ResearcherMemorial Sloan Kettering Cancer CenterActive in the last 2 years
Richard J. Wong
Matched on ORCID iD 0000-0001-6259-7314; OpenAlex author ID A5078178893; NIH RePORTER profile ID 2052781. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 0, last author 1)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 16
2012: 1 paper · 2013: none · 2014: 1 paper · 2015: 1 grant · 2016: none · 2017: 1 paper · 2018: none · 2019: none · 2020: none · 2021: none · 2022: 1 paper · 2023: none · 2024: none · 2025: none · 2026: 2 papers
Papers 6
- Pancreatic cancer fibrosis activates protumorigenic Schwann cells through a nuclear mechanosensing mechanism. Co-author DOI 10.1136/gutjnl-2025-337316↗
- Invasion status stratifies the composition of the human pancreatic cancer perineural niche Co-author DOI 10.64898/2026.08.05.743048↗
- Reprogrammed Schwann Cells Organize into Dynamic Tracks that Promote Pancreatic Cancer Invasion. Last author DOI 10.1158/2159-8290.cd-21-1690↗
- Upregulation of RET induces perineurial invasion of pancreatic adenocarcinoma. Co-author DOI 10.1038/onc.2016.483↗
- Macrophages mediate gemcitabine resistance of pancreatic adenocarcinoma by upregulating cytidine deaminase. Co-author DOI 10.1038/onc.2013.357↗
- Endoneurial macrophages induce perineural invasion of pancreatic cancer cells by secretion of GDNF and activation of RET tyrosine kinase receptor. Co-author DOI 10.1158/0008-5472.can-12-0764↗
Grants 1
Closest collaborators
The 16 strongest of 16 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Sylvie Deborde · 3 shared papers, 2022–2026 · strength 0.66
- Paper Pancreatic cancer fibrosis activates protumorigenic Schwann cells through a nuclear mechanosensing mechanism. DOI 10.1136/gutjnl-2025-337316↗
- Paper Invasion status stratifies the composition of the human pancreatic cancer perineural niche DOI 10.64898/2026.08.05.743048↗
- Paper Reprogrammed Schwann Cells Organize into Dynamic Tracks that Promote Pancreatic Cancer Invasion. DOI 10.1158/2159-8290.cd-21-1690↗
- Pavel Stupakov · 2 shared papers, 2026 · strength 0.53
- İhsan Ekin Demir · 1 shared paper, 2026 · strength 0.27
- Olca Baştürk · 1 shared paper, 2026 · strength 0.26
- Zainab Hussain · 1 shared paper, 2026 · strength 0.26
- Nuray Tezcan · 1 shared paper, 2026 · strength 0.26
- Mara H. Sherman · 1 shared paper, 2026 · strength 0.26
- Joan Massague · 1 shared paper, 2026 · strength 0.26
- Moran Amit · 3 shared papers, 2012–2017 · strength 0.15
- Paper Upregulation of RET induces perineurial invasion of pancreatic adenocarcinoma. DOI 10.1038/onc.2016.483↗
- Paper Macrophages mediate gemcitabine resistance of pancreatic adenocarcinoma by upregulating cytidine deaminase. DOI 10.1038/onc.2013.357↗
- Paper Endoneurial macrophages induce perineural invasion of pancreatic cancer cells by secretion of GDNF and activation of RE… DOI 10.1158/0008-5472.can-12-0764↗
- Ziv Gil · 3 shared papers, 2012–2017 · strength 0.15
- Paper Upregulation of RET induces perineurial invasion of pancreatic adenocarcinoma. DOI 10.1038/onc.2016.483↗
- Paper Macrophages mediate gemcitabine resistance of pancreatic adenocarcinoma by upregulating cytidine deaminase. DOI 10.1038/onc.2013.357↗
- Paper Endoneurial macrophages induce perineural invasion of pancreatic cancer cells by secretion of GDNF and activation of RE… DOI 10.1158/0008-5472.can-12-0764↗
- Vinod P. Balachandran · 1 shared paper, 2022 · strength 0.13
- Masataka Amisaki · 1 shared paper, 2022 · strength 0.13
- Yoav Binenbaum · 2 shared papers, 2014–2017 · strength 0.12
- Noam Weizman · 2 shared papers, 2012–2014 · strength 0.08
- Paper Macrophages mediate gemcitabine resistance of pancreatic adenocarcinoma by upregulating cytidine deaminase. DOI 10.1038/onc.2013.357↗
- Paper Endoneurial macrophages induce perineural invasion of pancreatic cancer cells by secretion of GDNF and activation of RE… DOI 10.1158/0008-5472.can-12-0764↗
- Oren Cavel · 1 shared paper, 2012 · strength 0.03
- Yuman Fong · 1 shared paper, 2012 · strength 0.03
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.