ResearcherMemorial Sloan Kettering Cancer CenterActive in the last 2 years
Mara H. Sherman
Matched on ORCID iD 0000-0001-7826-3888; OpenAlex author ID A5035829341; NIH RePORTER profile ID 10431069. Sources: OpenAlex.
Their work
- Papers
- 22 (first author 3, last author 6)
- Trials
- 0
- Grants
- 5
- Labs led
- 2
- Collaborators
- 124
2014: 1 paper · 2015: 1 paper · 2016: 1 grant · 2017: 1 paper · 2018: none · 2019: 1 grant · 2020: 2 papers · 2021: none · 2022: 2 papers · 2023: 3 papers · 2024: 2 papers · 2025: 2 papers, 1 grant · 2026: 8 papers, 2 grants
Papers 22
- Niche-Specific Stromal Crosstalk in Pancreatic Ductal Adenocarcinoma Liver Metastasis. First author DOI 10.1016/j.jcmgh.2026.101907↗
- Inactivation of CDKN2AARF Promotes p53-Independent Remodeling of the PDAC Tumor Microenvironment. Co-author DOI 10.1158/0008-5472.can-25-1969↗
- Pancreatic Cancer: Translating Tumor Biology into Actionability. First author DOI 10.1158/2159-8290.cd-25-2014↗
- IFNγ-dependent tumor-cell MHC-II is restrained by MAPK signaling and associated with immunotherapy responsiveness in pancreatic ductal adenocarcinoma. Co-author DOI 10.1158/2326-6066.cir-25-1612↗
- Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic cancer. Co-author DOI 10.1172/jci.insight.192814↗
- Invasion status stratifies the composition of the human pancreatic cancer perineural niche Last author DOI 10.64898/2026.08.05.743048↗
- Continuous tissue fields organize immune composition in pancreatic cancer Co-author DOI 10.64898/2026.08.22.746444↗
- Neoantigen-reactive CD8 + T cell engagement marks exceptional survivors of pancreatic cancer Co-author DOI 10.64898/2026.09.17.752426↗
- Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. Last author DOI 10.1038/s43018-025-01035-9↗
- The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-3437↗
- Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. Last author DOI 10.1038/s43018-023-00703-y↗
- Pancreatic Cancer-Associated Fibroblasts: Where Do We Go from Here? Last author DOI 10.1158/0008-5472.can-24-2860↗
- Single-cell RNA sequencing reveals the effects of chemotherapy on human pancreatic adenocarcinoma and its tumor microenvironment. Co-author DOI 10.1038/s41467-023-36296-4↗
- Author Correction: Single-cell RNA sequencing reveals the effects of chemotherapy on human pancreatic adenocarcinoma and its tumor microenvironment. Co-author DOI 10.1038/s41467-023-39680-2↗
- Tumor Microenvironment in Pancreatic Cancer Pathogenesis and Therapeutic Resistance. Co-author DOI 10.1146/annurev-pathmechdis-031621-024600↗
- NUFIP1+ stroma keeps PDAC fed. First author DOI 10.1038/s43018-022-00421-x↗
- Mesenchymal Lineage Heterogeneity Underlies Nonredundant Functions of Pancreatic Cancer-Associated Fibroblasts. Last author DOI 10.1158/2159-8290.cd-21-0601↗
- Tumour-reprogrammed stromal BCAT1 fuels branched-chain ketoacid dependency in stromal-rich PDAC tumours. Co-author DOI 10.1038/s42255-020-0226-5↗
- Acidic fibroblast growth factor underlies microenvironmental regulation of MYC in pancreatic cancer. Last author DOI 10.1084/jem.20191805↗
- Stromal cues regulate the pancreatic cancer epigenome and metabolome. Co-author DOI 10.1073/pnas.1620164114↗
- Calcipotriol Targets LRP6 to Inhibit Wnt Signaling in Pancreatic Cancer. Co-author DOI 10.1158/1541-7786.mcr-15-0204↗
- Vitamin D receptor-mediated stromal reprogramming suppresses pancreatitis and enhances pancreatic cancer therapy. Co-author DOI 10.1016/j.cell.2014.08.007↗
Grants 5
- Fatty acid signaling in the pancreatic tumor microenvironment R01CA229580↗
- Origins and functions of pancreatic cancer-associated fibroblasts R01CA250917↗
