epigenetic landscapes with heterogeneity and plasticity in pancreatic cancer. DOI 10.1016/j.critrevonc.2024.104573↗
- Paper Stromal Hedgehog Signaling Is Associated with Favorable Outcomes in Pancreatic Cancer. DOI 10.3390/ijms26115200↗
- Paper Estrogen Production in Pancreatic Cancer Shapes a Tumor-Suppressive Stromal Microenvironment. DOI 10.1158/0008-5472.can-24-4707↗
- C. Arnold Spek · 14 shared papers, 2018–2024 · strength 2.53
- Paper Identification of pancreatic cancer-specific protease substrates for protease-dependent targeted delivery. DOI 10.1038/s41389-024-00542-1↗
- Paper CAPN2-responsive mesoporous silica nanoparticles: A promising nanocarrier for targeted therapy of pancreatic cancer. DOI 10.1016/j.canlet.2024.216845↗
- Paper Hypoxia Abrogates Tumor-Suppressive Activities of C/EBPδ in Pancreatic Cancer. DOI 10.3390/ijms25179449↗
- Marc G. Besselink · 16 shared papers, 2015–2026 · strength 1.96
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper Patient preferences for pancreatic cancer treatment (PERSEUS): a multicenter discrete choice experiment. DOI 10.1186/s12955-025-02440-5↗
- Paper Tumor gene expression is associated with venous thromboembolism in patients with ductal pancreatic adenocarcinoma. DOI 10.1016/j.thromres.2024.109240↗
- Johanna W. Wilmink · 13 shared papers, 2015–2026 · strength 1.92
- Paper Estrogen Production in Pancreatic Cancer Shapes a Tumor-Suppressive Stromal Microenvironment. DOI 10.1158/0008-5472.can-24-4707↗
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper KRAS mutation status integrated with RNA subtyping improves prognostic modeling in FOLFIRINOX-treated metastatic pancre… DOI 10.1016/j.medj.2025.100601↗
- Paul Manoukian · 8 shared papers, 2021–2026 · strength 1.86
- Paper Association of epigenetic landscapes with heterogeneity and plasticity in pancreatic cancer. DOI 10.1016/j.critrevonc.2024.104573↗
- Paper Stromal Hedgehog Signaling Is Associated with Favorable Outcomes in Pancreatic Cancer. DOI 10.3390/ijms26115200↗
- Paper Estrogen Production in Pancreatic Cancer Shapes a Tumor-Suppressive Stromal Microenvironment. DOI 10.1158/0008-5472.can-24-4707↗
- Frederike Dijk · 13 shared papers, 2018–2026 · strength 1.76
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper C/EBP-Family Redundancy Determines Patient Survival and Lymph Node Involvement in PDAC. DOI 10.3390/ijms24021537↗
- Paper Kinase activities in pancreatic ductal adenocarcinoma with prognostic and therapeutic avenues. DOI 10.1002/1878-0261.13625↗
- Geert Kazemier M · 11 shared papers, 2016–2026 · strength 1.39
- Paper Estrogen Production in Pancreatic Cancer Shapes a Tumor-Suppressive Stromal Microenvironment. DOI 10.1158/0008-5472.can-24-4707↗
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective mult… DOI 10.1093/oncolo/oyag373↗
- Joanne Verheij · 10 shared papers, 2020–2026 · strength 1.38
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper Kinase activities in pancreatic ductal adenocarcinoma with prognostic and therapeutic avenues. DOI 10.1002/1878-0261.13625↗
- Paper Tumor gene expression is associated with venous thromboembolism in patients with ductal pancreatic adenocarcinoma. DOI 10.1016/j.thromres.2024.109240↗
- Etienne J. Slapak · 6 shared papers, 2020–2024 · strength 1.27
- Paper Identification of pancreatic cancer-specific protease substrates for protease-dependent targeted delivery. DOI 10.1038/s41389-024-00542-1↗
- Paper CAPN2-responsive mesoporous silica nanoparticles: A promising nanocarrier for targeted therapy of pancreatic cancer. DOI 10.1016/j.canlet.2024.216845↗
- Paper Mesoporous Silica Nanoparticle-Based Drug Delivery Systems for the Treatment of Pancreatic Cancer: A Systematic Literat… DOI 10.3390/pharmaceutics14020390↗
