ResearcherOregon Health & Science UniversityActive in the last 2 years
Kevin Hawthorne
Matched on ORCID iD 0000-0002-2657-8762, 0000-0002-9195-5720; OpenAlex author ID A5008953662, A5019518419, A5060649081. Sources: OpenAlex.
Their work
- Papers
- 9
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 46
2025: 5 papers · 2026: 4 papers
Papers 9
- The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1136/jitc-2025-sitc2025.1250↗
- Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic cancer. Co-author DOI 10.1172/jci.insight.192814↗
- Spatially Resolved Epithelial States Delineate Immunosuppressive Niches of Impaired Myeloid Antigen Presentation in PDAC Co-author DOI 10.64898/2026.07.28.741300↗
- Epigenetic progression of pancreatic cancer to aggressive subtypes involves alternate routes of lineage reprogramming in subtype-intermediate progenitor cells Co-author DOI 10.64898/2026.09.28.754674↗
- Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in pancreatic cancer. Co-author DOI 10.1038/s43018-024-00881-3↗
- Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma Co-author DOI 10.1101/2025.02.07.632847↗
- Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance in a novel pancreatic cancer mouse model Co-author DOI 10.1101/2025.07.14.664767↗
- The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-3437↗
- HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer. Co-author DOI 10.1158/2767-9764.crc-25-0355↗
Closest collaborators
The 25 strongest of 46 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Rosalie C. Sears · 9 shared papers, 2025–2026 · strength 1.93
- Paper Spatially Resolved Epithelial States Delineate Immunosuppressive Niches of Impaired Myeloid Antigen Presentation in PDAC DOI 10.64898/2026.07.28.741300↗
- Paper Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic … DOI 10.1172/jci.insight.192814↗
- Paper Epigenetic progression of pancreatic cancer to aggressive subtypes involves alternate routes of lineage reprogramming i… DOI 10.64898/2026.09.28.754674↗
- Jonathan Robert Brody · 5 shared papers, 2025–2026 · strength 0.98
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer. DOI 10.1158/2767-9764.crc-25-0355↗
- Paper Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma DOI 10.1101/2025.02.07.632847↗
- Lisa M. Coussens · 4 shared papers, 2025–2026 · strength 0.76
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma DOI 10.1101/2025.02.07.632847↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Motoyuki Tsuda · 4 shared papers, 2025–2026 · strength 0.74
- Paper Epigenetic progression of pancreatic cancer to aggressive subtypes involves alternate routes of lineage reprogramming i… DOI 10.64898/2026.09.28.754674↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Ellen M. Langer · 4 shared papers, 2025–2026 · strength 0.74
- Paper Epigenetic progression of pancreatic cancer to aggressive subtypes involves alternate routes of lineage reprogramming i… DOI 10.64898/2026.09.28.754674↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Jennifer M. Finan · 3 shared papers, 2025–2026 · strength 0.64
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer. DOI 10.1158/2767-9764.crc-25-0355↗
- Paper Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma DOI 10.1101/2025.02.07.632847↗
- Yifei Guo · 3 shared papers, 2025–2026 · strength 0.64
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer. DOI 10.1158/2767-9764.crc-25-0355↗
- Paper Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma DOI 10.1101/2025.02.07.632847↗
- Zheng Xia · 3 shared papers, 2025–2026 · strength 0.64
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer. DOI 10.1158/2767-9764.crc-25-0355↗
- Paper Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma DOI 10.1101/2025.02.07.632847↗
- Canping Chen · 3 shared papers, 2025–2026 · strength 0.64
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper HuR-Regulated Extracellular Vesicles Promote Endothelial Cell Remodeling in Pancreatic Cancer. DOI 10.1158/2767-9764.crc-25-0355↗
- Paper Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma DOI 10.1101/2025.02.07.632847↗
- Katie E. Blise · 3 shared papers, 2025–2026 · strength 0.60
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper Post-transcriptional regulator HuR promotes immune evasion in pancreatic ductal adenocarcinoma DOI 10.1101/2025.02.07.632847↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Patrick Joseph Worth · 3 shared papers, 2025–2026 · strength 0.58
- Paper Epigenetic progression of pancreatic cancer to aggressive subtypes involves alternate routes of lineage reprogramming i… DOI 10.64898/2026.09.28.754674↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Brett C. Sheppard · 3 shared papers, 2025–2026 · strength 0.57
- Paper Epigenetic progression of pancreatic cancer to aggressive subtypes involves alternate routes of lineage reprogramming i… DOI 10.64898/2026.09.28.754674↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Jason M. Link · 3 shared papers, 2025–2026 · strength 0.57
- Paper Epigenetic progression of pancreatic cancer to aggressive subtypes involves alternate routes of lineage reprogramming i… DOI 10.64898/2026.09.28.754674↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Katherine R. Pelz · 3 shared papers, 2025–2026 · strength 0.55
- Paper The Posttranscriptional Regulator HuR Promotes Immune Evasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1136/jitc-2025-sitc2025.1250↗
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Isabel A. English · 3 shared papers, 2025 · strength 0.49
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Aaron J. Grossberg · 2 shared papers, 2025–2026 · strength 0.43
- Grace A. McCarthy · 2 shared papers, 2025–2026 · strength 0.43
- Robert L Eil · 2 shared papers, 2025–2026 · strength 0.43
- Mara H. Sherman · 2 shared papers, 2025–2026 · strength 0.42
- Paper Sympathetic nerve-fibroblast crosstalk drives nerve injury, fibroblast activation, and matrix remodeling in pancreatic … DOI 10.1172/jci.insight.192814↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Vidhi M. Shah · 2 shared papers, 2025–2026 · strength 0.38
- Amy S. Farrell · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Brittany L. Allen-Petersen · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-3437↗
- Terry K. Morgan · 2 shared papers, 2025 · strength 0.33
- Paper Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance … DOI 10.1101/2025.07.14.664767↗
- Paper Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in … DOI 10.1038/s43018-024-00881-3↗
- Michael Angelo · 1 shared paper, 2026 · strength 0.28
- Cameron R. Walker · 1 shared paper, 2026 · strength 0.28
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.