ResearcherUniversity of Mississippi Medical CenterActive in the last 2 years
Seema Singh
Matched on ORCID iD 0000-0002-7188-3803; OpenAlex author ID A5115603189; NIH RePORTER profile ID 10099269. Sources: OpenAlex.
Their work
- Papers
- 22 (first author 0, last author 4)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 19
2011: 2 papers · 2012: 1 paper · 2013: 1 paper, 1 grant · 2014: 1 paper · 2015: 3 papers · 2016: 3 papers · 2017: 2 papers · 2018: 1 paper · 2019: 1 paper · 2020: 3 papers · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: 1 paper · 2025: 1 paper
Papers 22
- MYB and HIF1α crosstalk drives hypoxia-induced transcriptional reprogramming and adaptive signaling alterations in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2025.217916↗
- Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function. Co-author DOI 10.1158/1541-7786.mcr-23-0138↗
- MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. Co-author DOI 10.15252/embr.202255643↗
- Detection of mitochondrial DNA mutations in circulating mitochondria-originated extracellular vesicles for potential diagnostic applications in pancreatic aden… Co-author DOI 10.1038/s41598-022-22006-5↗
- Hypoxia alters the release and size distribution of extracellular vesicles in pancreatic cancer cells to support their adaptive survival. Co-author DOI 10.1002/jcb.29328↗
- Co-targeting of CXCR4 and hedgehog pathways disrupts tumor-stromal crosstalk and improves chemotherapeutic efficacy in pancreatic cancer. Co-author DOI 10.1074/jbc.ra119.011748↗
- Comprehensive Analysis of Expression, Clinicopathological Association and Potential Prognostic Significance of RABs in Pancreatic Cancer. Co-author DOI 10.3390/ijms21155580↗
- Exosomal markers (CD63 and CD9) expression and their prognostic significance using immunohistochemistry in patients with pancreatic ductal adenocarcinoma. Co-author DOI 10.21037/jgo.2018.07.02↗
- ETV4: an emerging target in pancreatic cancer. Last author DOI 10.18632/oncoscience.471↗
- Exosomes confer chemoresistance to pancreatic cancer cells by promoting ROS detoxification and miR-155-mediated suppression of key gemcitabine-metabolising enz… Co-author DOI 10.1038/bjc.2017.18↗
- Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward. Co-author DOI 10.3390/ijms18040779↗
- p-21 activated kinase 4 (PAK4) maintains stem cell-like phenotypes in pancreatic cancer cells through activation of STAT3 signaling. Last author DOI 10.1016/j.canlet.2015.10.028↗
- Deep sequencing and in silico analyses identify MYB-regulated gene networks and signaling pathways in pancreatic cancer. Co-author DOI 10.1038/srep28446↗
- MYB Promotes Desmoplasia in Pancreatic Cancer through Direct Transcriptional Up-regulation and Cooperative Action of Sonic Hedgehog and Adrenomedullin. Co-author DOI 10.1074/jbc.m116.732651↗
- Insights into the Role of microRNAs in Pancreatic Cancer Pathogenesis: Potential for Diagnosis, Prognosis, and Therapy. Co-author DOI 10.1007/978-3-319-23730-5_5↗
- MicroRNA-345 induces apoptosis in pancreatic cancer cells through potentiation of caspase-dependent and -independent pathways. Co-author DOI 10.1038/bjc.2015.252↗
- Gemcitabine triggers angiogenesis-promoting molecular signals in pancreatic cancer cells: Therapeutic implications. Co-author DOI 10.18632/oncotarget.3784↗
- p-21 activated kinase 4 promotes proliferation and survival of pancreatic cancer cells through AKT- and ERK-dependent activation of NF-κB pathway. Last author DOI 10.18632/oncotarget.2398↗
- An undesired effect of chemotherapy: gemcitabine promotes pancreatic cancer cell invasiveness through reactive oxygen species-dependent, nuclear factor κB- and… Co-author DOI 10.1074/jbc.m113.484576↗