- Elisa Giovannetti · 11 shared papers, 2016–2024 · strength 1.26
- Paper Kinase activities in pancreatic ductal adenocarcinoma with prognostic and therapeutic avenues. DOI 10.1002/1878-0261.13625↗
- Paper Phosphoproteomics guides effective low-dose drug combinations against pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2023.112581↗
- Paper Pancreatic cancer-associated fibroblasts modulate macrophage differentiation via sialic acid-Siglec interactions. DOI 10.1038/s42003-024-06087-8↗
- Marjolein F. Lansbergen · 5 shared papers, 2024–2026 · strength 1.07
- Paper Estrogen Production in Pancreatic Cancer Shapes a Tumor-Suppressive Stromal Microenvironment. DOI 10.1158/0008-5472.can-24-4707↗
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper KRAS mutation status integrated with RNA subtyping improves prognostic modeling in FOLFIRINOX-treated metastatic pancre… DOI 10.1016/j.medj.2025.100601↗
- Mark P.G. Dings · 7 shared papers, 2020–2026 · strength 1.07
- Paper Estrogen Production in Pancreatic Cancer Shapes a Tumor-Suppressive Stromal Microenvironment. DOI 10.1158/0008-5472.can-24-4707↗
- Paper KRAS mutation status integrated with RNA subtyping improves prognostic modeling in FOLFIRINOX-treated metastatic pancre… DOI 10.1016/j.medj.2025.100601↗
- Paper Transcriptome-based classification to predict FOLFIRINOX response in a real-world metastatic pancreatic cancer cohort. DOI 10.1016/j.trsl.2024.08.002↗
- JanWillem Duitman · 6 shared papers, 2020–2024 · strength 1.02
- Paper Hypoxia Abrogates Tumor-Suppressive Activities of C/EBPδ in Pancreatic Cancer. DOI 10.3390/ijms25179449↗
- Paper C/EBP-Family Redundancy Determines Patient Survival and Lymph Node Involvement in PDAC. DOI 10.3390/ijms24021537↗
- Paper Non-Tumor CCAAT/Enhancer-Binding Protein Delta Potentiates Tumor Cell Extravasation and Pancreatic Cancer Metastasis Fo… DOI 10.3390/biom11081079↗
- Izaak Quintus Molenaar · 6 shared papers, 2022–2026 · strength 0.99
- Paper Patient preferences for pancreatic cancer treatment (PERSEUS): a multicenter discrete choice experiment. DOI 10.1186/s12955-025-02440-5↗
- Paper Tumor gene expression is associated with venous thromboembolism in patients with ductal pancreatic adenocarcinoma. DOI 10.1016/j.thromres.2024.109240↗
- Paper Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective mult… DOI 10.1093/oncolo/oyag373↗
- Connie R. Jiménez · 8 shared papers, 2017–2024 · strength 0.92
- Paper Kinase activities in pancreatic ductal adenocarcinoma with prognostic and therapeutic avenues. DOI 10.1002/1878-0261.13625↗
- Paper Phosphoproteomics guides effective low-dose drug combinations against pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2023.112581↗
- Paper Focal adhesion kinase inhibition synergizes with nab-paclitaxel to target pancreatic ductal adenocarcinoma. DOI 10.1186/s13046-021-01892-z↗
- Ignace H. J. T. de Hingh · 5 shared papers, 2023–2026 · strength 0.91
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper Patient preferences for pancreatic cancer treatment (PERSEUS): a multicenter discrete choice experiment. DOI 10.1186/s12955-025-02440-5↗
- Paper Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective mult… DOI 10.1093/oncolo/oyag373↗
- Judith de Vos‐Geelen · 5 shared papers, 2021–2026 · strength 0.88
- Paper KRAS mutation status integrated with RNA subtyping improves prognostic modeling in FOLFIRINOX-treated metastatic pancre… DOI 10.1016/j.medj.2025.100601↗
- Paper Patient preferences for pancreatic cancer treatment (PERSEUS): a multicenter discrete choice experiment. DOI 10.1186/s12955-025-02440-5↗
- Paper Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective mult… DOI 10.1093/oncolo/oyag373↗
