ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Tony Gutschner
Matched on ORCID iD 0000-0002-1722-3134; OpenAlex author ID A5063332455. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 0, last author 3)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 29
2017: 2 papers · 2018: none · 2019: none · 2020: 2 papers · 2021: none · 2022: none · 2023: none · 2024: none · 2025: 1 paper · 2026: 4 papers
Papers 9
- Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. Co-author DOI 10.1038/s41586-026-10995-6↗
- Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. Co-author DOI 10.1038/s41586-026-11134-x↗
- FOXM1 regulates platelet-induced anoikis resistance in pancreatic cancer cells. Co-author DOI 10.1186/s12964-025-02644-8↗
- Multi-level Analysis of Codon Usage Patterns Reveals Systematic Optimization of Oncogenic Gene Expression in Pancreatic Cancer Last author DOI 10.64898/2026.04.24.720399↗
- Long Non-Coding RNAs and RNA-Binding Proteins in Pancreatic Cancer Development and Progression. Last author DOI 10.3390/cancers17101601↗
- LINC00261 Is Differentially Expressed in Pancreatic Cancer Subtypes and Regulates a Pro-Epithelial Cell Identity. Co-author DOI 10.3390/cancers12051227↗
- Comprehensive Analysis of LincRNAs in Classical and Basal-Like Subtypes of Pancreatic Cancer. Last author DOI 10.3390/cancers12082077↗
- Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. Co-author DOI 10.1038/nature21052↗
- Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. Co-author DOI 10.1038/nature21064↗
Closest collaborators
The 25 strongest of 29 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Monika Hämmerle · 4 shared papers, 2020–2026 · strength 0.96
- Paper Multi-level Analysis of Codon Usage Patterns Reveals Systematic Optimization of Oncogenic Gene Expression in Pancreatic… DOI 10.64898/2026.04.24.720399↗
- Paper FOXM1 regulates platelet-induced anoikis resistance in pancreatic cancer cells. DOI 10.1186/s12964-025-02644-8↗
- Paper Comprehensive Analysis of LincRNAs in Classical and Basal-Like Subtypes of Pancreatic Cancer. DOI 10.3390/cancers12082077↗
- Stefan Hüttelmaier · 4 shared papers, 2020–2026 · strength 0.90
- Paper Multi-level Analysis of Codon Usage Patterns Reveals Systematic Optimization of Oncogenic Gene Expression in Pancreatic… DOI 10.64898/2026.04.24.720399↗
- Paper Long Non-Coding RNAs and RNA-Binding Proteins in Pancreatic Cancer Development and Progression. DOI 10.3390/cancers17101601↗
- Paper Comprehensive Analysis of LincRNAs in Classical and Basal-Like Subtypes of Pancreatic Cancer. DOI 10.3390/cancers12082077↗
- Markus Glaß · 3 shared papers, 2020–2026 · strength 0.63
- Paper Multi-level Analysis of Codon Usage Patterns Reveals Systematic Optimization of Oncogenic Gene Expression in Pancreatic… DOI 10.64898/2026.04.24.720399↗
- Paper Comprehensive Analysis of LincRNAs in Classical and Basal-Like Subtypes of Pancreatic Cancer. DOI 10.3390/cancers12082077↗
- Paper LINC00261 Is Differentially Expressed in Pancreatic Cancer Subtypes and Regulates a Pro-Epithelial Cell Identity. DOI 10.3390/cancers12051227↗
- Pit Preckwinkel · 2 shared papers, 2025–2026 · strength 0.62
- Andrea Viale · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Huamin Wang · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Giannicola Genovese · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Haoqiang Ying · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Ronald Anthony Depinho · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Jason B. Fleming · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Prasenjit Dey · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Anirban Maitra · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Giulio Francesco Draetta · 4 shared papers, 2017–2026 · strength 0.49
- Paper Author Correction: Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/s41586-026-11134-x↗
- Paper Author Correction: Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/s41586-026-10995-6↗
- Paper Genomic deletion of malic enzyme 2 confers collateral lethality in pancreatic cancer. DOI 10.1038/nature21052↗
- Lisa Mueller · 1 shared paper, 2026 · strength 0.36
- Alissa Ernesti-Soldatkin · 1 shared paper, 2026 · strength 0.33
- Ágnes Dorn · 2 shared papers, 2020 · strength 0.27
- Lifeng Yang · 2 shared papers, 2017–2026 · strength 0.26
- Di Zhao · 2 shared papers, 2017–2026 · strength 0.26
- Ya’an Kang · 2 shared papers, 2017–2026 · strength 0.26
- Deepak Nagrath · 2 shared papers, 2017–2026 · strength 0.26
- Alessandro Carugo · 2 shared papers, 2017–2026 · strength 0.23
- Virginia Giuliani · 2 shared papers, 2017–2026 · strength 0.23
- Timothy Paul Heffernan · 2 shared papers, 2017–2026 · strength 0.23
- Marco Dal Molin · 2 shared papers, 2017–2026 · strength 0.23
- Laura DeLong Wood · 2 shared papers, 2017–2026 · strength 0.23
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.