- CXCL12/CXCR4 protein signaling axis induces sonic hedgehog expression in pancreatic cancer cells via extracellular regulated kinase- and Akt kinase-mediated ac… Last author DOI 10.1074/jbc.m112.409581↗
- MicroRNA-150 directly targets MUC4 and suppresses growth and malignant behavior of pancreatic cancer cells. Co-author DOI 10.1093/carcin/bgr223↗
- Honokiol arrests cell cycle, induces apoptosis, and potentiates the cytotoxic effect of gemcitabine in human pancreatic cancer cells. Co-author DOI 10.1371/journal.pone.0021573↗
Grants 1
Closest collaborators
The 19 strongest of 19 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Ajay P. Singh · 21 shared papers, 2011–2024 · strength 1.83
- Paper Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mit… DOI 10.1158/1541-7786.mcr-23-0138↗
- Paper MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. DOI 10.15252/embr.202255643↗
- Paper Detection of mitochondrial DNA mutations in circulating mitochondria-originated extracellular vesicles for potential di… DOI 10.1038/s41598-022-22006-5↗
- Mohammad Aslam Khan · 11 shared papers, 2015–2025 · strength 1.55
- Paper MYB and HIF1α crosstalk drives hypoxia-induced transcriptional reprogramming and adaptive signaling alterations in panc… DOI 10.1016/j.canlet.2025.217916↗
- Paper Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mit… DOI 10.1158/1541-7786.mcr-23-0138↗
- Paper MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. DOI 10.15252/embr.202255643↗
- Shashi Anand · 5 shared papers, 2020–2025 · strength 1.03
- Paper MYB and HIF1α crosstalk drives hypoxia-induced transcriptional reprogramming and adaptive signaling alterations in panc… DOI 10.1016/j.canlet.2025.217916↗
- Paper Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mit… DOI 10.1158/1541-7786.mcr-23-0138↗
- Paper MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. DOI 10.15252/embr.202255643↗
- Kunwar Somesh Vikramdeo · 4 shared papers, 2022–2025 · strength 0.90
- Paper MYB and HIF1α crosstalk drives hypoxia-induced transcriptional reprogramming and adaptive signaling alterations in panc… DOI 10.1016/j.canlet.2025.217916↗
- Paper Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mit… DOI 10.1158/1541-7786.mcr-23-0138↗
- Paper MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. DOI 10.15252/embr.202255643↗
- Sanjeev K. Srivastava · 13 shared papers, 2011–2023 · strength 0.83
- Paper MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. DOI 10.15252/embr.202255643↗
- Paper Co-targeting of CXCR4 and hedgehog pathways disrupts tumor-stromal crosstalk and improves chemotherapeutic efficacy in … DOI 10.1074/jbc.ra119.011748↗
- Paper Exosomes confer chemoresistance to pancreatic cancer cells by promoting ROS detoxification and miR-155-mediated suppres… DOI 10.1038/bjc.2017.18↗
- Moh'd M. Khushman · 8 shared papers, 2016–2022 · strength 0.75
- Paper Detection of mitochondrial DNA mutations in circulating mitochondria-originated extracellular vesicles for potential di… DOI 10.1038/s41598-022-22006-5↗
- Paper Comprehensive Analysis of Expression, Clinicopathological Association and Potential Prognostic Significance of RABs in … DOI 10.3390/ijms21155580↗
- Paper Co-targeting of CXCR4 and hedgehog pathways disrupts tumor-stromal crosstalk and improves chemotherapeutic efficacy in … DOI 10.1074/jbc.ra119.011748↗
- Arun Bhardwaj · 13 shared papers, 2011–2019 · strength 0.67
- Paper Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward. DOI 10.3390/ijms18040779↗
- Paper Exosomal markers (CD63 and CD9) expression and their prognostic significance using immunohistochemistry in patients wit… DOI 10.21037/jgo.2018.07.02↗
- Paper Exosomes confer chemoresistance to pancreatic cancer cells by promoting ROS detoxification and miR-155-mediated suppres… DOI 10.1038/bjc.2017.18↗