- Marjolein Y.V. Homs · 5 shared papers, 2021–2026 · strength 0.88
- Paper KRAS mutation status integrated with RNA subtyping improves prognostic modeling in FOLFIRINOX-treated metastatic pancre… DOI 10.1016/j.medj.2025.100601↗
- Paper Patient preferences for pancreatic cancer treatment (PERSEUS): a multicenter discrete choice experiment. DOI 10.1186/s12955-025-02440-5↗
- Paper Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective mult… DOI 10.1093/oncolo/oyag373↗
- Leonie Hartl · 5 shared papers, 2020–2024 · strength 0.88
- Paper Hypoxia Abrogates Tumor-Suppressive Activities of C/EBPδ in Pancreatic Cancer. DOI 10.3390/ijms25179449↗
- Paper C/EBP-Family Redundancy Determines Patient Survival and Lymph Node Involvement in PDAC. DOI 10.3390/ijms24021537↗
- Paper Non-Tumor CCAAT/Enhancer-Binding Protein Delta Potentiates Tumor Cell Extravasation and Pancreatic Cancer Metastasis Fo… DOI 10.3390/biom11081079↗
- Casper H.J. van Eijck · 5 shared papers, 2019–2026 · strength 0.82
- Paper Transcriptome-Based Classification of Resected Pancreatic Ductal Adenocarcinoma Enhances Prognostic Modelling Accuracy … DOI 10.1002/ijc.70519↗
- Paper Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective mult… DOI 10.1093/oncolo/oyag373↗
- Paper Resectability stages of pancreatic cancer: nationwide reference. DOI 10.1093/bjs/znaf120↗
- Bert A. Bonsing · 5 shared papers, 2022–2025 · strength 0.81
- Paper Patient preferences for pancreatic cancer treatment (PERSEUS): a multicenter discrete choice experiment. DOI 10.1186/s12955-025-02440-5↗
- Paper Tumor gene expression is associated with venous thromboembolism in patients with ductal pancreatic adenocarcinoma. DOI 10.1016/j.thromres.2024.109240↗
- Paper Resectability stages of pancreatic cancer: nationwide reference. DOI 10.1093/bjs/znaf120↗
- Tessa Y. S. Le Large · 8 shared papers, 2016–2022 · strength 0.75
- Paper Analysis of the glyco-code in pancreatic ductal adenocarcinoma identifies glycan-mediated immune regulatory circuits. DOI 10.1038/s42003-021-02934-0↗
- Paper Focal adhesion kinase inhibition synergizes with nab-paclitaxel to target pancreatic ductal adenocarcinoma. DOI 10.1186/s13046-021-01892-z↗
- Paper Co-expression analysis of pancreatic cancer proteome reveals biology and prognostic biomarkers. DOI 10.1007/s13402-020-00548-y↗
- Helene Damhofer · 6 shared papers, 2013–2025 · strength 0.64
- Paper Stromal Hedgehog Signaling Is Associated with Favorable Outcomes in Pancreatic Cancer. DOI 10.3390/ijms26115200↗
- Paper Unsupervised class discovery in pancreatic ductal adenocarcinoma reveals cell-intrinsic mesenchymal features and high c… DOI 10.1038/s41598-019-56826-9↗
- Paper Stromal SPOCK1 supports invasive pancreatic cancer growth. DOI 10.1002/1878-0261.12073↗
- Jeanin Elise van Hooft · 5 shared papers, 2018–2026 · strength 0.61
- Paper Real-world implementation of a decision aid for treatment decisions in metastatic pancreatic cancer: a prospective mult… DOI 10.1093/oncolo/oyag373↗
- Paper Resectability stages of pancreatic cancer: nationwide reference. DOI 10.1093/bjs/znaf120↗
- Paper Readily available biomarkers predict poor survival in metastatic pancreatic cancer. DOI 10.1080/1354750x.2021.1893814↗
- Cansu Tekin · 5 shared papers, 2018–2020 · strength 0.61
- Paper Early macrophage infiltrates impair pancreatic cancer cell growth by TNF-α secretion. DOI 10.1186/s12885-020-07697-1↗
- Paper Matrix Metalloproteases in Pancreatic Ductal Adenocarcinoma: Key Drivers of Disease Progression? DOI 10.3390/biology9040080↗
- Paper Macrophage-secreted MMP9 induces mesenchymal transition in pancreatic cancer cells via PAR1 activation. DOI 10.1007/s13402-020-00549-x↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.