ResearcherMemorial Sloan Kettering Cancer CenterActive in the last 2 years
Alvin P. Makohon-Moore
Matched on ORCID iD 0000-0002-4781-3565; OpenAlex author ID A5036118290; NIH RePORTER profile ID 10610378. Sources: OpenAlex.
Their work
- Papers
- 20
- Trials
- 0
- Grants
- 3
- Labs led
- 1
- Collaborators
- 77
2013: 2 papers · 2014: 1 grant · 2015: 2 papers · 2016: 2 papers · 2017: 4 papers · 2018: none · 2019: 2 papers · 2020: 3 papers, 1 grant · 2021: 1 paper · 2022: 1 paper · 2023: 1 paper · 2024: 1 grant · 2025: 2 papers
Papers 20
- The Evolutionary Forest of Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-23-1541↗
- The Noncoding Mutational Landscape of Pancreatic Cancer Reveals Recurrent Somatic Mutations in Enhancer Regions. Co-author DOI 10.1158/2767-9764.crc-24-0167↗
- Application of high-throughput single-nucleus DNA sequencing in pancreatic cancer. Co-author DOI 10.1038/s41467-023-36344-z↗
- Neoantigen quality predicts immunoediting in survivors of pancreatic cancer. Co-author DOI 10.1038/s41586-022-04735-9↗
- Pancreatic cancer prognosis is predicted by an ATAC-array technology for assessing chromatin accessibility. Co-author DOI 10.1038/s41467-021-23237-2↗
- A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s43018-019-0010-1↗
- Evolutionary Dynamics of Non-Coding Regions in Pancreatic Ductal Adenocarcinoma Co-author DOI 10.1101/2020.09.11.294389↗
- The Evolutionary Origins of Recurrent Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-19-1508↗
- Characterization of genetic subclonal evolution in pancreatic cancer mouse models. Co-author DOI 10.1038/s41467-019-13100-w↗
- The Genetic Basis of Transcriptional and Spatial Heterogeneity of Squamous Features in Pancreatic Ductal Adenocarcinoma Co-author DOI 10.1101/548354↗
- Epigenomic reprogramming during pancreatic cancer progression links anabolic glucose metabolism to distant metastasis. Co-author DOI 10.1038/ng.3753↗
- Limited heterogeneity of known driver gene mutations among the metastases of individual patients with pancreatic cancer. Co-author DOI 10.1038/ng.3764↗
- An unusual genomic variant of pancreatic ductal adenocarcinoma with an indolent clinical course. Co-author DOI 10.1101/mcs.a001701↗
- Mutant p53 Together with TGFβ Signaling Influence Organ-Specific Hematogenous Colonization Patterns of Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-15-1615↗
- Pancreatic cancer biology and genetics from an evolutionary perspective. Co-author DOI 10.1038/nrc.2016.66↗
- Whole Genome Sequencing Defines the Genetic Heterogeneity of Familial Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-15-0402↗
- Transflip mutations produce deletions in pancreatic cancer. Co-author DOI 10.1002/gcc.22258↗
- Retrotransposon insertions in the clonal evolution of pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/nm.3919↗
- Considerations for sequencing analyses of pancreatic cancer progression and metastasis. Co-author DOI 10.1007/978-1-62703-287-2_6↗
- Pancreatic cancer genomics: insights and opportunities for clinical translation. Co-author DOI 10.1186/gm430↗
Grants 3
Closest collaborators
The 25 strongest of 77 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Christine A. Iacobuzio–Donahue · 20 shared papers, 2013–2025 · strength 1.80
- Paper The Noncoding Mutational Landscape of Pancreatic Cancer Reveals Recurrent Somatic Mutations in Enhancer Regions. DOI 10.1158/2767-9764.crc-24-0167↗
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper Application of high-throughput single-nucleus DNA sequencing in pancreatic cancer. DOI 10.1038/s41467-023-36344-z↗
- Jungeui Hong · 8 shared papers, 2017–2025 · strength 0.94
- Paper The Noncoding Mutational Landscape of Pancreatic Cancer Reveals Recurrent Somatic Mutations in Enhancer Regions. DOI 10.1158/2767-9764.crc-24-0167↗
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper Application of high-throughput single-nucleus DNA sequencing in pancreatic cancer. DOI 10.1038/s41467-023-36344-z↗
- Akimasa Hayashi · 7 shared papers, 2019–2025 · strength 0.89
- Paper The Noncoding Mutational Landscape of Pancreatic Cancer Reveals Recurrent Somatic Mutations in Enhancer Regions. DOI 10.1158/2767-9764.crc-24-0167↗