- Santanu Dasgupta · 3 shared papers, 2020–2024 · strength 0.53
- Paper Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mit… DOI 10.1158/1541-7786.mcr-23-0138↗
- Paper Detection of mitochondrial DNA mutations in circulating mitochondria-originated extracellular vesicles for potential di… DOI 10.1038/s41598-022-22006-5↗
- Paper Comprehensive Analysis of Expression, Clinicopathological Association and Potential Prognostic Significance of RABs in … DOI 10.3390/ijms21155580↗
- Sachin Kumar Deshmukh · 3 shared papers, 2016–2023 · strength 0.37
- Paper MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. DOI 10.15252/embr.202255643↗
- Paper ETV4: an emerging target in pancreatic cancer. DOI 10.18632/oncoscience.471↗
- Paper p-21 activated kinase 4 (PAK4) maintains stem cell-like phenotypes in pancreatic cancer cells through activation of STA… DOI 10.1016/j.canlet.2015.10.028↗
- Ajay Pratap Singh · 1 shared paper, 2025 · strength 0.33
- Girijesh Kumar Patel · 4 shared papers, 2017–2020 · strength 0.32
- Paper Co-targeting of CXCR4 and hedgehog pathways disrupts tumor-stromal crosstalk and improves chemotherapeutic efficacy in … DOI 10.1074/jbc.ra119.011748↗
- Paper Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward. DOI 10.3390/ijms18040779↗
- Paper Exosomal markers (CD63 and CD9) expression and their prognostic significance using immunohistochemistry in patients wit… DOI 10.21037/jgo.2018.07.02↗
- Shafquat Azim · 3 shared papers, 2016–2023 · strength 0.31
- Paper MYB sustains hypoxic survival of pancreatic cancer cells by facilitating metabolic reprogramming. DOI 10.15252/embr.202255643↗
- Paper Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward. DOI 10.3390/ijms18040779↗
- Paper Deep sequencing and in silico analyses identify MYB-regulated gene networks and signaling pathways in pancreatic cancer. DOI 10.1038/srep28446↗
- Sumit Arora · 7 shared papers, 2011–2016 · strength 0.29
- Paper MYB Promotes Desmoplasia in Pancreatic Cancer through Direct Transcriptional Up-regulation and Cooperative Action of So… DOI 10.1074/jbc.m116.732651↗
- Paper Gemcitabine triggers angiogenesis-promoting molecular signals in pancreatic cancer cells: Therapeutic implications. DOI 10.18632/oncotarget.3784↗
- Paper MicroRNA-345 induces apoptosis in pancreatic cancer cells through potentiation of caspase-dependent and -independent pa… DOI 10.1038/bjc.2015.252↗
- Mary C. Patton · 3 shared papers, 2017–2020 · strength 0.29
- Paper Hypoxia alters the release and size distribution of extracellular vesicles in pancreatic cancer cells to support their … DOI 10.1002/jcb.29328↗
- Paper Exosomal markers (CD63 and CD9) expression and their prognostic significance using immunohistochemistry in patients wit… DOI 10.21037/jgo.2018.07.02↗
- Paper Exosomes confer chemoresistance to pancreatic cancer cells by promoting ROS detoxification and miR-155-mediated suppres… DOI 10.1038/bjc.2017.18↗
- Mary Oluwadamilola Haastrup · 1 shared paper, 2024 · strength 0.23
- Nikhil Tyagi · 4 shared papers, 2014–2016 · strength 0.22
- Paper MYB Promotes Desmoplasia in Pancreatic Cancer through Direct Transcriptional Up-regulation and Cooperative Action of So… DOI 10.1074/jbc.m116.732651↗
- Paper p-21 activated kinase 4 (PAK4) maintains stem cell-like phenotypes in pancreatic cancer cells through activation of STA… DOI 10.1016/j.canlet.2015.10.028↗
- Paper p-21 activated kinase 4 promotes proliferation and survival of pancreatic cancer cells through AKT- and ERK-dependent a… DOI 10.18632/oncotarget.2398↗
- Martin J. Heslin · 1 shared paper, 2022 · strength 0.16
- Marcus Chuan Beng Tan · 1 shared paper, 2019 · strength 0.07
- Gary Anthony Piazza · 1 shared paper, 2013 · strength 0.03
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.