ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Andrea Viale
Matched on ORCID iD 0000-0003-4328-1044; OpenAlex author ID A5075562619; NIH RePORTER profile ID 10590694. Sources: OpenAlex.
Their work
- Papers
- 20 (first author 1, last author 1)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 73
2014: 2 papers · 2015: 1 paper · 2016: 1 paper · 2017: 2 papers · 2018: none · 2019: 1 paper · 2020: 2 papers · 2021: 1 paper · 2022: 1 paper · 2023: none · 2024: 3 papers · 2025: 1 paper, 1 grant · 2026: 5 papers
Papers 20
- Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. Co-author DOI 10.1038/s41586-026-10488-6↗
- Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. Co-author DOI 10.1038/s41586-026-10995-6↗
- Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. Co-author DOI 10.1038/s41586-026-11134-x↗
- GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic Adenocarcinoma. Co-author DOI 10.1158/0008-5472.can-24-4211↗
- DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-25-3849↗
- KRAS Inhibition Activates an Actionable CD24 "Do Not Eat Me" Signal in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-25-2024↗
- WRAD core perturbation impairs DNA replication fidelity promoting immunoediting in pancreatic cancer Co-author DOI 10.1101/2024.10.21.619543↗
- Clonal dominance defines metastatic dissemination in pancreatic cancer. Last author DOI 10.1126/sciadv.add9342↗
- Activation of endogenous retroviruses and induction of viral mimicry by MEK1/2 inhibition in pancreatic cancer. Co-author DOI 10.1126/sciadv.adk5386↗
- Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic adenocarcinoma cells. Co-author DOI 10.1038/s43018-022-00463-1↗
- PRMT1-dependent regulation of RNA metabolism and DNA damage response sustains pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41467-021-24798-y↗
- Clonal Heterogeneity Supports Mitochondrial Metabolism in Pancreatic Cancer Co-author DOI 10.1101/2020.05.15.098368↗
- Diversity Across the Pancreatic Ductal Adenocarcinoma Disease Spectrum Revealed by Network-Anchored Functional Genomics Co-author DOI 10.1101/2020.09.17.302034↗
- Syndecan 1 is a critical mediator of macropinocytosis in pancreatic cancer. Co-author DOI 10.1038/s41586-019-1062-1↗
- Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. Co-author DOI 10.1038/nature21052↗
- Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. Co-author DOI 10.1038/nature21064↗
- In Vivo Functional Platform Targeting Patient-Derived Xenografts Identifies WDR5-Myc Association as a Critical Determinant of Pancreatic Cancer. Co-author DOI 10.1016/j.celrep.2016.05.063↗
- Genetic events that limit the efficacy of MEK and RTK inhibitor therapies in a mouse model of KRAS-driven pancreatic cancer. First author DOI 10.1158/0008-5472.can-14-1854↗
- Yap1 activation enables bypass of oncogenic Kras addiction in pancreatic cancer. Co-author DOI 10.1016/j.cell.2014.06.003↗
- Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. Co-author DOI 10.1038/nature13611↗
Grants 1
Closest collaborators
The 25 strongest of 73 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Giulio Francesco Draetta · 16 shared papers, 2014–2026 · strength 1.70
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Giannicola Genovese · 14 shared papers, 2014–2026 · strength 1.61
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Huamin Wang · 13 shared papers, 2014–2026 · strength 1.44
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Haoqiang Ying · 13 shared papers, 2014–2026 · strength 1.44
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Timothy Paul Heffernan · 14 shared papers, 2014–2026 · strength 1.40
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-3849↗
- Alessandro Carugo · 12 shared papers, 2014–2026 · strength 1.20
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-3849↗
- Wantong Yao · 11 shared papers, 2014–2026 · strength 1.17
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-3849↗
- Ronald Anthony Depinho · 11 shared papers, 2014–2026 · strength 1.05
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Virginia Giuliani · 9 shared papers, 2014–2026 · strength 1.03
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-3849↗
- I-Lin Ho · 7 shared papers, 2019–2026 · strength 0.96
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-3849↗
- Paper Activation of endogenous retroviruses and induction of viral mimicry by MEK1/2 inhibition in pancreatic cancer. DOI 10.1126/sciadv.adk5386↗
- Jason B. Fleming · 10 shared papers, 2014–2026 · strength 0.93
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Anirban Maitra · 9 shared papers, 2016–2026 · strength 0.89
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Francesca Citron · 5 shared papers, 2024–2026 · strength 0.82
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-3849↗
- Paper Activation of endogenous retroviruses and induction of viral mimicry by MEK1/2 inhibition in pancreatic cancer. DOI 10.1126/sciadv.adk5386↗
- Ziheng Chen · 5 shared papers, 2019–2026 · strength 0.72
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper DPY30 Is an Epigenetic Decoupler Linking Replication Stress to Immunoediting in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-3849↗
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Ya’an Kang · 5 shared papers, 2014–2026 · strength 0.53
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Prasenjit Dey · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Tony Gutschner · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Johnathon Lynn Rose · 5 shared papers, 2016–2026 · strength 0.47
- Paper GFER Represents a Target for Dual Disruption of Redox Homeostasis and Reactivation of the Immune Response in Pancreatic… DOI 10.1158/0008-5472.can-24-4211↗
- Paper PRMT1-dependent regulation of RNA metabolism and DNA damage response sustains pancreatic ductal adenocarcinoma. DOI 10.1038/s41467-021-24798-y↗
- Paper Diversity Across the Pancreatic Ductal Adenocarcinoma Disease Spectrum Revealed by Network-Anchored Functional Genomics DOI 10.1101/2020.09.17.302034↗
- Piergiorgio Pettazzoni · 7 shared papers, 2014–2026 · strength 0.43
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Syndecan 1 is a critical mediator of macropinocytosis in pancreatic cancer. DOI 10.1038/s41586-019-1062-1↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Costas Andreas Lyssiotis · 4 shared papers, 2014–2026 · strength 0.42
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic … DOI 10.1038/s43018-022-00463-1↗
- Paper Clonal Heterogeneity Supports Mitochondrial Metabolism in Pancreatic Cancer DOI 10.1101/2020.05.15.098368↗
- Avnish Kapoor · 5 shared papers, 2014–2026 · strength 0.35
- Paper Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. DOI 10.1038/s41586-026-10488-6↗
- Paper Syndecan 1 is a critical mediator of macropinocytosis in pancreatic cancer. DOI 10.1038/s41586-019-1062-1↗
- Paper Genetic events that limit the efficacy of MEK and RTK inhibitor therapies in a mouse model of KRAS-driven pancreatic ca… DOI 10.1158/0008-5472.can-14-1854↗
- Luigi Perelli · 2 shared papers, 2024–2026 · strength 0.31
- Michael P. Kim · 3 shared papers, 2020–2024 · strength 0.30
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Paper PRMT1-dependent regulation of RNA metabolism and DNA damage response sustains pancreatic ductal adenocarcinoma. DOI 10.1038/s41467-021-24798-y↗
- Paper Diversity Across the Pancreatic Ductal Adenocarcinoma Disease Spectrum Revealed by Network-Anchored Functional Genomics DOI 10.1101/2020.09.17.302034↗
- Lynda Chin · 4 shared papers, 2014–2026 · strength 0.29
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Paper In Vivo Functional Platform Targeting Patient-Derived Xenografts Identifies WDR5-Myc Association as a Critical Determin… DOI 10.1016/j.celrep.2016.05.063↗
- Lifeng Yang · 2 shared papers, 2017–2026 · strength 0.26
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.