- Administrative and Biostatistics Core P01CA244114↗
- Regulation of cancer cell metabolism and growth by the pancreatic tumor stroma R00CA188259↗
- Regulation of cancer cell metabolism and growth by the pancreatic tumor stroma K99CA188259↗
Closest collaborators
The 25 strongest of 124 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Sohinee Bhattacharyya · 5 shared papers, 2020–2026 · strength 0.93
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Inactivation of CDKN2AARF Promotes p53-Independent Remodeling of the PDAC Tumor Microenvironment. DOI 10.1158/0008-5472.can-25-1969↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Laura D Attardi · 1 shared paper, 1 shared grant, 2025–2026 · strength 0.79
- Holly Sandborg · 3 shared papers, 2022–2025 · strength 0.78
- Paper NUFIP1+ stroma keeps PDAC fed. DOI 10.1038/s43018-022-00421-x↗
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Rosalie C. Sears · 4 shared papers, 2020–2026 · strength 0.78
- Paper Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic … DOI 10.1172/jci.insight.192814↗
- Paper IFNγ-dependent tumor-cell MHC-II is restrained by MAPK signaling and associated with immunotherapy responsiveness in pa… DOI 10.1158/2326-6066.cir-25-1612↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Marc Nater · 1 shared paper, 2026 · strength 0.63
- Garry Nolan · 1 shared grant, 2025 · strength 0.57
- Seung K Kim · 1 shared grant, 2025 · strength 0.57
- Steven E Artandi · 1 shared grant, 2025 · strength 0.57
- Edgar G. Engleman · 1 shared grant, 2025 · strength 0.57
- Walter Gwang-Up Park · 1 shared grant, 2025 · strength 0.57
- Marie C. Hasselluhn · 2 shared papers, 2024–2026 · strength 0.50
- Marsha Reyngold · 2 shared papers, 2026 · strength 0.49
- Kevin C. Soares · 2 shared papers, 2026 · strength 0.49
- Wungki Park · 2 shared papers, 2026 · strength 0.49
- Eileen Mary O'Reilly · 2 shared papers, 2026 · strength 0.49
- Fiyinfolu O. Balogun · 2 shared papers, 2026 · strength 0.49
- Christine A. Iacobuzio–Donahue · 2 shared papers, 2026 · strength 0.49
- Terry K. Morgan · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Luis Francisco Diaz · 2 shared papers, 2024–2025 · strength 0.45
- Paper Author Correction: Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progre… DOI 10.1038/s43018-025-01035-9↗
- Paper Autotaxin-lysolipid signaling suppresses a CCL11-eosinophil axis to promote pancreatic cancer progression. DOI 10.1038/s43018-023-00703-y↗
- Olca Baştürk · 2 shared papers, 2026 · strength 0.45
- Nuray Tezcan · 2 shared papers, 2026 · strength 0.45
- Kevin Hawthorne · 2 shared papers, 2025–2026 · strength 0.42
- Paper Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic … DOI 10.1172/jci.insight.192814↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Ariana L. Sattler · 2 shared papers, 2022–2026 · strength 0.39
- Paper Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic … DOI 10.1172/jci.insight.192814↗
- Paper Mesenchymal Lineage Heterogeneity Underlies Nonredundant Functions of Pancreatic Cancer-Associated Fibroblasts. DOI 10.1158/2159-8290.cd-21-0601↗
- Gregory Lawrence Beatty · 1 shared paper, 2023 · strength 0.39
- Zheng Xia · 2 shared papers, 2022–2026 · strength 0.35
- Paper IFNγ-dependent tumor-cell MHC-II is restrained by MAPK signaling and associated with immunotherapy responsiveness in pa… DOI 10.1158/2326-6066.cir-25-1612↗
- Paper Mesenchymal Lineage Heterogeneity Underlies Nonredundant Functions of Pancreatic Cancer-Associated Fibroblasts. DOI 10.1158/2159-8290.cd-21-0601↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.