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper Application of high-throughput single-nucleus DNA sequencing in pancreatic cancer. DOI 10.1038/s41467-023-36344-z↗
- Marc A. Attiyeh · 7 shared papers, 2017–2025 · strength 0.64
- Zachary A. Kohutek · 7 shared papers, 2015–2025 · strength 0.57
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-019-0010-1↗
- Paper An unusual genomic variant of pancreatic ductal adenocarcinoma with an indolent clinical course. DOI 10.1101/mcs.a001701↗
- Laura DeLong Wood · 6 shared papers, 2016–2025 · strength 0.51
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper The Evolutionary Origins of Recurrent Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1508↗
- Paper A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-019-0010-1↗
- Eileen Mary O'Reilly · 4 shared papers, 2020–2025 · strength 0.46
- Paper The Noncoding Mutational Landscape of Pancreatic Cancer Reveals Recurrent Somatic Mutations in Enhancer Regions. DOI 10.1158/2767-9764.crc-24-0167↗
- Paper Evolutionary Dynamics of Non-Coding Regions in Pancreatic Ductal Adenocarcinoma DOI 10.1101/2020.09.11.294389↗
- Paper The Evolutionary Origins of Recurrent Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1508↗
- Hitomi Sakamoto · 4 shared papers, 2019–2025 · strength 0.43
- Paper The Evolutionary Forest of Pancreatic Cancer. DOI 10.1158/2159-8290.cd-23-1541↗
- Paper The Evolutionary Origins of Recurrent Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1508↗
- Paper A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-019-0010-1↗
- Ralph H. Hruban MD · 7 shared papers, 2015–2020 · strength 0.40
- Paper The Evolutionary Origins of Recurrent Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1508↗
- Paper A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-019-0010-1↗
- Paper The Genetic Basis of Transcriptional and Spatial Heterogeneity of Squamous Features in Pancreatic Ductal Adenocarcinoma DOI 10.1101/548354↗
- Katelyn Mullen · 2 shared papers, 2023–2025 · strength 0.36
- Caitlin A. McIntyre · 2 shared papers, 2023–2025 · strength 0.36
- Haochen Zhang · 2 shared papers, 2023–2025 · strength 0.36
- Yi Zhong · 5 shared papers, 2017–2020 · strength 0.36
- Paper Characterization of genetic subclonal evolution in pancreatic cancer mouse models. DOI 10.1038/s41467-019-13100-w↗
- Paper A unifying paradigm for transcriptional heterogeneity and squamous features in pancreatic ductal adenocarcinoma. DOI 10.1038/s43018-019-0010-1↗
- Paper The Genetic Basis of Transcriptional and Spatial Heterogeneity of Squamous Features in Pancreatic Ductal Adenocarcinoma DOI 10.1101/548354↗
- Johannes G. Reiter · 3 shared papers, 2017–2020 · strength 0.23
- Paper Evolutionary Dynamics of Non-Coding Regions in Pancreatic Ductal Adenocarcinoma DOI 10.1101/2020.09.11.294389↗
- Paper The Evolutionary Origins of Recurrent Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1508↗
- Paper Limited heterogeneity of known driver gene mutations among the metastases of individual patients with pancreatic cancer. DOI 10.1038/ng.3764↗
- Wungki Park · 1 shared paper, 2025 · strength 0.21
- Marinela Capanu · 1 shared paper, 2025 · strength 0.20
- Benjamin J. Raphael · 1 shared paper, 2025 · strength 0.20
- Vinod P. Balachandran · 2 shared papers, 2021–2022 · strength 0.19
- Steven D Leach · 2 shared papers, 2017–2021 · strength 0.15
- Paper Pancreatic cancer prognosis is predicted by an ATAC-array technology for assessing chromatin accessibility. DOI 10.1038/s41467-021-23237-2↗
- Paper Mutant p53 Together with TGFβ Signaling Influence Organ-Specific Hematogenous Colonization Patterns of Pancreatic Cance… DOI 10.1158/1078-0432.ccr-15-1615↗
- Olca Baştürk · 2 shared papers, 2019–2020 · strength 0.15
- Noushin Niknafs · 2 shared papers, 2016–2019 · strength 0.13
- Rachel Karchin · 2 shared papers, 2016–2019 · strength 0.13
- Joseph M. Herman · 2 shared papers, 2016–2017 · strength 0.11
- Benjamin D. Greenbaum · 1 shared paper, 2022 · strength 0.10
- Taha Merghoub Merghoub · 1 shared paper, 2022 · strength 0.10
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.